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Conclusion: When replication is stalled, accessory subunits of DNA Pol participate in recruitment of translesion DNA Pol . Significance: This finding provides a novel mechanism of DNA lesion bypass in eukaryotes.
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T. Tahirov
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Y. Pavlov
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H. Siebler
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A. G. Lada
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Yinbo Zhang
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Andrey G. Baranovskiy
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