p16/CDKN2 Gene and p53 Gene Alterations in Japanese Non‐smoking Female Lung Adenocarcinoma
暂无分享,去创建一个
[1] M. Raffeld,et al. Involvement of CDKN2 (p16INK4A/MTS1) and p15INK4B/MTS2 in human leukemias and lymphomas. , 1995, Cancer research.
[2] G. Hannon,et al. Mutations in the p16INK4/MTS1/CDKN2, p15INK4B/MTS2, and p18 genes in primary and metastatic lung cancer. , 1995, Cancer research.
[3] B. Tycko,et al. MTS1/p16/CDKN2 lesions in primary glioblastoma multiforme. , 1995, The American journal of pathology.
[4] A. Okamoto,et al. IS-12 Mutation and altered expression of P16^ in human cancer. , 1995 .
[5] A. Rustgi,et al. MTS-1 (CDKN2) tumor suppressor gene deletions are a frequent event in esophagus squamous cancer and pancreatic adenocarcinoma cell lines. , 1995, Oncogene.
[6] C. D. Edwards,et al. Reciprocal Rb inactivation and p16INK4 expression in primary lung cancers and cell lines. , 1995, Cancer research.
[7] R. Ueda,et al. In vivo occurrence of p16 (MTS1) and p15 (MTS2) alterations preferentially in non-small cell lung cancers. , 1995, Cancer research.
[8] G. Finocchiaro,et al. Mutation rate of the CDKN2 gene in malignant gliomas. , 1994, Cancer research.
[9] K. Kinzler,et al. Deletion of p16 and p15 genes in brain tumors. , 1994, Cancer research.
[10] G. Reifenberger,et al. CDKN2 (p16/MTS1) gene deletion or CDK4 amplification occurs in the majority of glioblastomas. , 1994, Cancer research.
[11] J. Abraham,et al. The MTS1 gene is frequently mutated in primary human esophageal tumors. , 1994, Oncogene.
[12] C. James,et al. CDK4 amplification is an alternative mechanism to p16 gene homozygous deletion in glioma cell lines. , 1994, Cancer research.
[13] A. Okamoto,et al. Mutations and altered expression of p16INK4 in human cancer. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[14] H. Dosaka-akita,et al. Abnormal p53 expression in human lung cancer is associated with histologic subtypes and patient smoking history. , 1994, American journal of clinical pathology.
[15] O. Olopade,et al. p16 alterations and deletion mapping of 9p21-p22 in malignant mesothelioma. , 1994, Cancer research.
[16] D. Louis,et al. Mutational analysis of CDKN2 (MTS1/p16ink4) in human breast carcinomas. , 1994, Cancer research.
[17] D Hoffmann,et al. Smoking and lung cancer: scientific challenges and opportunities. , 1994, Cancer research.
[18] A. Klein-Szanto,et al. Higher frequency of alterations in the p16/CDKN2 gene in squamous cell carcinoma cell lines than in primary tumors of the head and neck. , 1994, Cancer research.
[19] L. Clark,et al. Loss of heterozygosity of chromosome 9p21 is associated with the immortal phenotype of neoplastic human head and neck keratinocytes. , 1994, Cancer research.
[20] R. Metcalf,et al. p53 mutations in lung cancers from Japanese mustard gas workers. , 1994, Carcinogenesis.
[21] C. Harris,et al. Mutations in the p53 tumor suppressor gene: clues to cancer etiology and molecular pathogenesis. , 1994, Cancer research.
[22] W. Clark,et al. Germline p16 mutations in familial melanoma , 1994, Nature Genetics.
[23] R. Hruban,et al. Frequent somatic mutations and homozygous deletions of the p16 (MTS1) gene in pancreatic adenocarcinoma , 1994, Nature Genetics.
[24] M. Ogawa,et al. Somatic mutations of the MTS (multiple tumor suppressor) 1/CDK4l (cyclin-dependent kinase-4 inhibitor) gene in human primary non-small cell lung carcinomas. , 1994, Biochemical and biophysical research communications.
[25] Peter A. Jones,et al. P16 gene in uncultured tumours , 1994, Nature.
[26] D. Sidransky,et al. Rates of p16 (MTS1) mutations in primary tumors with 9p loss. , 1994, Science.
[27] S. Mori,et al. Frequent somatic mutation of the MTS1/CDK4I (multiple tumor suppressor/cyclin-dependent kinase 4 inhibitor) gene in esophageal squamous cell carcinoma. , 1994, Cancer research.
[28] E. Wynder,et al. Re: Endocrine factors and adenocarcinoma of the lung in women. , 1994, Journal of the National Cancer Institute.
[29] D. Carson,et al. Deletions of the cyclin-dependent kinase-4 inhibitor gene in multiple human cancers , 1994, Nature.
[30] M. Skolnick,et al. A cell cycle regulator potentially involved in genesis of many tumor types. , 1994, Science.
[31] F. Askin,et al. Frequent loss of chromosome 9 in human primary non-small cell lung cancer. , 1994, Cancer research.
[32] C. Harris,et al. p53: at the crossroads of molecular carcinogenesis and risk assessment. , 1993, Science.
[33] Y. Takeshima,et al. p53 mutations in lung cancers from non-smoking atomic-bomb survivors , 1993, The Lancet.
[34] G. Hannon,et al. A new regulatory motif in cell-cycle control causing specific inhibition of cyclin D/CDK4 , 1993, Nature.
[35] S. Goodman,et al. Overexpression of the p53 Tumor Suppressor Gene Product in Primary Lung Adenocarcinomas Is Associated with Cigarette Smoking , 1993, The American journal of surgical pathology.
[36] I. Baas,et al. Detection of p53 overexpression in routinely paraffin-embedded tissue of human carcinomas using a novel target unmasking fluid. , 1993, The American journal of pathology.
[37] M. Skolnick,et al. Assignment of a locus for familial melanoma, MLM, to chromosome 9p13-p22. , 1992, Science.
[38] G. Gyapay,et al. A second-generation linkage map of the human genome , 1992, Nature.
[39] G. Lyman,et al. Environmental tobacco smoke and lung cancer risk in nonsmoking women. , 1992, Journal of the National Cancer Institute.
[40] Y. Nakamura,et al. Detection of loss of heterozygosity at the human TP53 locus using a dinucleotide repeat polymorphism , 1992, Genes, chromosomes & cancer.
[41] R. Ueda,et al. p53 mutations in non-small cell lung cancer in Japan: association between mutations and smoking. , 1992, Cancer research.
[42] R. Metcalf,et al. Archival analysis of p53 genetic and protein alterations in Chinese esophageal cancer. , 1991, Oncogene.
[43] B. Vogelstein,et al. p53 mutations in human cancers. , 1991, Science.
[44] T. Sekiya,et al. Rapid and sensitive detection of point mutations and DNA polymorphisms using the polymerase chain reaction. , 1989, Genomics.
[45] A. Churg,et al. Pathology of Occupational Lung Disease , 1988 .
[46] B. Vogelstein,et al. Purification of DNA from formaldehyde fixed and paraffin embedded human tissue. , 1985, Biochemical and biophysical research communications.
[47] E. Radford,et al. Lung cancer in Swedish iron miners exposed to low doses of radon daughters. , 1984, The New England journal of medicine.
[48] A. Kaye,et al. Homozygous deletions of the multiple tumor suppressor gene 1 in the progression of human astrocytomas. , 1995, Cancer research.
[49] K. Kikuchi,et al. A Clinical Analysis of Lung Cancer in Women. , 1995 .
[50] D. Lane,et al. High levels of p53 protein in UV-irradiated normal human skin. , 1993, Oncogene.