BID regulation by p53 contributes to chemosensitivity
暂无分享,去创建一个
Joanna K. Sax | S. Korsmeyer | W. El-Deiry | M. Murphy | J. Sax | P. Fei | E. Bernhard | Maureen E. Murphy | Stanley J. Korsmeyer | Wafik S. El-Deiry | Peiwen Fei | Eric Bernhard
[1] Xin Lu,et al. Live or let die: the cell's response to p53 , 2002, Nature Reviews Cancer.
[2] W. El-Deiry,et al. Apoptotic threshold is lowered by p53 transactivation of caspase-6 , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[3] Jiandong Chen,et al. Transcriptional Repression of the Anti-apoptoticsurvivin Gene by Wild Type p53* , 2002, The Journal of Biological Chemistry.
[4] Luca Scorrano,et al. A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis. , 2002, Developmental cell.
[5] W. El-Deiry,et al. The cyclin-dependent kinase inhibitor butyrolactone is a potent inhibitor of p21 (WAF1/CIP1 expression). , 2002, Cell cycle.
[6] G. Blandino,et al. DNA damage-dependent acetylation of p73 dictates the selective activation of apoptotic target genes. , 2002, Molecular cell.
[7] R. Iggo,et al. Chromatin immunoprecipitation analysis fails to support the latency model for regulation of p53 DNA binding activity in vivo , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[8] W. el-Deiry. Insights into cancer therapeutic design based on p53 and TRAIL receptor signaling , 2001, Cell Death and Differentiation.
[9] T. Burns,et al. Tissue specific expression of p53 target genes suggests a key role for KILLER/DR5 in p53-dependent apoptosis in vivo , 2001, Oncogene.
[10] K. Vousden,et al. Regulation and function of the p53 tumor suppressor protein. , 2001, Current opinion in cell biology.
[11] K. Helin,et al. Apaf-1 is a transcriptional target for E2F and p53 , 2001, Nature Cell Biology.
[12] J. Pietenpol,et al. Kinetics of p53 Binding to Promoter Sites In Vivo , 2001, Molecular and Cellular Biology.
[13] S. Korsmeyer,et al. Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death , 2001, Science.
[14] K. Vousden,et al. PUMA, a novel proapoptotic gene, is induced by p53. , 2001, Molecular cell.
[15] K. Kinzler,et al. PUMA induces the rapid apoptosis of colorectal cancer cells. , 2001, Molecular cell.
[16] S. Korsmeyer,et al. Posttranslational N-myristoylation of BID as a molecular switch for targeting mitochondria and apoptosis. , 2000, Science.
[17] Yusuke Nakamura,et al. p53AIP1, a Potential Mediator of p53-Dependent Apoptosis, and Its Regulation by Ser-46-Phosphorylated p53 , 2000, Cell.
[18] Yunping Lin,et al. Pidd, a new death-domain–containing protein, is induced by p53 and promotes apoptosis , 2000, Nature Genetics.
[19] V. Mootha,et al. tBID, a membrane-targeted death ligand, oligomerizes BAK to release cytochrome c. , 2000, Genes & development.
[20] T. Taniguchi,et al. Noxa, a BH3-only member of the Bcl-2 family and candidate mediator of p53-induced apoptosis. , 2000, Science.
[21] W. El-Deiry,et al. Wild-type p53 transactivates the KILLER/DR5 gene through an intronic sequence-specific DNA-binding site , 2000, Oncogene.
[22] S. Lowe,et al. PERP, an apoptosis-associated target of p53, is a novel member of the PMP-22/gas3 family. , 2000, Genes & development.
[23] W. El-Deiry,et al. BRCA1 Effects on the Cell Cycle and the DNA Damage Response Are Linked to Altered Gene Expression* , 2000, The Journal of Biological Chemistry.
[24] S. Korsmeyer,et al. Damage-induced apoptosis in intestinal epithelia from bcl-2-null and bax-null mice: investigations of the mechanistic determinants of epithelial apoptosis in vivo , 1999, Oncogene.
[25] A. Levine,et al. Transcriptional repression by wild-type p53 utilizes histone deacetylases, mediated by interaction with mSin3a. , 1999, Genes & development.
[26] S. Korsmeyer,et al. Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis , 1999, Nature.
[27] K. Kinzler,et al. Disruption of p53 in human cancer cells alters the responses to therapeutic agents. , 1999, The Journal of clinical investigation.
[28] E. Alnemri,et al. Molecular cloning and functional analysis of the mouse homologue of the KILLER/DR5 tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) death receptor. , 1999, Cancer research.
[29] S. Korsmeyer,et al. Caspase Cleaved BID Targets Mitochondria and Is Required for Cytochrome c Release, while BCL-XL Prevents This Release but Not Tumor Necrosis Factor-R1/Fas Death* , 1999, The Journal of Biological Chemistry.
[30] D. Israeli,et al. p53 Activates the CD95 (APO-1/Fas) Gene in Response to DNA Damage by Anticancer Drugs , 1998, The Journal of experimental medicine.
[31] S. Korsmeyer,et al. BID, a proapoptotic BCL-2 family member, is localized to mouse chromosome 6 and human chromosome 22q11. , 1998, Genomics.
[32] S. Cory,et al. The Bcl-2 protein family: arbiters of cell survival. , 1998, Science.
[33] Junying Yuan,et al. Cleavage of BID by Caspase 8 Mediates the Mitochondrial Damage in the Fas Pathway of Apoptosis , 1998, Cell.
[34] Xiaodong Wang,et al. Bid, a Bcl2 Interacting Protein, Mediates Cytochrome c Release from Mitochondria in Response to Activation of Cell Surface Death Receptors , 1998, Cell.
[35] M. Peter,et al. Two CD95 (APO‐1/Fas) signaling pathways , 1998, The EMBO journal.
[36] Y. Houvras,et al. Arrest of the cell cycle by the tumour-suppressor BRCA1 requires the CDK-inhibitor p21WAF1/CiPl , 1997, Nature.
[37] W. El-Deiry,et al. Detection and analysis of living, growth-inhibited mammalian cells following transfection. , 1997, BioTechniques.
[38] I. Krantz,et al. KILLER/DR5 is a DNA damage–inducible p53–regulated death receptor gene , 1997, Nature Genetics.
[39] C. Milliman,et al. BID: a novel BH3 domain-only death agonist. , 1996, Genes & development.
[40] A. Levine,et al. mdm-2 inhibits the G1 arrest and apoptosis functions of the p53 tumor suppressor protein , 1996, Molecular and cellular biology.
[41] W. El-Deiry,et al. Regulation of p21WAF1/CIP1 expression by p53-independent pathways. , 1996, Oncogene.
[42] D. Lane,et al. Coupling between gamma irradiation, p53 induction and the apoptotic response depends upon cell type in vivo. , 1995, Journal of cell science.
[43] D. Thorley-Lawson,et al. A novel form of Epstein-Barr virus latency in normal B cells in vivo , 1995, Cell.
[44] John Calvin Reed,et al. Tumor suppressor p53 is a direct transcriptional activator of the human bax gene , 1995, Cell.
[45] K. Kinzler,et al. Sequence-specific transcriptional activation is essential for growth suppression by p53. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[46] J. Trent,et al. WAF1, a potential mediator of p53 tumor suppression , 1993, Cell.
[47] Yi-Song Wang,et al. Reconstitution of wild-type p53 expression triggers apoptosis in a p53-negative v-myc retrovirus-induced T-cell lymphoma line. , 1993, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[48] Scott W. Lowe,et al. p53 is required for radiation-induced apoptosis in mouse thymocytes , 1993, Nature.
[49] K. Kinzler,et al. Definition of a consensus binding site for p53 , 1992, Nature Genetics.
[50] A. Levine,et al. p53 alteration is a common event in the spontaneous immortalization of primary BALB/c murine embryo fibroblasts. , 1991, Genes & development.