RESP18 is Involved in the Cytotoxicity of Dopaminergic Neurotoxins in MN9D Cells
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J. Xu | Mei Yu | Fang Huang | Yufang Huang | H. Suo | Minrui Liang | Jie Liu | Xiaoqi Hong | Jing Xu
[1] Mei Yu,et al. Overexpression of Parkin Ameliorates Dopaminergic Neurodegeneration Induced by 1- Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine in Mice , 2012, PloS one.
[2] H. Nishitoh. CHOP is a multifunctional transcription factor in the ER stress response. , 2012, Journal of biochemistry.
[3] David S. Park,et al. Parkin is transcriptionally regulated by ATF4: evidence for an interconnection between mitochondrial stress and ER stress , 2011, Cell Death and Differentiation.
[4] 江川 斉宏. The endoplasmic reticulum stress sensor, ATF6α, protects against neurotoxin-induced dopaminergic neuronal death , 2011 .
[5] Mei Yu,et al. Requirement of regulated endocrine-specific protein-18 for development and expression of regulated endocrine-specific protein-18 isoform c in mice , 2011, Molecular Biology Reports.
[6] K. Mori,et al. The Endoplasmic Reticulum Stress Sensor, ATF6α, Protects against Neurotoxin-induced Dopaminergic Neuronal Death* , 2010, The Journal of Biological Chemistry.
[7] T. Omura,et al. Meloxicam protects cell damage from 1-methyl-4-phenyl pyridinium toxicity via the phosphatidylinositol 3-kinase/Akt pathway in human dopaminergic neuroblastoma SH-SY5Y cells , 2010, Brain Research.
[8] Miao Wang,et al. Targeted Mutation of the Mouse Grp94 Gene Disrupts Development and Perturbs Endoplasmic Reticulum Stress Signaling , 2010, PloS one.
[9] M. Permutt,et al. AATF mediates anti-apoptotic effect of the unfolded protein response through transcriptional regulation of AKT1 , 2009, Cell Death and Differentiation.
[10] Mei Yu,et al. Alteration of NRSF expression exacerbating 1-methyl-4-phenyl-pyridinium ion-induced cell death of SH-SY5Y cells , 2009, Neuroscience Research.
[11] J. Segura-Aguilar,et al. Neurotoxins and neurotoxic species implicated in neurodegeneration , 2009, Neurotoxicity Research.
[12] Jia Luo,et al. GSK3beta and endoplasmic reticulum stress mediate rotenone-induced death of SK-N-MC neuroblastoma cells. , 2008, Biochemical pharmacology.
[13] T Tabira,et al. A molecular chaperone inducer protects neurons from ER stress , 2008, Cell Death and Differentiation.
[14] R. Sitia,et al. Protein quality control in the early secretory pathway , 2008, The EMBO journal.
[15] J. Roth,et al. Protein quality control: the who’s who, the where’s and therapeutic escapes , 2007, Histochemistry and Cell Biology.
[16] P. Dent,et al. The Kinase Inhibitor Sorafenib Induces Cell Death through a Process Involving Induction of Endoplasmic Reticulum Stress , 2007, Molecular and Cellular Biology.
[17] N. Belluardo,et al. Endoplasmic Reticulum Stress Inhibition Protects against Excitotoxic Neuronal Injury in the Rat Brain , 2007, The Journal of Neuroscience.
[18] M. Beal,et al. Mitochondrial dysfunction and oxidative stress in neurodegenerative diseases , 2006, Nature.
[19] D. Surmeier,et al. Differentiated Dopaminergic MN9D Cells Only Partially Recapitulate the Electrophysiological Properties of Midbrain Dopaminergic Neurons , 2006, Developmental Neuroscience.
[20] N. Wood,et al. Expanding insights of mitochondrial dysfunction in Parkinson's disease , 2006, Nature Reviews Neuroscience.
[21] Houeto Jean-Luc. [Parkinson's disease]. , 2022, La Revue du praticien.
[22] Kai Long,et al. A selective inhibitor of eIF2alpha dephosphorylation protects cells from ER stress. , 2005, Science.
[23] Keith Hyland,et al. Distinct Mechanisms of Neurodegeneration Induced by Chronic Complex I Inhibition in Dopaminergic and Non-dopaminergic Cells* , 2004, Journal of Biological Chemistry.
[24] L. Hendershot,et al. ER chaperone functions during normal and stress conditions , 2004, Journal of Chemical Neuroanatomy.
[25] Vladimir N. Uversky,et al. Neurotoxicant-induced animal models of Parkinson’s disease: understanding the role of rotenone, maneb and paraquat in neurodegeneration , 2004, Cell and Tissue Research.
[26] Joachim Klose,et al. Mitochondrial Dysfunction and Oxidative Damage in parkin-deficient Mice* , 2004, Journal of Biological Chemistry.
[27] S. Oyadomari,et al. Roles of CHOP/GADD153 in endoplasmic reticulum stress , 2004, Cell Death and Differentiation.
[28] W. Dauer,et al. Parkinson's Disease Mechanisms and Models , 2003, Neuron.
[29] Y. Oh,et al. Caspase-Dependent and -Independent Cell Death Pathways in Primary Cultures of Mesencephalic Dopaminergic Neurons after Neurotoxin Treatment , 2003, The Journal of Neuroscience.
[30] K. O’Malley,et al. Parkinsonian Mimetics Induce Aspects of Unfolded Protein Response in Death of Dopaminergic Neurons* , 2003, Journal of Biological Chemistry.
[31] K. Mori,et al. Distinct roles of activating transcription factor 6 (ATF6) and double-stranded RNA-activated protein kinase-like endoplasmic reticulum kinase (PERK) in transcription during the mammalian unfolded protein response. , 2002, The Biochemical journal.
[32] J. Bilsland,et al. Caspase Inhibitors Attenuate 1-Methyl-4-Phenylpyridinium Toxicity in Primary Cultures of Mesencephalic Dopaminergic Neurons , 2002, The Journal of Neuroscience.
[33] Stevan R. Hubbard,et al. IRE1 couples endoplasmic reticulum load to secretory capacity by processing the XBP-1 mRNA , 2002, Nature.
[34] N. Hattori,et al. An Unfolded Putative Transmembrane Polypeptide, which Can Lead to Endoplasmic Reticulum Stress, Is a Substrate of Parkin , 2001, Cell.
[35] X. Chen,et al. ER stress induces cleavage of membrane-bound ATF6 by the same proteases that process SREBPs. , 2000, Molecular cell.
[36] Shinsei Minoshima,et al. Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase , 2000, Nature Genetics.
[37] Anne Bertolotti,et al. Dynamic interaction of BiP and ER stress transducers in the unfolded-protein response , 2000, Nature Cell Biology.
[38] D. Ron,et al. Perk is essential for translational regulation and cell survival during the unfolded protein response. , 2000, Molecular cell.
[39] K. Mori,et al. Mammalian transcription factor ATF6 is synthesized as a transmembrane protein and activated by proteolysis in response to endoplasmic reticulum stress. , 1999, Molecular biology of the cell.
[40] Y. Oh,et al. Two distinct mechanisms are involved in 6‐hydroxydopamine‐ and MPP+‐induced dopaminergic neuronal cell death: Role of caspases, ROS, and JNK , 1999, Journal of neuroscience research.
[41] R. Elble. Animal Models of Action Tremor , 2008, Movement disorders : official journal of the Movement Disorder Society.
[42] A. Lang,et al. Parkinson's disease. First of two parts. , 1998, The New England journal of medicine.
[43] Xiaozhong Wang,et al. CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum. , 1998, Genes & development.
[44] S. Przedborski,et al. Mechanisms of MPTP toxicity. , 1998, Movement disorders : official journal of the Movement Disorder Society.
[45] M. Schiller,et al. A novel neuroendocrine intracellular signaling pathway. , 1997, Molecular endocrinology.
[46] M. Schiller,et al. Expression of RESP18 in Peptidergic and Catecholaminergic Neurons , 1997, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[47] M. Youdim,et al. Mechanism of 6-hydroxydopamine neurotoxicity. , 1997, Journal of neural transmission. Supplementum.
[48] Xiaozhong Wang,et al. Signals from the stressed endoplasmic reticulum induce C/EBP-homologous protein (CHOP/GADD153) , 1996, Molecular and cellular biology.
[49] P. Walter,et al. Oligomerization and phosphorylation of the Ire1p kinase during intracellular signaling from the endoplasmic reticulum to the nucleus. , 1996, The EMBO journal.
[50] R. Mains,et al. Location of neurons that express regulated endocrine-specific protein-18 in the rat diencephalon , 1996, Neuroscience.
[51] M. Schiller,et al. A Neuroendocrine-specific Protein Localized to the Endoplasmic Reticulum by Distal Degradation (*) , 1995, The Journal of Biological Chemistry.
[52] A. Fox,et al. Immortalization of embryonic mesencephalic dopaminergic neurons by somatic cell fusion , 1991, Brain Research.
[53] H J Gundersen,et al. The absolute number of nerve cells in substantia nigra in normal subjects and in patients with Parkinson's disease estimated with an unbiased stereological method. , 1991, Journal of neurology, neurosurgery, and psychiatry.