Lipopolysaccharide pretreatment increases protease-activated receptor-2 expression and monocyte chemoattractant protein-1 secretion in vascular endothelial cells

[1]  D. Weibel,et al.  Bacterial Cell Mechanics. , 2017, Biochemistry.

[2]  Dongye Li,et al.  Lipopolysaccharide induced vascular smooth muscle cells proliferation: A new potential therapeutic target for proliferative vascular diseases , 2017, Cell proliferation.

[3]  Wei-jian Zhang,et al.  Quercetin inhibits LPS-induced adhesion molecule expression and oxidant production in human aortic endothelial cells by p38-mediated Nrf2 activation and antioxidant enzyme induction , 2016, Redox biology.

[4]  S. Tucker,et al.  Protease-activated receptors (PARs)—biology and role in cancer invasion and metastasis , 2015, Cancer and Metastasis Reviews.

[5]  Xiaoming Yang,et al.  Activation of protease‑activated receptor‑2 is associated with increased expression of inflammatory factors in the adipose tissues of obese mice. , 2015, Molecular medicine reports.

[6]  Regina M. Krohn,et al.  Arsenic Exposure Increases Monocyte Adhesion to the Vascular Endothelium, a Pro-Atherogenic Mechanism , 2015, PloS one.

[7]  G. Dworacki,et al.  Circulating monocyte chemotactic protein 1 (MCP-1), vascular cell adhesion molecule 1 (VCAM-1) and angiogenin in type 2 diabetic patients treated with statins in low doses. , 2014, European journal of pharmacology.

[8]  Xu Li,et al.  Unfractionated heparin suppresses lipopolysaccharide-induced monocyte chemoattractant protein-1 expression in human microvascular endothelial cells by blocking Krüppel-like factor 5 and nuclear factor-κB pathway. , 2014, Immunobiology.

[9]  Arifur Rahman,et al.  Effects of protease activated receptor (PAR)2 blocking peptide on endothelin-1 levels in kidney tissues in endotoxemic rat mode. , 2014, Life sciences.

[10]  Huiyun Zhang,et al.  Evaluation on Potential Contributions of Protease Activated Receptors Related Mediators in Allergic Inflammation , 2014, Mediators of inflammation.

[11]  David W. Johnson,et al.  PAR2-induced inflammatory responses in human kidney tubular epithelial cells. , 2013, American journal of physiology. Renal physiology.

[12]  R. Meli,et al.  Cross‐talk between toll‐like receptor 4 (TLR4) and proteinase‐activated receptor 2 (PAR2) is involved in vascular function , 2013, British journal of pharmacology.

[13]  Guang-rong Gao,et al.  Protease-activated receptor-2 induces expression of vascular endothelial growth factor and cyclooxygenase-2 via the mitogen-activated protein kinase pathway in gastric cancer cells. , 2012, Oncology reports.

[14]  J. Panee.  Monocyte Chemoattractant Protein 1 (MCP-1) in obesity and diabetes. , 2012, Cytokine.

[15]  T. Cheng,et al.  Inhibition of voltage-gated K^+ channels and Ca^2+ channels by diphenidol , 2012, Pharmacological reports : PR.

[16]  W. Ouwehand,et al.  Identification of candidate genes linking systemic inflammation to atherosclerosis; results of a human in vivo LPS infusion study , 2011, BMC Medical Genomics.

[17]  N. Mackman,et al.  Protease‐activated receptors and myocardial infarction , 2011, IUBMB life.

[18]  Gyu Hwan Park,et al.  Essential role of mitogen-activated protein kinase pathways in protease activated receptor 2-mediated nitric-oxide production from rat primary astrocytes. , 2009, Nitric oxide : biology and chemistry.

[19]  S. Amini,et al.  Monocyte chemoattractant protein-1 (MCP-1): an overview. , 2009, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.

[20]  K. Ley,et al.  Immune and inflammatory mechanisms of atherosclerosis (*). , 2009, Annual review of immunology.

[21]  R. Ganju,et al.  LPS-induced MCP-1 expression in human microvascular endothelial cells is mediated by the tyrosine kinase, Pyk2 via the p38 MAPK/NF-kappaB-dependent pathway. , 2009, Molecular immunology.

[22]  Hui-Lung Sun,et al.  THE p38 MITOGEN-ACTIVATED PROTEIN KINASE PATHWAY PLAYS A CRITICAL ROLE IN PAR2-INDUCED ENDOTHELIAL IL-8 PRODUCTION AND LEUKOCYTE ADHESION , 2008, Shock.

[23]  G. Reiser,et al.  PAR-2 activation and LPS synergistically enhance inflammatory signaling in airway epithelial cells by raising PAR expression level and interleukin-8 release. , 2007, American journal of physiology. Lung cellular and molecular physiology.

[24]  Shaoheng He,et al.  Induction of monocyte chemoattractant protein-1 release from A549 cells by agonists of protease-activated receptor-1 and -2. , 2007, European journal of cell biology.

[25]  G. Cirino,et al.  Protease-activated receptor-2 (PAR2) in cardiovascular system. , 2005, Vascular pharmacology.

[26]  Gerene M Denning,et al.  Potential role of endotoxin as a proinflammatory mediator of atherosclerosis. , 2004, Arteriosclerosis, thrombosis, and vascular biology.

[27]  C. Napoli,et al.  Evidence that protease activated receptor 2 expression is enhanced in human coronary atherosclerotic lesions , 2004, Journal of Clinical Pathology.

[28]  S. Opal,et al.  Development of an anti-core lipopolysaccharide vaccine for the prevention and treatment of sepsis. , 2004, Vaccine.

[29]  W. Aird.  The role of the endothelium in severe sepsis and multiple organ dysfunction syndrome. , 2003, Blood.

[30]  C. Szabó,et al.  NHE blockade inhibits chemokine production and NF-kappaB activation in immunostimulated endothelial cells. , 2002, American journal of physiology. Cell physiology.

[31]  T. van der Poll,et al.  Anti-Inflammatory Effects of a p38 Mitogen-Activated Protein Kinase Inhibitor During Human Endotoxemia1 , 2002, The Journal of Immunology.

[32]  H. Arai,et al.  Distinct Signaling Pathways for MCP-1-dependent Integrin Activation and Chemotaxis* , 2001, The Journal of Biological Chemistry.

[33]  R. Berge,et al.  Interaction between chemokines and oxidative stress: possible pathogenic role in acute coronary syndromes. , 2001, Journal of the American College of Cardiology.

[34]  S. Anker,et al.  The endotoxin-lipoprotein hypothesis , 2000, The Lancet.

[35]  S. Opal,et al.  Relationship between plasma levels of lipopolysaccharide (LPS) and LPS-binding protein in patients with severe sepsis and septic shock. , 1999, The Journal of infectious diseases.

[36]  R. Sorrentino,et al.  Protease-activated receptor-2 involvement in hypotension in normal and endotoxemic rats in vivo. , 1999, Circulation.

[37]  J. Hamilton,et al.  Atypical protease-activated receptor mediates endothelium-dependent relaxation of human coronary arteries. , 1998, Circulation research.

[38]  J. Loscalzo,et al.  Endothelial cells in physiology and in the pathophysiology of vascular disorders. , 1998, Blood.

[39]  A. Luster,et al.  Chemokines--chemotactic cytokines that mediate inflammation. , 1998, The New England journal of medicine.

[40]  S. Coughlin,et al.  Molecular cloning, expression and potential functions of the human proteinase-activated receptor-2. , 1996, The Biochemical journal.

[41]  R. Busse,et al.  Calcium signaling in endothelial cells involves activation of tyrosine kinases and leads to activation of mitogen-activated protein kinases. , 1995, Circulation research.

[42]  ビー． キャロル，シーン,et al.  Prevention and treatment of sepsis , 1993 .

[43]  A. J. Valente,et al.  Purification of a monocyte chemotactic factor secreted by nonhuman primate vascular cells in culture. , 1988, Biochemistry.

[44]  W. Ruf,et al.  Protease-activated receptor 2 signaling in inflammation , 2011, Seminars in Immunopathology.

[45]  David B Sacks,et al.  Regulation of MAP kinase signaling by calcium. , 2010, Methods in molecular biology.

[46]  G. Hansson.  Atherosclerosis--an immune disease: The Anitschkov Lecture 2007. , 2009, Atherosclerosis.

[47]  A. Karsan,et al.  Lipopolysaccharide signaling in endothelial cells , 2006, Laboratory Investigation.

[48]  I-Chung Lo,et al.  Reactive oxygen species and ERK 1/2 mediate monocyte chemotactic protein-1-stimulated smooth muscle cell migration. , 2005, Journal of biomedical science.

[49]  T. Kanke,et al.  Physiology and pathophysiology of proteinase-activated receptors (PARs): PAR-2 as a potential therapeutic target. , 2005, Journal of pharmacological sciences.

[50]  A. Böyum,et al.  Isolation of mononuclear cells and granulocytes from human blood. Isolation of monuclear cells by one centrifugation, and of granulocytes by combining centrifugation and sedimentation at 1 g. , 1968, Scandinavian journal of clinical and laboratory investigation. Supplementum.