Cigarette smoke-induced DNA-damage: role of hydroquinone and catechol in the formation of the oxidative DNA-adduct, 8-hydroxydeoxyguanosine.
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[1] S. Weitzman,et al. Hydrogen peroxide induces squamous metaplasia in a hamster tracheal organ explant culture model. , 1989, Carcinogenesis.
[2] U. Stenius,et al. The role of GSH depletion and toxicity in hydroquinone-induced development of enzyme-altered foci. , 1989, Carcinogenesis.
[3] W. Pryor,et al. Quantitative analysis of the hydrogen peroxide formed in aqueous cigarette tar extracts. , 1989, Free radical biology & medicine.
[4] P. Leanderson,et al. Cigarette smoke potentiates the DNA-damaging effect of manmade mineral fibers. , 1989, American journal of industrial medicine.
[5] R. C. Garner,et al. Correlation of DNA adduct levels in human lung with cigarette smoking , 1988, Nature.
[6] W. Pryor. Why is the hydroxyl radical the only radical that commonly adds to DNA? Hypothesis: it has a rare combination of high electrophilicity, high thermochemical reactivity, and a mode of production that can occur near DNA. , 1988, Free radical biology & medicine.
[7] M. Aida,et al. An ab initio molecular orbital study on the characteristics of 8-hydroxyguanine. , 1987, Mutation research.
[8] H. Kasai,et al. Misreading of DNA templates containing 8-hydroxydeoxyguanosine at the modified base and at adjacent residues , 1987, Nature.
[9] R. Floyd,et al. Hydroxyl free radical adduct of deoxyguanosine: sensitive detection and mechanisms of formation. , 1986, Free radical research communications.
[10] B. J. Dean. Recent findings on the genetic toxicology of benzene, toluene, xylenes and phenols. , 1985, Mutation research.
[11] T. Nakayama,et al. Cigarette smoke induces DNA single-strand breaks in human cells , 1985, Nature.
[12] P. Cerutti. Prooxidant states and tumor promotion. , 1985, Science.
[13] W. Troll,et al. The role of oxygen radicals as a possible mechanism of tumor promotion. , 1985, Annual review of pharmacology and toxicology.
[14] S. Nishimura,et al. Hydroxylation of deoxyguanosine at the C-8 position by ascorbic acid and other reducing agents. , 1984, Nucleic acids research.
[15] T. Nakayama,et al. Generation of hydrogen peroxide and superoxide anion radical from cigarette smoke. , 1984, Gan.
[16] S. Wolff,et al. Induction of sister-chromatid exchanges in human lymphocytes by microsomal activation of benzene metabolites. , 1983, Mutation research.
[17] S. Hecht,et al. A study of tobacco carcinogenesis. XX. Role of catechol as a major cocarcinogen in the weakly acidic fraction of smoke condensate. , 1981, Journal of the National Cancer Institute.
[18] R. Doll. An epidemiological perspective of the biology of cancer. , 1978, Cancer research.
[19] A. Stoppani,et al. Evaluation of the horseradish peroxidase-scopoletin method for the measurement of hydrogen peroxide formation in biological systems. , 1977, Analytical biochemistry.
[20] B. L. Van Duuren,et al. Cocarcinogenic and tumor-promoting agents in tobacco carcinogenesis. , 1976, Journal of the National Cancer Institute.
[21] H. Hayatsu,et al. The permanganate oxidation of deoxyribonucleic acid , 1971 .
[22] E. Valentine. Squamous metaplasia of the bronchus. A study of metaplastic changes occurring in the epithelium of the major bronchi in cancerous and noncancerous cases , 1957, Cancer.