Elevated levels of active matrix metalloproteinase-9 cause hypertrophy in skeletal muscle of normal and dystrophin-deficient mdx mice.
暂无分享,去创建一个
Shephali Bhatnagar | Saurabh Dahiya | S. Kuang | S. Bhatnagar | Ashok Kumar | Chunhui Jiang | S. Hindi | P. Paul | S. Dahiya | Ashok Kumar | Shihuan Kuang | Sajedah M Hindi | Chunhui Jiang | Pradyut K Paul | Shephali Bhatnagar
[1] S. Trappe,et al. Time course of myogenic and metabolic gene expression in response to acute exercise in human skeletal muscle. , 2005, Journal of applied physiology.
[2] M. Rudnicki,et al. Molecular mechanisms regulating myogenic determination and differentiation. , 2000, Frontiers in bioscience : a journal and virtual library.
[3] A. Emery,et al. The muscular dystrophies , 2002, The Lancet.
[4] D. Glass,et al. Molecular mechanisms modulating muscle mass. , 2003, Trends in molecular medicine.
[5] N. Vayssiere,et al. Comparative evolution of muscular dystrophy in diaphragm, gastrocnemius and masseter muscles from old male mdx mice , 2004, Journal of Muscle Research & Cell Motility.
[6] A. Donnelly,et al. Skeletal muscle collagen content in humans after high-force eccentric contractions. , 2004, Journal of applied physiology.
[7] N. LeBrasseur,et al. Metabolic benefits of resistance training and fast glycolytic skeletal muscle. , 2011, American journal of physiology. Endocrinology and metabolism.
[8] S. Lachkar,et al. Expression of matrix metalloproteinases 2 and 9 in regenerating skeletal muscle: a study in experimentally injured and mdx muscles. , 1999, Developmental biology.
[9] Thiennu H. Vu,et al. Matrix metalloproteinases: effectors of development and normal physiology. , 2000, Genes & development.
[10] L. Matrisian,et al. Matrix metalloproteinases: they're not just for matrix anymore! , 2001, Current opinion in cell biology.
[11] D. Allen,et al. Matrix Metalloproteinase-9 Deficiency Results in Decreased Fiber Cross-Sectional Area and Alters Fiber Type Distribution in Mouse Hindlimb Skeletal Muscle , 2011, Cells Tissues Organs.
[12] D. Glass. Signaling pathways perturbing muscle mass , 2010, Current opinion in clinical nutrition and metabolic care.
[13] S. Bhatnagar,et al. Matrix metalloproteinase inhibitor batimastat alleviates pathology and improves skeletal muscle function in dystrophin-deficient mdx mice. , 2010, The American journal of pathology.
[14] S. Rhie,et al. Myogenic Akt signaling upregulates the utrophin–glycoprotein complex and promotes sarcolemma stability in muscular dystrophy , 2008, Human molecular genetics.
[15] T. Suuronen,et al. Type IV collagen and its degradation in paralyzed human muscle: Effect of functional electrical stimulation , 2000, Muscle & nerve.
[16] L. Kunkel,et al. Muscular dystrophies: genes to pathogenesis. , 2003, Current opinion in genetics & development.
[17] Michael Kjaer,et al. Role of extracellular matrix in adaptation of tendon and skeletal muscle to mechanical loading. , 2004, Physiological reviews.
[18] S. Powers,et al. High intensity exercise increases expression of matrix metalloproteinases in fast skeletal muscle fibres , 2005, Experimental physiology.
[19] D. Lockshon,et al. MyoD is a sequence-specific DNA binding protein requiring a region of myc homology to bind to the muscle creatine kinase enhancer , 1989, Cell.
[20] E. Carmeli,et al. Expression of matrix metalloproteinases, inhibitor, and acid phosphatase in muscles of immobilized hindlimbs of rats , 2003, Muscle & nerve.
[21] G. Yancopoulos,et al. Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo , 2001, Nature Cell Biology.
[22] A. Nakamura,et al. Up-regulation of mitogen activated protein kinases in mdx skeletal muscle following chronic treadmill exercise. , 2005, Biochimica et biophysica acta.
[23] N. Rosenthal,et al. Paired MyoD-binding sites regulate myosin light chain gene expression. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[24] E. Laird,et al. Engineering autoactivating forms of matrix metalloproteinase-9 and expression of the active enzyme in cultured cells and transgenic mouse brain. , 2002, Biochemistry.
[25] M. Rudnicki,et al. Asymmetric Self-Renewal and Commitment of Satellite Stem Cells in Muscle , 2007, Cell.
[26] F. Rivier,et al. Modulation of p38 mitogen-activated protein kinase cascade and metalloproteinase activity in diaphragm muscle in response to free radical scavenger administration in dystrophin-deficient Mdx mice. , 2007, The American journal of pathology.
[27] K. Campbell,et al. Animal models for muscular dystrophy: valuable tools for the development of therapies. , 2000, Human molecular genetics.
[28] P. Iversen,et al. Enhanced matrix metalloproteinase activity in skeletal muscles of rats with congestive heart failure. , 2005, American journal of physiology. Regulatory, integrative and comparative physiology.
[29] C. Dogra,et al. TNF‐related weak inducer of apoptosis (TWEAK) is a potent skeletal muscle‐wasting cytokine , 2007, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[30] Andrew J. Ewald,et al. Matrix metalloproteinases and the regulation of tissue remodelling , 2007, Nature Reviews Molecular Cell Biology.
[31] R. Carvalho,et al. Heart failure alters matrix metalloproteinase gene expression and activity in rat skeletal muscle , 2006, International journal of experimental pathology.
[32] J. Roma,et al. Evolution of pathological changes in the gastrocnemius of the mdx mice correlate with utrophin and β-dystroglycan expression , 2004, Acta Neuropathologica.
[33] J. Tidball,et al. A nitric oxide synthase transgene ameliorates muscular dystrophy in mdx mice , 2001, The Journal of cell biology.
[34] S. Bhatnagar,et al. Matrix metalloproteinase-9 inhibition ameliorates pathogenesis and improves skeletal muscle regeneration in muscular dystrophy. , 2009, Human molecular genetics.
[35] Randall W. Bryner,et al. Satellite cell proliferation is reduced in muscles of obese Zucker rats but restored with loading. , 2008, American journal of physiology. Cell physiology.
[36] A. Nakamura,et al. Activation and localization of matrix metalloproteinase-2 and -9 in the skeletal muscle of the muscular dystrophy dog (CXMDJ) , 2007, BMC musculoskeletal disorders.
[37] L. Mcleay,et al. Improved muscle healing through enhanced regeneration and reduced fibrosis in myostatin-null mice , 2005, Journal of Cell Science.
[38] Angela K. Peter,et al. Hypertrophic response of Duchenne and limb-girdle muscular dystrophies is associated with activation of Akt pathway. , 2006, Experimental cell research.
[39] S. Carpenter,et al. Duchenne muscular dystrophy: plasma membrane loss initiates muscle cell necrosis unless it is repaired. , 1979, Brain : a journal of neurology.
[40] C. Rommel,et al. Mediation of IGF-1-induced skeletal myotube hypertrophy by PI(3)K/Akt/mTOR and PI(3)K/Akt/GSK3 pathways , 2001, Nature Cell Biology.
[41] J. W. Von den Hoff,et al. Regulatory factors and cell populations involved in skeletal muscle regeneration , 2010, Journal of cellular physiology.
[42] I. Baumgartner,et al. Basement Membrane Remodeling in Skeletal Muscles of Patients with Limb Ischemia Involves Regulation of Matrix Metalloproteinases and Tissue Inhibitor of Matrix Metalloproteinases , 2007, Journal of Vascular Research.
[43] L. Kaczmarek,et al. β-Dystroglycan as a Target for MMP-9, in Response to Enhanced Neuronal Activity* , 2007, Journal of Biological Chemistry.
[44] D. Pette. Historical Perspectives: plasticity of mammalian skeletal muscle. , 2001, Journal of applied physiology.
[45] C. Dogra,et al. Regulation of phosphatidylinositol 3‐kinase (PI3K)/Akt and nuclear factor‐kappa B signaling pathways in dystrophin‐deficient skeletal muscle in response to mechanical stretch , 2006, Journal of cellular physiology.
[46] A. Nakamura,et al. Matrix metalloproteinase-2 ablation in dystrophin-deficient mdx muscles reduces angiogenesis resulting in impaired growth of regenerated muscle fibers. , 2011, Human molecular genetics.
[47] H. Blau,et al. Primary mouse myoblast purification, characterization, and transplantation for cell-mediated gene therapy , 1994, The Journal of cell biology.
[48] Thiennu H. Vu,et al. Matrix Metalloproteinase 9 and Vascular Endothelial Growth Factor Are Essential for Osteoclast Recruitment into Developing Long Bones , 2000, The Journal of cell biology.
[49] K. L. Gardner,et al. Interplay of IKK/NF-kappaB signaling in macrophages and myofibers promotes muscle degeneration in Duchenne muscular dystrophy. , 2007, The Journal of clinical investigation.
[50] D. Glass,et al. Skeletal muscle hypertrophy and atrophy signaling pathways. , 2005, The international journal of biochemistry & cell biology.
[51] A. N. Fullenkamp,et al. Research resource: estrogen-driven prolactin-mediated gene-expression networks in hormone-induced prostatic intraepithelial neoplasia. , 2010, Molecular endocrinology.
[52] J. D’Armiento,et al. Overexpression of MMP9 in macrophages attenuates pulmonary fibrosis induced by bleomycin. , 2007, The international journal of biochemistry & cell biology.
[53] G. Yancopoulos,et al. The IGF-1/PI3K/Akt pathway prevents expression of muscle atrophy-induced ubiquitin ligases by inhibiting FOXO transcription factors. , 2004, Molecular cell.
[54] David J. Kosek,et al. Efficacy of myonuclear addition may explain differential myofiber growth among resistance-trained young and older men and women. , 2006, American journal of physiology. Endocrinology and metabolism.
[55] W. Feeser,et al. Multiple sites of the propeptide region of human stromelysin-1 are required for maintaining a latent form of the enzyme. , 1994, The Journal of biological chemistry.
[56] Jialiang Hu,et al. Matrix metalloproteinase inhibitors as therapy for inflammatory and vascular diseases , 2007, Nature Reviews Drug Discovery.
[57] James J. Lee,et al. Major basic protein-1 promotes fibrosis of dystrophic muscle and attenuates the cellular immune response in muscular dystrophy. , 2008, Human molecular genetics.
[58] E. Raines,et al. Macrophage expression of active MMP-9 induces acute plaque disruption in apoE-deficient mice. , 2005, The Journal of clinical investigation.
[59] P. Eriksson,et al. A single bout of exercise activates matrix metalloproteinase in human skeletal muscle , 2007, Journal of applied physiology.
[60] Molecular mechanisms regulating myogenic determination and differentiation. , 2000 .
[61] H. Birkedal‐Hansen,et al. The cysteine switch: a principle of regulation of metalloproteinase activity with potential applicability to the entire matrix metalloproteinase gene family. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[62] P. Libby,et al. Targeted deletion of matrix metalloproteinase-9 attenuates left ventricular enlargement and collagen accumulation after experimental myocardial infarction. , 2000, The Journal of clinical investigation.
[63] H. Nagase,et al. Disruption of the cysteine-75 and zinc ion coordination is not sufficient to activate the precursor of human matrix metalloproteinase 3 (stromelysin 1). , 1993, Biochemistry.
[64] J. Petrella,et al. Potent myofiber hypertrophy during resistance training in humans is associated with satellite cell-mediated myonuclear addition: a cluster analysis. , 2008, Journal of applied physiology.
[65] J. Shrager,et al. The mdx mouse diaphragm reproduces the degenerative changes of Duchenne muscular dystrophy , 1991, Nature.
[66] L. Matrisian,et al. Mutational analysis of the transin (rat stromelysin) autoinhibitor region demonstrates a role for residues surrounding the "cysteine switch". , 1991, The Journal of biological chemistry.
[67] I. Nishino,et al. Proteolysis of β-dystroglycan in muscular diseases , 2005, Neuromuscular Disorders.
[68] J. Sanes. The Basement Membrane/Basal Lamina of Skeletal Muscle* , 2003, The Journal of Biological Chemistry.
[69] Hsin C. Lin,et al. Insulin-like Growth Factor-1 (IGF-1) Inversely Regulates Atrophy-induced Genes via the Phosphatidylinositol 3-Kinase/Akt/Mammalian Target of Rapamycin (PI3K/Akt/mTOR) Pathway* , 2005, Journal of Biological Chemistry.
[70] M. Fardeau,et al. Matrix metalloproteinases MMP‐2 and MMP‐9 in denervated muscle and injured nerve , 1998, Neuropathology and applied neurobiology.
[71] M. Rudnicki,et al. Distinct roles for Pax7 and Pax3 in adult regenerative myogenesis , 2006, The Journal of cell biology.
[72] Z. Werb,et al. Regulation of matrix biology by matrix metalloproteinases. , 2004, Current opinion in cell biology.
[73] M. Rudnicki,et al. Cellular and molecular regulation of muscle regeneration. , 2004, Physiological reviews.
[74] E. Carmeli,et al. Matrix metalloproteinases and skeletal muscle: A brief review , 2004, Muscle & nerve.