Cardiac Allograft Rejection Factor Accelerates Murine T Cell-Mediated Donor Deficiency of Decay-Accelerating Medof and

[1]  P. Heeger,et al.  Locally produced C5a binds to T cell-expressed C5aR to enhance effector T-cell expansion by limiting antigen-induced apoptosis. , 2008, Blood.

[2]  V. Tuohy,et al.  IFN-γ and IL-17 Production in Experimental Autoimmune Encephalomyelitis Depends on Local APC–T Cell Complement Production1 , 2008, The Journal of Immunology.

[3]  G. Dubyak,et al.  Locally produced complement fragments C5a and C3a provide both costimulatory and survival signals to naive CD4+ T cells. , 2008, Immunity.

[4]  P. Heeger,et al.  Decay Accelerating Factor Can Control T Cell Differentiation into IFN-γ-Producing Effector Cells via Regulating Local C5a-Induced IL-12 Production1 , 2007, The Journal of Immunology.

[5]  Hao Wang,et al.  Inhibition of Terminal Complement Components in Presensitized Transplant Recipients Prevents Antibody-Mediated Rejection Leading to Long-Term Graft Survival and Accommodation1 , 2007, The Journal of Immunology.

[6]  C. Fang,et al.  Complement-Dependent Enhancement of CD8+ T Cell Immunity to Lymphocytic Choriomeningitis Virus Infection in Decay-Accelerating Factor-Deficient Mice1 , 2007, The Journal of Immunology.

[7]  Devin R. Lindsey,et al.  Mycobacteria‐primed macrophages and dendritic cells induce an up‐regulation of complement C5a anaphylatoxin receptor (CD88) in CD3+ murine T cells , 2007, Journal of leukocyte biology.

[8]  R. Smith,et al.  Therapeutic strategy with a membrane-localizing complement regulator to increase the number of usable donor organs after prolonged cold storage. , 2006, Journal of the American Society of Nephrology : JASN.

[9]  Wuding Zhou,et al.  Macrophages from C3-deficient mice have impaired potency to stimulate alloreactive T cells. , 2006, Blood.

[10]  Wuding Zhou,et al.  Dendritic Cell Synthesis of C3 Is Required for Full T Cell Activation and Development of a Th1 Phenotype1 , 2006, The Journal of Immunology.

[11]  Wuding Zhou,et al.  Local Extravascular Pool of C3 Is a Determinant of Postischemic Acute Renal Failure , 2022 .

[12]  A. Valujskikh,et al.  Lymphoid Sequestration of Alloreactive Memory CD4 T Cells Promotes Cardiac Allograft Survival1 , 2006, The Journal of Immunology.

[13]  M. Sykes,et al.  Decay-Accelerating Factor Prevents Acute Humoral Rejection Induced by Low Levels of Anti-αGal Natural Antibodies , 2006, Transplantation.

[14]  A. Valujskikh,et al.  Decay-accelerating factor modulates induction of T cell immunity , 2005, The Journal of experimental medicine.

[15]  Hao Wang,et al.  Prevention of Acute Vascular Rejection by a Functionally Blocking Anti-C5 Monoclonal Antibody Combined with Cyclosporine , 2005, Transplantation.

[16]  J. Lambris,et al.  The complement inhibitory protein DAF (CD55) suppresses T cell immunity in vivo , 2005, The Journal of experimental medicine.

[17]  H. Schuurman,et al.  Transgenic expression in pig hearts of both human decay-accelerating factor and human membrane cofactor protein does not provide an additional benefit to that of human decay-accelerating factor alone in pig-to-baboon xenotransplantation. , 2004, Transplantation.

[18]  A. Valujskikh,et al.  In Vivo Helper Functions of Alloreactive Memory CD4+ T Cells Remain Intact Despite Donor-Specific Transfusion and Anti-CD40 Ligand Therapy1 , 2004, The Journal of Immunology.

[19]  A. Valujskikh,et al.  Antigen location contributes to the pathological features of a transplanted heart graft. , 2004, The American journal of pathology.

[20]  M. Nangaku,et al.  Critical Protection from Renal Ischemia Reperfusion Injury by CD55 and CD591 , 2004, The Journal of Immunology.

[21]  S. Schenk,et al.  Effects of T Cell Frequency and Graft Size on Transplant Outcome in Mice 1 , 2004, The Journal of Immunology.

[22]  John D Lambris,et al.  Complement Component 3 Is Required for Optimal Expansion of CD8 T Cells During a Systemic Viral Infection1 , 2003, The Journal of Immunology.

[23]  O. Lantz,et al.  Cross-primed CD8+ T cells mediate graft rejection via a distinct effector pathway , 2002, Nature Immunology.

[24]  S. Sacks,et al.  Local synthesis of complement component C3 regulates acute renal transplant rejection , 2002, Nature Medicine.

[25]  C. Harris,et al.  Tissue distribution of products of the mouse decay‐accelerating factor (DAF) genes. Exploitation of a Daf1 knock‐out mouse and site‐specific monoclonal antibodies , 2001, Immunology.

[26]  A. Valujskikh,et al.  Characterization and manipulation of T cell immunity to skin grafts expressing a transgenic minor antigen. , 1999, Transplantation.

[27]  A. Valujskikh,et al.  Contributions of direct and indirect T cell alloreactivity during allograft rejection in mice. , 1999, Journal of immunology.

[28]  A. Valujskikh,et al.  Type 2 immune deviation has differential effects on alloreactive CD4+ and CD8+ T cells. , 1998, Journal of immunology.

[29]  A. Singer,et al.  Cellular Interactions Resulting in Skin‐Allograft Rejection , 1988, Annals of the New York Academy of Sciences.

[30]  W. Roberts,et al.  Decay accelerating factor of complement is anchored to cells by a C-terminal glycolipid. , 1986, Biochemistry.

[31]  V. Nussenzweig,et al.  Inhibition of complement activation on the surface of cells after incorporation of decay-accelerating factor (DAF) into their membranes , 1984, The Journal of experimental medicine.

[32]  J. Markmann,et al.  Homeostatic proliferation is a barrier to transplantation tolerance , 2004, Nature Medicine.