Doxorubicin–Mediated miR–433 Expression on Exosomes Promotes Bystander Senescence in Multiple Myeloma Cells in a DDR–Independent Manner
暂无分享,去创建一个
G. Caracciolo | A. Santoni | M. Cippitelli | L. Masuelli | C. Fionda | F. Antonangeli | A. Soriani | A. Zingoni | E. Vulpis | Lorenzo Cuollo | C. Borrelli | Maria Teresa Petrucci
[1] Weixiong Yang,et al. Circ_0003489 facilitates multiple myeloma progression by targeting miR-433-3p/PBX3 axis , 2022, Hematology.
[2] G. Caracciolo,et al. Impact on NK cell functions of acute versus chronic exposure to extracellular vesicle‐associated MICA: Dual role in cancer immunosurveillance , 2022, Journal of extracellular vesicles.
[3] T. M. Zanotto,et al. MicroRNA sequence codes for small extracellular vesicle release and cellular retention , 2021, Nature.
[4] A. Vacca,et al. Role of Extracellular Vesicle-Based Cell-to-Cell Communication in Multiple Myeloma Progression , 2021, Cells.
[5] N. Giannakoulas,et al. The Role of Marrow Microenvironment in the Growth and Development of Malignant Plasma Cells in Multiple Myeloma , 2021, International journal of molecular sciences.
[6] S. Stewart,et al. Unmasking senescence: context-dependent effects of SASP in cancer , 2019, Nature Reviews Cancer.
[7] K. Crasta,et al. Exosomes as Emerging Pro-Tumorigenic Mediators of the Senescence-Associated Secretory Phenotype , 2019, International journal of molecular sciences.
[8] S. Kristensen,et al. Extracellular vesicle-associated procoagulant phospholipid and tissue factor activity in multiple myeloma , 2019, PloS one.
[9] Teng Liu,et al. Tumor-derived extracellular vesicles inhibit osteogenesis and exacerbate myeloma bone disease , 2019, Theranostics.
[10] N. Suh. MicroRNA controls of cellular senescence , 2018, BMB reports.
[11] A. Santoni,et al. Drug-Induced Senescent Multiple Myeloma Cells Elicit NK Cell Proliferation by Direct or Exosome-Mediated IL15 Trans-Presentation , 2018, Cancer Immunology Research.
[12] M. Takasugi. Emerging roles of extracellular vesicles in cellular senescence and aging , 2018, Aging cell.
[13] G. Nelson,et al. The senescent bystander effect is caused by ROS-activated NF-κB signalling , 2017, Mechanisms of Ageing and Development.
[14] E. Hara,et al. Small extracellular vesicles secreted from senescent cells promote cancer cell proliferation through EphA2 , 2017, Nature Communications.
[15] G. Caracciolo,et al. Genotoxic stress modulates the release of exosomes from multiple myeloma cells capable of activating NK cell cytokine production: Role of HSP70/TLR2/NF-kB axis , 2017, Oncoimmunology.
[16] R. Silvestri,et al. p38 MAPK differentially controls NK activating ligands at transcriptional and post-transcriptional level on multiple myeloma cells , 2017, Oncoimmunology.
[17] Qiong Wang,et al. MiR-433-3p suppresses cell growth and enhances chemosensitivity by targeting CREB in human glioma , 2016, Oncotarget.
[18] G. Bernardini,et al. Natural killer cell recognition of in vivo drug-induced senescent multiple myeloma cells , 2016, Oncoimmunology.
[19] R. Westendorp,et al. Secreted microvesicular miR‐31 inhibits osteogenic differentiation of mesenchymal stem cells , 2016, Aging cell.
[20] F. Sánchez‐Madrid,et al. Immunomodulatory role of microRNAs transferred by extracellular vesicles , 2015, Biology of the cell.
[21] J. O’Sullivan,et al. Overexpression of the microRNA miR-433 promotes resistance to paclitaxel through the induction of cellular senescence in ovarian cancer cells , 2015, Cancer medicine.
[22] Tianzhi Yang,et al. Exosome Delivered Anticancer Drugs Across the Blood-Brain Barrier for Brain Cancer Therapy in Danio Rerio , 2015, Pharmaceutical Research.
[23] A. Santoni,et al. Reactive Oxygen Species– and DNA Damage Response–Dependent NK Cell Activating Ligand Upregulation Occurs at Transcriptional Levels and Requires the Transcriptional Factor E2F1 , 2014, The Journal of Immunology.
[24] F. Sánchez‐Madrid,et al. Sumoylated hnRNPA2B1 controls the sorting of miRNAs into exosomes through binding to specific motifs , 2013, Nature Communications.
[25] A. Santoni,et al. Chemotherapy-elicited upregulation of NKG2D and DNAM-1 ligands as a therapeutic target in multiple myeloma , 2013, Oncoimmunology.
[26] S. Lowe,et al. p53-dependent chemokine production by senescent tumor cells supports NKG2D-dependent tumor elimination by natural killer cells , 2013, The Journal of experimental medicine.
[27] Zhihong Yang,et al. MicroRNA-433 Inhibits Liver Cancer Cell Migration by Repressing the Protein Expression and Function of cAMP Response Element-binding Protein* , 2013, The Journal of Biological Chemistry.
[28] F. Wang,et al. The Tumor Suppressor Roles of miR-433 and miR-127 in Gastric Cancer , 2013, International journal of molecular sciences.
[29] D. Scadden,et al. BM mesenchymal stromal cell-derived exosomes facilitate multiple myeloma progression. , 2013, The Journal of clinical investigation.
[30] H. Tahara,et al. The role of exosomes and microRNAs in senescence and aging. , 2013, Advanced drug delivery reviews.
[31] Graça Raposo,et al. Extracellular vesicles: Exosomes, microvesicles, and friends , 2013, The Journal of cell biology.
[32] Judith Campisi,et al. Senescent cells as a source of inflammatory factors for tumor progression , 2010, Cancer and Metastasis Reviews.
[33] J. Campisi,et al. The senescence-associated secretory phenotype: the dark side of tumor suppression. , 2010, Annual review of pathology.
[34] J. Campisi,et al. Protocols to detect senescence-associated beta-galactosidase (SA-βgal) activity, a biomarker of senescent cells in culture and in vivo , 2009, Nature Protocols.
[35] R. Foà,et al. ATM-ATR-dependent up-regulation of DNAM-1 and NKG2D ligands on multiple myeloma cells by therapeutic agents results in enhanced NK-cell susceptibility and is associated with a senescent phenotype. , 2009, Blood.
[36] Judith Campisi,et al. Senescence-Associated Secretory Phenotypes Reveal Cell-Nonautonomous Functions of Oncogenic RAS and the p53 Tumor Suppressor , 2008, PLoS biology.
[37] D. Terrian,et al. Senescence-associated exosome release from human prostate cancer cells. , 2008, Cancer research.
[38] S. Lowe,et al. Senescence of Activated Stellate Cells Limits Liver Fibrosis , 2008, Cell.
[39] C. Schmitt. Senescence, apoptosis and therapy — cutting the lifelines of cancer , 2003, Nature Reviews Cancer.
[40] A. Palumbo,et al. Multiple myeloma. , 2011, The New England journal of medicine.