Transcriptome analysis of human brain tissue identifies reduced expression of complement complex C1Q Genes in Rett syndrome
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Richard J. Edwards | Amelia A. Assareh | A. Assareh | I. Voineagu | Peijie Lin | Hassan Assareh | Z. Fang | T. Amos | Laura Nicholls | H. Assareh
[1] Uday Kishore,et al. Emerging and Novel Functions of Complement Protein C1q , 2015, Front. Immunol..
[2] Michael J. Yetman,et al. Wild-type microglia do not reverse pathology in mouse models of Rett syndrome , 2015, Nature.
[3] Steffen Jung,et al. Methyl-CpG Binding Protein 2 Regulates Microglia and Macrophage Gene Expression in Response to Inflammatory Stimuli. , 2015, Immunity.
[4] Harrison W. Gabel,et al. Disruption of DNA methylation-dependent long gene repression in Rett syndrome , 2015, Nature.
[5] Matthew E. Ritchie,et al. limma powers differential expression analyses for RNA-sequencing and microarray studies , 2015, Nucleic acids research.
[6] L. Pozzo-Miller,et al. Dendritic spine dysgenesis in Rett syndrome , 2014, Front. Neuroanat..
[7] S. Dudoit,et al. Normalization of RNA-seq data using factor analysis of control genes or samples , 2014, Nature Biotechnology.
[8] David P. Kreil,et al. The concordance between RNA-seq and microarray data depends on chemical treatment and transcript abundance , 2014, Nature Biotechnology.
[9] Björn Usadel,et al. Trimmomatic: a flexible trimmer for Illumina sequence data , 2014, Bioinform..
[10] G. Ananiev,et al. Mutant astrocytes differentiated from Rett syndrome patients-specific iPSCs have adverse effects on wild-type neurons. , 2014, Human molecular genetics.
[11] Wei Shi,et al. featureCounts: an efficient general purpose program for assigning sequence reads to genomic features , 2013, Bioinform..
[12] Beth Stevens,et al. TGF-β Signaling Regulates Neuronal C1q Expression and Developmental Synaptic Refinement , 2013, Nature Neuroscience.
[13] Renaud Gaujoux,et al. CellMix: a comprehensive toolbox for gene expression deconvolution , 2013, Bioinform..
[14] Johann A. Gagnon-Bartsch,et al. Using control genes to correct for unwanted variation in microarray data. , 2012, Biostatistics.
[15] Chris Williams,et al. RNA-SeQC: RNA-seq metrics for quality control and process optimization , 2012, Bioinform..
[16] James C. Cronk,et al. Wild type microglia arrest pathology in a mouse model of Rett syndrome , 2012, Nature.
[17] J. Leek,et al. Temporal dynamics and genetic control of transcription in the human prefrontal cortex , 2011, Nature.
[18] R. Veerhuis,et al. Complement in the brain. , 2011, Molecular immunology.
[19] J. Raber,et al. A role for glia in the progression of Rett’s syndrome , 2011, Nature.
[20] Rodney C. Samaco,et al. Complexities of Rett Syndrome and MeCP2 , 2011, The Journal of Neuroscience.
[21] Xiangqin Cui,et al. Design and validation issues in RNA-seq experiments , 2011, Briefings Bioinform..
[22] Robert S. Illingworth,et al. Neuronal MeCP2 is expressed at near histone-octamer levels and globally alters the chromatin state. , 2010, Molecular cell.
[23] Mark D. Robinson,et al. edgeR: a Bioconductor package for differential expression analysis of digital gene expression data , 2009, Bioinform..
[24] J. Christodoulou,et al. Downstream targets of methyl CpG binding protein 2 and their abnormal expression in the frontal cortex of the human Rett syndrome brain , 2010, BMC Neuroscience.
[25] Matthew D. Young,et al. Gene ontology analysis for RNA-seq: accounting for selection bias , 2010, Genome Biology.
[26] Christina Thaller,et al. Mouse models of MeCP2 disorders share gene expression changes in the cerebellum and hypothalamus , 2009, Human molecular genetics.
[27] Lior Pachter,et al. Sequence Analysis , 2020, Definitions.
[28] Pan Du,et al. lumi: a pipeline for processing Illumina microarray , 2008, Bioinform..
[29] Stephen T. C. Wong,et al. MeCP2, a Key Contributor to Neurological Disease, Activates and Represses Transcription , 2008, Science.
[30] Y. Xing,et al. A Transcriptome Database for Astrocytes, Neurons, and Oligodendrocytes: A New Resource for Understanding Brain Development and Function , 2008, The Journal of Neuroscience.
[31] Jonathan Pevsner,et al. FXYD1 is an MeCP2 target gene overexpressed in the brains of Rett syndrome patients and Mecp2-null mice. , 2007, Human molecular genetics.
[32] K. Shiota,et al. Methyl-CpG-binding Protein, MeCP2, Is a Target Molecule for Maintenance DNA Methyltransferase, Dnmt1* , 2003, The Journal of Biological Chemistry.
[33] Eric P. Hoffman,et al. Gene Expression Profiling in Postmortem Rett Syndrome Brain: Differential Gene Expression and Patient Classification , 2001, Neurobiology of Disease.
[34] H. Zoghbi,et al. Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2 , 1999, Nature Genetics.
[35] Colin A. Johnson,et al. Transcriptional repression by the methyl-CpG-binding protein MeCP2 involves a histone deacetylase complex , 1998, Nature.