MCP-1 chemokine receptor CCR2 is decreased on circulating monocytes in sporadic amyotrophic lateral sclerosis (sALS)

[1]  M. Parmentier,et al.  Extracellular adenine nucleotides inhibit the release of major monocyte recruiters by human monocyte‐derived dendritic cells , 2006, FEBS letters.

[2]  Matthias Mack,et al.  Modulating CCR2 and CCL2 at the blood-brain barrier: relevance for multiple sclerosis pathogenesis. , 2006, Brain : a journal of neurology.

[3]  N. Bresolin,et al.  Production of monocyte chemoattractant protein‐1 in amyotrophic lateral sclerosis , 2005, Muscle & nerve.

[4]  Beatriz León,et al.  Monocyte-derived dendritic cells. , 2005, Seminars in immunology.

[5]  L. Minghetti.  Role of inflammation in neurodegenerative diseases , 2005, Current opinion in neurology.

[6]  R. Keep,et al.  Monocyte Chemoattractant Protein-1 Regulation of Blood–Brain Barrier Permeability , 2005, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[7]  T. Owens,et al.  Cytokine and chemokine inter-regulation in the inflamed or injured CNS , 2005, Brain Research Reviews.

[8]  M. McGrath,et al.  Evidence for systemic immune system alterations in sporadic amyotrophic lateral sclerosis (sALS) , 2005, Journal of Neuroimmunology.

[9]  N. Bresolin,et al.  MCP-1 in Alzheimer’s disease patients: A-2518G polymorphism and serum levels , 2004, Neurobiology of Aging.

[10]  B. Chesebro,et al.  MCP-1 and CCR2 Contribute to Non-Lymphocyte-Mediated Brain Disease Induced by Fr98 Polytropic Retrovirus Infection in Mice: Role for Astrocytes in Retroviral Neuropathogenesis , 2004, Journal of Virology.

[11]  S. Appel,et al.  Increased lipid peroxidation in sera of ALS patients , 2004, Neurology.

[12]  F. Barkhof,et al.  Monocyte activation and disease activity in multiple sclerosis. A longitudinal analysis of serum MRP8/14 levels , 2004, Journal of Neuroimmunology.

[13]  T. Siddique,et al.  Presence of dendritic cells, MCP‐1, and activated microglia/macrophages in amyotrophic lateral sclerosis spinal cord tissue , 2004, Annals of neurology.

[14]  H. Vinters,et al.  Inflammation in amyotrophic lateral sclerosis spinal cord and brain is mediated by activated macrophages, mast cells and T cells , 2004, Amyotrophic lateral sclerosis and other motor neuron disorders : official publication of the World Federation of Neurology, Research Group on Motor Neuron Diseases.

[15]  M. Duddy,et al.  Analyses of all matrix metalloproteinase members in leukocytes emphasize monocytes as major inflammatory mediators in multiple sclerosis. , 2003, Brain : a journal of neurology.

[16]  R. Keep,et al.  Potential role of MCP-1 in endothelial cell tight junction `opening': signaling via Rho and Rho kinase , 2003, Journal of Cell Science.

[17]  G. Deuschl,et al.  Intrathecal synthesis of monocyte chemoattractant protein-1 (MCP-1) in amyotrophic lateral sclerosis: further evidence for microglial activation in neurodegeneration , 2003, Journal of Neuroimmunology.

[18]  G. Davies-Jones,et al.  Cytokine regulation of MCP-1 expression in brain and retinal microvascular endothelial cells , 2003, Journal of Neuroimmunology.

[19]  J. Mudgett,et al.  Impaired neuropathic pain responses in mice lacking the chemokine receptor CCR2 , 2003, Proceedings of the National Academy of Sciences of the United States of America.

[20]  E. Hirsch,et al.  The Role of Glial Reaction and Inflammation in Parkinson's Disease , 2003, Annals of the New York Academy of Sciences.

[21]  P. Mcgeer,et al.  Inflammatory processes in amyotrophic lateral sclerosis , 2002, Muscle & nerve.

[22]  T. Espevik,et al.  The Proinflammatory CD14+CD16+DR++ Monocytes Are a Major Source of TNF1 , 2002, The Journal of Immunology.

[23]  T. Smirnova,et al.  Recruitment of dendritic cells to the cerebrospinal fluid in bacterial neuroinfections , 2002, Journal of Neuroimmunology.

[24]  S. Appel,et al.  Immune reactivity in a mouse model of familial ALS correlates with disease progression , 2001, Neurology.

[25]  B. Rollins,et al.  Absence of Monocyte Chemoattractant Protein 1 in Mice Leads to Decreased Local Macrophage Recruitment and Antigen-Specific T Helper Cell Type 1 Immune Response in Experimental Autoimmune Encephalomyelitis , 2001, The Journal of experimental medicine.

[26]  Deborah F. Gelanis.  Respiratory failure or impairment in amyotrophic lateral sclerosis , 2001, Current treatment options in neurology.

[27]  M. Graeber,et al.  Neuronal MCP-1 Expression in Response to Remote Nerve Injury , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[28]  W. Brück,et al.  The chemokine receptor CCR2 is involved in macrophage recruitment to the injured peripheral nervous system , 2000, Journal of Neuroimmunology.

[29]  W. Kuziel,et al.  Cc Chemokine Receptor 2 Is Critical for Induction of Experimental Autoimmune Encephalomyelitis , 2000, The Journal of experimental medicine.

[30]  C. Plata-salamán,et al.  Inflammation and Alzheimer’s disease , 2000, Neurobiology of Aging.

[31]  C. Weber,et al.  Differential chemokine receptor expression and function in human monocyte subpopulations , 2000, Journal of leukocyte biology.

[32]  J. Cedarbaum,et al.  The ALSFRS-R: a revised ALS functional rating scale that incorporates assessments of respiratory function , 1999, Journal of the Neurological Sciences.

[33]  G. Opdenakker,et al.  The MCP/eotaxin subfamily of CC chemokines. , 1999, Cytokine & growth factor reviews.

[34]  R. Maki,et al.  Chemokines and chemokine receptors in the CNS: a possible role in neuroinflammation and patterning. , 1999, Trends in pharmacological sciences.

[35]  B. Rollins,et al.  Monocyte chemoattractant protein-1. , 1999, Chemical immunology.

[36]  R. Strieter,et al.  Acute and relapsing experimental autoimmune encephalomyelitis are regulated by differential expression of the CC chemokines macrophage inflammatory protein-1α and monocyte chemotactic protein-1 , 1998, Journal of Neuroimmunology.

[37]  P. Mcgeer,et al.  The importance of inflammatory mechanisms in alzheimer disease , 1998, Experimental Gerontology.

[38]  C. Brosnan,et al.  MCP-1, MCP-2 and MCP-3 expression in multiple sclerosis lesions: an immunohistochemical and in situ hybridization study , 1998, Journal of Neuroimmunology.

[39]  B. Rollins,et al.  Abnormalities in Monocyte Recruitment and Cytokine Expression in Monocyte Chemoattractant Protein 1–deficient Mice , 1998, The Journal of experimental medicine.

[40]  Robert V Farese,et al.  Impaired monocyte migration and reduced type 1 (Th1) cytokine responses in C-C chemokine receptor 2 knockout mice. , 1997, The Journal of clinical investigation.

[41]  T. Miyakawa,et al.  Identification of monocyte chemoattractant protein‐1 in senile plaques and reactive microglia of Alzheimer's disease , 1997, Psychiatry and clinical neurosciences.

[42]  K. Murao,et al.  Regulation of Expression of the Human Monocyte Chemotactic Protein-1 Receptor (hCCR2) by Cytokines* , 1997, The Journal of Biological Chemistry.

[43]  T. Kurihara,et al.  Cloning and Functional Expression of mCCR2, a Murine Receptor for the C-C Chemokines JE and FIC (*) , 1996, The Journal of Biological Chemistry.

[44]  I. Charo,et al.  Molecular cloning and functional expression of murine JE (monocyte chemoattractant protein 1) and murine macrophage inflammatory protein 1alpha receptors: evidence for two closely linked C-C chemokine receptors on chromosome 9. , 1996, The Journal of biological chemistry.

[45]  T. Sternsdorf,et al.  Differential cytokine expression in human blood monocyte subpopulations: a polymerase chain reaction analysis. , 1996, Blood.

[46]  B. Brooks,et al.  El escorial World Federation of Neurology criteria for the diagnosis of amyotrophic lateral sclerosis , 1994, Journal of the Neurological Sciences.

[47]  S. Coughlin,et al.  Molecular cloning and functional expression of two monocyte chemoattractant protein 1 receptors reveals alternative splicing of the carboxyl-terminal tails. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[48]  B. Passlick,et al.  The novel subset of CD14+/CD16+ blood monocytes exhibits features of tissue macrophages , 1993, European journal of immunology.

[49]  Mulder Dw.  Clinical limits of amyotrophic lateral sclerosis. , 1982 .