Expression of extracellular matrix genes during myocardial recovery from heart failure after left ventricular assist device support.

[1]  Francis G Spinale,et al.  Myocardial matrix remodeling and the matrix metalloproteinases: influence on cardiac form and function. , 2007, Physiological reviews.

[2]  T. Koide.  Designed triple-helical peptides as tools for collagen biochemistry and matrix engineering , 2007, Philosophical Transactions of the Royal Society B: Biological Sciences.

[3]  N. Rosenthal,et al.  Enhancing Repair of the Mammalian Heart , 2007, Circulation research.

[4]  O H Frazier,et al.  Cardiac Improvement During Mechanical Circulatory Support: A Prospective Multicenter Study of the LVAD Working Group , 2007, Circulation.

[5]  N. Rosenthal,et al.  Local expression of IGF‐1 accelerates muscle regeneration by rapidly modulating inflammatory cytokines and chemokines , 2007, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[6]  D. Burkhoff,et al.  The impact of angiotensin-converting enzyme inhibitor therapy on the extracellular collagen matrix during left ventricular assist device support in patients with end-stage heart failure. , 2007, Journal of the American College of Cardiology.

[7]  Magdi H Yacoub,et al.  A quantitative gene expression profile of matrix metalloproteinases (MMPS) and their inhibitors (TIMPS) in the myocardium of patients with deteriorating heart failure requiring left ventricular assist device support. , 2006, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.

[8]  N. de Jonge,et al.  Reverse remodeling of the myocardial extracellular matrix after prolonged left ventricular assist device support follows a biphasic pattern. , 2006, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.

[9]  R. Körfer,et al.  Influence of ACE‐inhibition and mechanical unloading on the regulation of extracellular matrix proteins in the myocardium of heart transplantation candidates bridged by ventricular assist devices , 2006, European journal of heart failure.

[10]  N. de Jonge,et al.  Brain natriuretic peptide is produced both by cardiomyocytes and cells infiltrating the heart in patients with severe heart failure supported by a left ventricular assist device. , 2006, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.

[11]  S. Chillcott,et al.  Assessment of recurrent heart failure associated with left ventricular assist device dysfunction. , 2005, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.

[12]  M. Yacoub,et al.  Myocardial Insulin-Like Growth Factor-I Gene Expression During Recovery From Heart Failure After Combined Left Ventricular Assist Device and Clenbuterol Therapy , 2005, Circulation.

[13]  R. Hetzer,et al.  Long-Term Results in Patients With Idiopathic Dilated Cardiomyopathy After Weaning From Left Ventricular Assist Devices , 2005, Circulation.

[14]  D. Burkhoff,et al.  Mechanical unloading during left ventricular assist device support increases left ventricular collagen cross-linking and myocardial stiffness. , 2005, Circulation.

[15]  Xinqiang Han,et al.  Genomic profiling of the human heart before and after mechanical support with a ventricular assist device reveals alterations in vascular signaling networks. , 2004, Physiological genomics.

[16]  A. Khaghani,et al.  Clinical Recovery From End-Stage Heart Failure Using Left-Ventricular Assist Device and Pharmacological Therapy Correlates With Increased Sarcoplasmic Reticulum Calcium Content but Not With Regression of Cellular Hypertrophy , 2004, Circulation.

[17]  E. Vuorio,et al.  Characterization of the murine Timp4 gene, localization within intron 5 of the synapsin 2 gene and tissue distribution of the mRNA. , 2002, Biochimica et biophysica acta.

[18]  A. Feldman,et al.  Downregulation of Matrix Metalloproteinases and Reduction in Collagen Damage in the Failing Human Heart After Support With Left Ventricular Assist Devices , 2001, Circulation.

[19]  D. Burkhoff,et al.  Time course of reverse remodeling of the left ventricle during support with a left ventricular assist device. , 2001, The Journal of thoracic and cardiovascular surgery.

[20]  G. Noon,et al.  Regression of fibrosis and hypertrophy in failing myocardium following mechanical circulatory support. , 2001, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.

[21]  M. Yacoub.  A novel strategy to maximize the efficacy of left ventricular assist devices as a bridge to recovery. , 2001, European heart journal.

[22]  M Flannery,et al.  Low incidence of myocardial recovery after left ventricular assist device implantation in patients with chronic heart failure. , 1998, Circulation.

[23]  H Harasaki,et al.  Left ventricular echocardiographic and histologic changes: impact of chronic unloading by an implantable ventricular assist device. , 1996, Journal of the American College of Cardiology.

[24]  W. Frishman,et al.  Left Ventricular Assist Device and Drug Therapy for the Reversal of Heart Failure , 2007 .

[25]  P. Barton,et al.  Real-time quantitative polymerase chain reaction in cardiac transplant research. , 2006, Methods in molecular biology.

[26]  M. Oz,et al.  Mechanical unloading leads to echocardiographic, electrocardiographic, neurohormonal, and histologic recovery. , 2006, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.

[27]  M. Loebe,et al.  Plasma neurohormone levels correlate with left ventricular functional and morphological improvement in LVAD patients. , 2005, The Journal of surgical research.

[28]  S. Houser,et al.  Sarcomeric genes involved in reverse remodeling of the heart during left ventricular assist device support. , 2005, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.

[29]  M. Loebe,et al.  Degree of cardiac fibrosis and hypertrophy at time of implantation predicts myocardial improvement during left ventricular assist device support. , 2004, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.