Dual role for CXCR3 and CCR5 in asthmatic Type 1 inflammation.

[1]  N. Aghaeepour,et al.  High-dimensional profiling clusters asthma severity by lymphoid and non-lymphoid status , 2021, Cell reports.

[2]  A. Ray,et al.  Immune responses and exacerbations in severe asthma. , 2021, Current opinion in immunology.

[3]  S. Ratcliffe,et al.  Human Th1 and Th2 Cells Targeting Rhinovirus and Allergen Coordinately Promote Allergic Asthma. , 2020, The Journal of allergy and clinical immunology.

[4]  Joakim S. Dahlin,et al.  Localization-Specific Expression of CCR1 and CCR5 by Mast Cell Progenitors , 2020, Frontiers in Immunology.

[5]  G. Pejler The emerging role of mast cell proteases in asthma , 2019, European Respiratory Journal.

[6]  A. Kanda,et al.  Critical role of CCL4 in eosinophil recruitment into the airway , 2019, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

[7]  Tonya S. King,et al.  Mometasone or Tiotropium in Mild Asthma with a Low Sputum Eosinophil Level. , 2019, The New England journal of medicine.

[8]  J. Krings,et al.  Role of Biologics in Asthma , 2019, American journal of respiratory and critical care medicine.

[9]  T. Nurmagambetov,et al.  The Economic Burden of Asthma in the United States, 2008‐2013 , 2018, Annals of the American Thoracic Society.

[10]  J. Blanco,et al.  Maraviroc improves hepatic triglyceride content but not inflammation in a murine nonalcoholic fatty liver disease model induced by a chronic exposure to high‐fat diet , 2018, Translational research : the journal of laboratory and clinical medicine.

[11]  J. Patrie,et al.  TH1 signatures are present in the lower airways of children with severe asthma, regardless of allergic status , 2017, The Journal of allergy and clinical immunology.

[12]  S. Wenzel,et al.  Severe asthma in humans and mouse model suggests a CXCL10 signature underlies corticosteroid-resistant Th1 bias. , 2017, JCI insight.

[13]  Wei Wu,et al.  Gene Expression Correlated with Severe Asthma Characteristics Reveals Heterogeneous Mechanisms of Severe Disease , 2017, American journal of respiratory and critical care medicine.

[14]  S. Wenzel,et al.  IRF5 distinguishes severe asthma in humans and drives Th1 phenotype and airway hyperreactivity in mice. , 2017, JCI insight.

[15]  Jinming Gao,et al.  Characteristics of Allergic Pulmonary Inflammation in CXCR3Knockout Mice Sensitized and Challenged with House Dust Mite Protein , 2016, PloS one.

[16]  O. Tliba,et al.  IFN-γ-induced JAK/STAT, but not NF-κB, signaling pathway is insensitive to glucocorticoid in airway epithelial cells. , 2015, American journal of physiology. Lung cellular and molecular physiology.

[17]  S. Wenzel,et al.  High IFN-γ and low SLPI mark severe asthma in mice and humans. , 2015, The Journal of clinical investigation.

[18]  E. Ferrannini,et al.  Chemokine (C-X-C motif) ligand (CXCL)10 in autoimmune diseases. , 2014, Autoimmunity reviews.

[19]  M. Tanimoto,et al.  Requirement for chemokine receptor 5 in the development of allergen-induced airway hyperresponsiveness and inflammation. , 2011, American journal of respiratory cell and molecular biology.

[20]  Yotaro Takaku,et al.  IFN-γ-inducible protein of 10 kDa upregulates the effector functions of eosinophils through β2 integrin and CXCR3 , 2011, Respiratory research.

[21]  Kewu Huang,et al.  Attenuation of antigen-induced airway hyperresponsiveness and inflammation in CXCR3 knockout mice , 2011, Respiratory research.

[22]  Alexander A. Morgan,et al.  Identification of an IFN-γ/mast cell axis in a mouse model of chronic asthma. , 2011, The Journal of clinical investigation.

[23]  J. Christensen,et al.  CXCR3 Directs Antigen-Specific Effector CD4+ T Cell Migration to the Lung During Parainfluenza Virus Infection1 , 2009, The Journal of Immunology.

[24]  Barmak Modrek,et al.  T-helper type 2-driven inflammation defines major subphenotypes of asthma. , 2009, American journal of respiratory and critical care medicine.

[25]  A. Antonelli,et al.  Immunopathogenesis of HCV-related endocrine manifestations in chronic hepatitis and mixed cryoglobulinemia. , 2008, Autoimmunity reviews.

[26]  R. Pawankar,et al.  Increased Expression of RANTES, CCR3 and CCR5 in the Lesional Skin of Patients with Atopic Eczema , 2006, International Archives of Allergy and Immunology.

[27]  C. Brightling,et al.  The CXCL10/CXCR3 axis mediates human lung mast cell migration to asthmatic airway smooth muscle. , 2005, American journal of respiratory and critical care medicine.

[28]  S. Wenzel,et al.  Increased TGF-β2 in severe asthma with eosinophilia , 2005 .

[29]  D. Robinson The role of the mast cell in asthma: induction of airway hyperresponsiveness by interaction with smooth muscle? , 2004, The Journal of allergy and clinical immunology.

[30]  B. Medoff,et al.  IFN-γ-Inducible Protein 10 (CXCL10) Contributes to Airway Hyperreactivity and Airway Inflammation in a Mouse Model of Asthma1 , 2002, The Journal of Immunology.

[31]  James J. Campbell,et al.  Expression of Chemokine Receptors by Lung T Cells from Normal and Asthmatic Subjects1 , 2001, The Journal of Immunology.

[32]  Sha Quan,et al.  CXCR3 Expression and Activation of Eosinophils: Role of IFN-γ-Inducible Protein-10 and Monokine Induced by IFN-γ1 , 2000, The Journal of Immunology.

[33]  Q. Hamid,et al.  The T cell-specific CXC chemokines IP-10, Mig, and I-TAC are expressed by activated human bronchial epithelial cells. , 1999, Journal of immunology.

[34]  C. Mackay,et al.  The chemokine receptors CXCR3 and CCR5 mark subsets of T cells associated with certain inflammatory reactions. , 1998, The Journal of clinical investigation.

[35]  V. Brusasco,et al.  Dissociation between airway inflammation and airway hyperresponsiveness in allergic asthma. , 1998, American journal of respiratory and critical care medicine.