Transcriptomic landscape of prurigo nodularis lesional skin CD3+ T cells using single-cell RNA-Sequencing.

[1]  V. Julia,et al.  Transcriptomic characterization of prurigo nodularis and the therapeutic response to nemolizumab. , 2021, The Journal of allergy and clinical immunology.

[2]  D. Corcoran,et al.  Prurigo Nodularis is Characterized by Systemic and Cutaneous Th22 Immune Polarization. , 2021, The Journal of investigative dermatology.

[3]  S. Teichmann,et al.  Developmental cell programs are co-opted in inflammatory skin disease , 2021, Science.

[4]  S. Kwatra,et al.  Prurigo Nodularis: Pathogenesis and Management. , 2020, Journal of the American Academy of Dermatology.

[5]  M. Mildner,et al.  Single-cell transcriptomics combined with interstitial fluid proteomics defines cell-type-specific immune regulation in atopic dermatitis. , 2020, The Journal of allergy and clinical immunology.

[6]  Chih-Hung Lee,et al.  IL-17A induces endothelin-1 expression through p38 pathway in prurigo nodularis. , 2020, The Journal of investigative dermatology.

[7]  L. Misery,et al.  Effectiveness and safety of dupilumab for the treatment of prurigo nodularis in a French multicenter adult cohort of 16 patients , 2020, Journal of the European Academy of Dermatology and Venereology : JEADV.

[8]  M. Dugas,et al.  European academy of dermatology and venereology European prurigo project: expert consensus on the definition, classification and terminology of chronic prurigo , 2018, Journal of the European Academy of Dermatology and Venereology : JEADV.

[9]  H. Das,et al.  KLF2 in Regulation of NF-κB-Mediated Immune Cell Function and Inflammation , 2017, International journal of molecular sciences.

[10]  Z. Modrušan,et al.  Comparative transcriptomic analyses of atopic dermatitis and psoriasis reveal shared neutrophilic inflammation. , 2012, The Journal of allergy and clinical immunology.

[11]  K. Yamasaki,et al.  Nuclear localization of activated STAT6 and STAT3 in epidermis of prurigo nodularis , 2011, The British journal of dermatology.

[12]  Tomoko Mori,et al.  Increased expression of mRNAs for IL-4, IL-17, IL-22 and IL-31 in skin lesions of subacute and chronic forms of prurigo. , 2011, European journal of dermatology : EJD.

[13]  T. Luger,et al.  Low density of sympathetic nerve fibers relative to substance P-positive nerve fibers in lesional skin of chronic pruritus and prurigo nodularis. , 2010, Journal of dermatological science.