Programmed Cell Death of Developing Mammalian Neurons after Genetic Deletion of Caspases
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R W Oppenheim | P. Rakic | R. Oppenheim | R. Flavell | P Rakic | R A Flavell | C Y Kuan | D. Prevette | S Vinsant | D Prevette | S. Vinsant | Chia-Yin Kuan
[1] R. Oppenheim. Cell death of motoneurons in the chick embryo spinal cord. VIII. Motoneurons prevented from dying in the embryo persist after hatching. , 1984, Developmental biology.
[2] P. Rakic,et al. Mechanisms of programmed cell death in the developing brain , 2000, Trends in Neurosciences.
[3] R. Oppenheim,et al. Muscle-specific cell ablation conditional upon Cre-mediated DNA recombination in transgenic mice leads to massive spinal and cranial motoneuron loss. , 1998, Developmental biology.
[4] R. Oppenheim,et al. Neurotrophic agents prevent motoneuron death following sciatic nerve section in the neonatal mouse. , 1994, Journal of neurobiology.
[5] P. Rakic,et al. Epistatic and independent functions of caspase-3 and Bcl-X(L) in developmental programmed cell death. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[6] Alan G. Porter,et al. Caspase-3 Is Required for DNA Fragmentation and Morphological Changes Associated with Apoptosis* , 1998, The Journal of Biological Chemistry.
[7] K. Yamamura,et al. An auxiliary mode of apoptotic DNA fragmentation provided by phagocytes. , 2000, Genes & development.
[8] P. Gruss,et al. Interdigital cell death can occur through a necrotic and caspase-independent pathway , 1999, Current Biology.
[9] I. Ferrer,et al. Role of caspases in ionizing radiation-induced apoptosis in the developing cerebellum. , 1999, Journal of neurobiology.
[10] A. Faden,et al. Ribozyme-Mediated Inhibition of Caspase-3 Protects Cerebellar Granule Cells from Apoptosis Induced by Serum–Potassium Deprivation , 2000, The Journal of Neuroscience.
[11] H. Li,et al. Deciphering the pathways of life and death. , 1999, Current opinion in cell biology.
[12] B. Pettmann,et al. Programmed Cell Death of Embryonic Motoneurons Triggered through the FAS Death Receptor , 1999, The Journal of cell biology.
[13] P. Rakic,et al. Caspase‐3 is required for apoptosis‐associated DNA fragmentation but not for cell death in neurons deprived of potassium , 2000, Journal of neuroscience research.
[14] P. Rakic,et al. Bcl-XL–Caspase-9 Interactions in the Developing Nervous System: Evidence for Multiple Death Pathways , 2001, The Journal of Neuroscience.
[15] H. Horvitz,et al. The genetics of programmed cell death in the nematode Caenorhabditis elegans. , 1994, Cold Spring Harbor symposia on quantitative biology.
[16] C. Lemaire,et al. Inhibition of caspase activity induces a switch from apoptosis to necrosis , 1998, FEBS letters.
[17] R. Oppenheim,et al. Neuron death in vertebrate development: in vitro methods. , 1995, Methods in cell biology.
[18] Francesco Cecconi,et al. Apaf1 (CED-4 Homolog) Regulates Programmed Cell Death in Mammalian Development , 1998, Cell.
[19] David S. Park,et al. Bax-Dependent Caspase-3 Activation Is a Key Determinant in p53-Induced Apoptosis in Neurons , 1999, The Journal of Neuroscience.
[20] S. Wilson,et al. Programmed cell death in zebrafish rohon beard neurons is influenced by TrkC1/NT-3 signaling. , 2000, Developmental biology.
[21] S. Lowe,et al. Essential contribution of caspase 3/CPP32 to apoptosis and its associated nuclear changes. , 1998, Genes & development.
[22] Eugene M. Johnson,et al. Caspase Inhibition Extends the Commitment to Neuronal Death Beyond Cytochrome c Release to the Point of Mitochondrial Depolarization , 2000, The Journal of cell biology.
[23] M. Bähr,et al. Long-Term Effect of Inhibition of ced 3-Like Caspases on the Survival of Axotomized Retinal Ganglion Cells in Vivo , 1999, Experimental Neurology.
[24] H. Nakanishi,et al. A Predominant Apoptotic Death Pathway of Neuronal PC12 Cells Induced by Activated Microglia Is Displaced by A Non-apoptotic Death Pathway Following Blockage of Caspase-3-dependent Cascade* , 1999, The Journal of Biological Chemistry.
[25] R. Oppenheim,et al. Cell death of motoneurons in the chick embryo spinal cord. II. A quantitative and qualitative analysis of degenerationin the ventral root, including evidence for axon outgrowth and limb innervation prior to cell death , 1978, The Journal of comparative neurology.
[26] M. J. Harris,et al. Mini-review: toward understanding mechanisms of genetic neural tube defects in mice. , 1999, Teratology.
[27] A. Tolkovsky,et al. Autophagy Is Activated by Apoptotic Signalling in Sympathetic Neurons: An Alternative Mechanism of Death Execution , 1999, Molecular and Cellular Neuroscience.
[28] J. Xiang,et al. BAX-induced cell death may not require interleukin 1 beta-converting enzyme-like proteases. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[29] C. Bortner,et al. Caspase Independent/Dependent Regulation of K+, Cell Shrinkage, and Mitochondrial Membrane Potential during Lymphocyte Apoptosis* , 1999, The Journal of Biological Chemistry.
[30] R. Oppenheim,et al. Cell death of motoneurons in the chick embryo spinal cord. IX. The loss of motoneurons following removal of afferent inputs , 1984, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[31] David S. Park,et al. Caspase-Dependent and -Independent Death of Camptothecin-Treated Embryonic Cortical Neurons , 1999, The Journal of Neuroscience.
[32] T. J. Cunningham,et al. Developmental neuron death in the rat superior cervical sympathetic ganglion: cell counts and ultrastructure , 1983, Journal of neurocytology.
[33] J. Boyer,et al. Caspase-3 controls both cytoplasmic and nuclear events associated with Fas-mediated apoptosis in vivo. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[34] T. Mak,et al. Essential role of the mitochondrial apoptosis-inducing factor in programmed cell death , 2001, Nature.
[35] M. Raff,et al. Are caspases involved in the death of cells with a transcriptionally inactive nucleus? Sperm and chicken erythrocytes. , 1998, Journal of cell science.
[36] V. Cryns,et al. Proteases to die for. , 1998, Genes & development.
[37] José Luis de la Pompa,et al. Differential Requirement for Caspase 9 in Apoptotic Pathways In Vivo , 1998, Cell.
[38] R. Oppenheim,et al. A Novel Type of Programmed Neuronal Death in the Cervical Spinal Cord of the Chick Embryo , 1996, The Journal of Neuroscience.
[39] Patrick Ng,et al. Caspase-3 Is Required for α-Fodrin Cleavage but Dispensable for Cleavage of Other Death Substrates in Apoptosis* , 1998, The Journal of Biological Chemistry.
[40] R. Oppenheim,et al. The development of motoneurons in the embryonic spinal cord of the mouse mutant, muscular dysgenesis (mdg/mdg): survival, morphology, and biochemical differentiation. , 1986, Developmental biology.
[41] R. Oppenheim,et al. Caspase Activity Is Involved in, but Is Dispensable for, Early Motoneuron Death in the Chick Embryo Cervical Spinal Cord , 2001, Molecular and Cellular Neuroscience.
[42] L. Landmesser,et al. Ultrastructural differences during embryonic cell death in normal and peripherally deprived ciliary ganglia , 1976, The Journal of cell biology.
[43] J. P. Bennett,et al. Evidence That Spinal Interneurons Undergo Programmed Cell Death Postnatally in the Rat , 1997, The European journal of neuroscience.
[44] Y. Uchiyama,et al. Regulation of a novel pathway for cell death by lysosomal aspartic and cysteine proteinases , 1999, Neuroscience.
[45] D. Bredesen,et al. An alternative, nonapoptotic form of programmed cell death. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[46] R. Oppenheim,et al. Chapter 13 Neuron Death in Vertebrate Development: In Vivo Methods , 1995 .
[47] Y. Lazebnik,et al. Deficiency in caspase-9 or caspase-3 induces compensatory caspase activation , 2000, Nature Medicine.
[48] R. Oppenheim,et al. Characterization of the execution pathway of developing motoneurons deprived of trophic support. , 2001, Journal of neurobiology.
[49] G. Kroemer,et al. The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell death , 1997, Oncogene.
[50] J B Schulz,et al. Caspases as treatment targets in stroke and neurodegenerative diseases , 1999, Annals of neurology.
[51] T. Mak,et al. Apaf1 Is Required for Mitochondrial Pathways of Apoptosis and Brain Development , 1998, Cell.
[52] P. Ernfors,et al. Attenuation of a Caspase-3 Dependent Cell Death in NT4- and p75-Deficient Embryonic Sensory Neurons , 2000, Molecular and Cellular Neuroscience.
[53] Keisuke Kuida,et al. Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice , 1996, Nature.
[54] Keisuke Kuida,et al. Reduced Apoptosis and Cytochrome c–Mediated Caspase Activation in Mice Lacking Caspase 9 , 1998, Cell.
[55] G. Evan,et al. Inhibition of Ced-3/ICE-related Proteases Does Not Prevent Cell Death Induced by Oncogenes, DNA Damage, or the Bcl-2 Homologue Bak , 1997, The Journal of cell biology.
[56] David S. Park,et al. Involvement of Caspase 3 in Apoptotic Death of Cortical Neurons Evoked by DNA Damage , 2000, Molecular and Cellular Neuroscience.
[57] H. Horvitz,et al. Mechanisms and functions of cell death. , 1991, Annual review of cell biology.
[58] L. Greene,et al. Caspase-2 (Nedd-2) Processing and Death of Trophic Factor-Deprived PC12 Cells and Sympathetic Neurons Occur Independently of Caspase-3 (CPP32)-Like Activity , 1998, The Journal of Neuroscience.
[59] S. Korsmeyer,et al. Bax Deletion Further Orders the Cell Death Pathway in Cerebellar Granule Cells and Suggests a Caspase-independent Pathway to Cell Death , 1997, The Journal of cell biology.