Role of ATP-sensitive potassium channels on hypoxic pulmonary vasoconstriction in endotoxemia
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[1] N. Weissmann,et al. Oxygen sensing and signal transduction in hypoxic pulmonary vasoconstriction , 2015, European Respiratory Journal.
[2] C. Teunissen,et al. Inhaled carbon monoxide protects time-dependently from loss of hypoxic pulmonary vasoconstriction in endotoxemic mice , 2015, Respiratory Research.
[3] Anlong Li,et al. Hypotension Due to Kir6.1 Gain‐of‐Function in Vascular Smooth Muscle , 2013, Journal of the American Heart Association.
[4] P. von der Weid,et al. Involvement of the NO-cGMP-K(ATP) channel pathway in the mesenteric lymphatic pump dysfunction observed in the guinea pig model of TNBS-induced ileitis. , 2013, American journal of physiology. Gastrointestinal and liver physiology.
[5] E. Jacobs,et al. A soluble epoxide hydrolase inhibitor--8-HUDE increases pulmonary vasoconstriction through inhibition of K(ATP) channels. , 2012, Pulmonary pharmacology & therapeutics.
[6] P. Asfar,et al. Vascular ATP-sensitive potassium channels are over-expressed and partially regulated by nitric oxide in experimental septic shock , 2011, Intensive Care Medicine.
[7] Daling Zhu,et al. Lipopolysaccharides Up-regulate Kir6.1/SUR2B Channel Expression and Enhance Vascular KATP Channel Activity via NF-κB-dependent Signaling* , 2009, The Journal of Biological Chemistry.
[8] C. Shih,et al. ABNORMAL ACTIVATION OF POTASSIUM CHANNELS IN AORTIC SMOOTH MUSCLE OF RATS WITH PERITONITIS-INDUCED SEPTIC SHOCK , 2009, Shock.
[9] A. Tinker,et al. Ca2+/calcineurin regulation of cloned vascular KATP channels: crosstalk with the protein kinase A pathway , 2009, British journal of pharmacology.
[10] P. Teschendorf,et al. Selective inhibition of guanylate cyclase prevents impairment of hypoxic pulmonary vasoconstriction in endotoxemic mice. , 2009, Journal of physiology and pharmacology : an official journal of the Polish Physiological Society.
[11] T. Martin,et al. Animal models of acute lung injury , 2008, American journal of physiology. Lung cellular and molecular physiology.
[12] Jin Han,et al. Physiological roles of K+ channels in vascular smooth muscle cells. , 2008, Journal of smooth muscle research = Nihon Heikatsukin Gakkai kikanshi.
[13] M. Westphal,et al. Inhibition of potassium channels in critical illness , 2008, Current opinion in anaesthesiology.
[14] W. Kuebler,et al. 4-Aminopyridine Restores Impaired Hypoxic Pulmonary Vasoconstriction in Endotoxemic Mice , 2007, Anesthesiology.
[15] M. Westphal,et al. GLIBENCLAMIDE DOSE RESPONSE IN PATIENTS WITH SEPTIC SHOCK: EFFECTS ON NOREPINEPHRINE REQUIREMENTS, CARDIOPULMONARY PERFORMANCE, AND GLOBAL OXYGEN TRANSPORT , 2007, Shock.
[16] Susumu Seino,et al. Gene knockout of the KCNJ8‐encoded Kir6.1 KATP channel imparts fatal susceptibility to endotoxemia , 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[17] A. T. Demiryürek,et al. Role of NO and Prostaglandins in Acute Hypoxic Vasoconstriction in Sheep Pulmonary Veins , 2006, Pharmacology.
[18] N. Teramoto. Pharmacological Profile of U-37883A, a Channel Blocker of Smooth Muscle-Type ATP-Sensitive K Channels. , 2006, Cardiovascular drug reviews.
[19] S. Naito,et al. Multiple actions of U-37883A, an ATP-sensitive K+ channel blocker, on membrane currents in pig urethra. , 2005, European journal of pharmacology.
[20] Min Gao,et al. Iptakalim, opener of KATP, reverses the enhanced expression of genes encoding KATP subunits in spontaneously hypertensive rats , 2005 .
[21] E. Martin,et al. Role of endogenous nitric oxide in endotoxin-induced alteration of hypoxic pulmonary vasoconstriction in mice. , 2005, American journal of physiology. Heart and circulatory physiology.
[22] M. Singer,et al. The pore‐forming subunit of the KATP channel is an important molecular target for LPS‐induced vascular hyporeactivity in vitro , 2005, British journal of pharmacology.
[23] H. Van Aken,et al. Modulation of hypoxic pulmonary vasoconstriction is time and nitric oxide dependent in a peritonitis model of sepsis , 2004, Intensive Care Medicine.
[24] T. Kamishima,et al. Molecular analysis of the subtype‐selective inhibition of cloned KATP channels by PNU‐37883A , 2004, British journal of pharmacology.
[25] A. Al-Mehdi,et al. Shear stress increases expression of a KATP channel in rat and bovine pulmonary vascular endothelial cells. , 2003, American journal of physiology. Cell physiology.
[26] Fang Liu,et al. Carbon monoxide modulates endotoxin-induced production of granulocyte macrophage colony-stimulating factor in macrophages. , 2002, American journal of respiratory cell and molecular biology.
[27] Andrew J. Wilson,et al. The molecular site of action of K(ATP) channel inhibitors determines their ability to inhibit iNOS-mediated relaxation in rat aorta. , 2002, Cardiovascular research.
[28] J. Bonventre,et al. Cytosolic phospholipase A(2) in hypoxic pulmonary vasoconstriction. , 2002, The Journal of clinical investigation.
[29] J. Bonventre,et al. Cytosolic phospholipase A2 in hypoxic pulmonary vasoconstriction , 2002 .
[30] J. Martens,et al. Molecular basis of hypoxia-induced pulmonary vasoconstriction: role of voltage-gated K+ channels. , 2001, American journal of physiology. Lung cellular and molecular physiology.
[31] S. Archer,et al. Potassium channels regulate tone in rat pulmonary veins. , 2001, American journal of physiology. Lung cellular and molecular physiology.
[32] W. Zapol,et al. Congenital NOS2 deficiency protects mice from LPS-induced hyporesponsiveness to inhaled nitric oxide. , 1999, Anesthesiology.
[33] W. Zapol,et al. Hypoxic pulmonary blood flow redistribution and arterial oxygenation in endotoxin-challenged NOS2-deficient mice. , 1999, The Journal of clinical investigation.
[34] M. Yen,et al. Role of nitric oxide and K+-channels in vascular hyporeactivity induced by endotoxin , 1999, Naunyn-Schmiedeberg's Archives of Pharmacology.
[35] C. Mélot,et al. Inhibition of cyclooxygenase and nitric oxide synthase in hypoxic vasoconstriction and oleic acid-induced lung injury. , 1999, American journal of respiratory and critical care medicine.
[36] A. Kristof,et al. Role of inducible nitric oxide synthase in endotoxin-induced acute lung injury. , 1998, American journal of respiratory and critical care medicine.
[37] S. Barman. Potassium channels modulate hypoxic pulmonary vasoconstriction. , 1998, American journal of physiology. Lung cellular and molecular physiology.
[38] M. Lazdunski,et al. Kv2.1/Kv9.3, a novel ATP‐dependent delayed‐rectifier K+ channel in oxygen‐sensitive pulmonary artery myocytes , 1997, The EMBO journal.
[39] L. Clapp,et al. Abnormal activation of K+ channels underlies relaxation to bacterial lipopolysaccharide in rat aorta. , 1996, Biochemical and biophysical research communications.
[40] T. Ishizaki,et al. Adenine nucleotides via activation of ATP-sensitive K+ channels modulate hypoxic response in rat pulmonary artery. , 1996, The American journal of physiology.
[41] A. Paterson,et al. Role of glucosamine synthesis in the stimulation of TGF-alpha gene transcription by glucose and EGF. , 1996, The American journal of physiology.
[42] J. H. Ludens,et al. Renal and Vascular Effects of Chemically Distinct ATP‐Sensitive K+ Channel Blockers in Rats , 1995, Journal of cardiovascular pharmacology.
[43] P. Barnes,et al. Regulation of pulmonary vascular tone. , 1995, Pharmacological reviews.
[44] Y. Nakaya,et al. Nonendothelial‐derived nitric oxide activates the ATP‐sensitive K+ channel of vascular smooth muscle cells , 1994, FEBS letters.
[45] C. Hanson,et al. Role of hypoxic pulmonary vasoconstriction in pulmonary gas exchange and blood flow distribution , 1994, Intensive Care Medicine.
[46] W. Mitzner,et al. On the purported discovery of the bronchial circulation by Leonardo da Vinci. , 1992, Journal of applied physiology.
[47] B. E. Robertson,et al. Opposing actions of tolbutamide and glibenclamide on hypoxic pulmonary vasoconstriction. , 1992, Comparative biochemistry and physiology. C, Comparative pharmacology and toxicology.
[48] I. McMurtry,et al. Inhibitors of Oxidative ATP Production Cause Transient Vasoconstriction and Block Subsequent Pressor Responses in Rat Lungs , 1981, Circulation research.
[49] R. Riley,et al. HEMODYNAMICS OF COLLAPSIBLE VESSELS WITH TONE: THE VASCULAR WATERFALL. , 1963, Journal of applied physiology.
[50] Min Gao,et al. Iptakalim, opener of K(ATP), reverses the enhanced expression of genes encoding K(ATP) subunits in spontaneously hypertensive rats. , 2005, Life sciences.
[51] Yi Cui,et al. The molecular composition of K(ATP) channels in human pulmonary artery smooth muscle cells and their modulation by growth. , 2002, American journal of respiratory cell and molecular biology.