Sequential assembly of the nucleotide excision repair factors in vivo.

[1]  A. Lehmann,et al.  Failure of RNA synthesis to recover after UV irradiation: an early defect in cells from individuals with Cockayne's syndrome and xeroderma pigmentosum. , 1982, Cancer research.

[2]  R. Groenendijk,et al.  Recovery of growth-arrested human fibroblasts from UV-induced lethal damage is inhibited by low cell density or sodium butyrate. , 1985, Mutation research.

[3]  G. Chu,et al.  Xeroderma pigmentosum group E cells lack a nuclear factor that binds to damaged DNA. , 1988, Science.

[4]  A. Yasui,et al.  Analysis of a human DNA excision repair gene involved in group A xeroderma pigmentosum and containing a zinc-finger domain , 1990, Nature.

[5]  L. Mullenders,et al.  The genetic defect in Cockayne syndrome is associated with a defect in repair of UV-induced DNA damage in transcriptionally active DNA. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[6]  D. Lawrence,et al.  Preferential repair of DNA damage on the transcribed strand of the human metallothionein genes requires RNA polymerase II. , 1991, Mutation research.

[7]  O. Nikaido,et al.  SIMULTANEOUS ESTABLISHMENT OF MONOCLONAL ANTIBODIES SPECIFIC FOR EITHER CYCLOBUTANE PYRIMIDINE DIMER OR (6‐4)PHOTOPRODUCT FROM THE SAME MOUSE IMMUNIZED WITH ULTRAVIOLET‐IRRADIATED DNA , 1991, Photochemistry and photobiology.

[8]  L. Mullenders,et al.  Xeroderma pigmentosum complementation group C cells remove pyrimidine dimers selectively from the transcribed strand of active genes , 1991, Molecular and cellular biology.

[9]  L. Mullenders,et al.  Transcription affects the rate but not the extent of repair of cyclobutane pyrimidine dimers in the human adenosine deaminase gene. , 1992, The Journal of biological chemistry.

[10]  A. Bardwell,et al.  Dual roles of a multiprotein complex from S. cerevisiae in transcription and DNA repair , 1993, Cell.

[11]  P. Chambon,et al.  DNA repair helicase: a component of BTF2 (TFIIH) basic transcription factor. , 1993, Science.

[12]  L. Mullenders,et al.  Deficient repair of the transcribed strand of active genes in Cockayne's syndrome cells. , 1993, Nucleic acids research.

[13]  K. Tanaka,et al.  The XPA protein is a zinc metalloprotein with an ability to recognize various kinds of DNA damage. , 1994, Mutation research.

[14]  D. Reinberg,et al.  Dual role of TFIIH in DNA excision repair and in transcription by RNA polymerase II , 1994, Nature.

[15]  D. Jackson,et al.  Sites in human nuclei where DNA damaged by ultraviolet light is repaired: visualization and localization relative to the nucleoskeleton. , 1994, Journal of cell science.

[16]  L. Mullenders,et al.  Transcription‐coupled repair removes both cyclobutane pyrimidine dimers and 6‐4 photoproducts with equal efficiency and in a sequential way from transcribed DNA in xeroderma pigmentosum group C fibroblasts. , 1995, The EMBO journal.

[17]  R. Legerski,et al.  Mutations in XPA that prevent association with ERCC1 are defective in nucleotide excision repair , 1995, Molecular and cellular biology.

[18]  A. Sancar,et al.  The General Transcription-Repair Factor TFIIH Is Recruited to the Excision Repair Complex by the XPA Protein Independent of the TFIIE Transcription Factor (*) , 1995, The Journal of Biological Chemistry.

[19]  R. Legerski,et al.  An interaction between the DNA repair factor XPA and replication protein A appears essential for nucleotide excision repair , 1995, Molecular and cellular biology.

[20]  P. Hanawalt.  DNA repair comes of age. , 1995, Mutation research.

[21]  D. Bushnell,et al.  Different forms of TFIIH for transcription and DNA repair: Holo-TFIIH and a nucleotide excision repairosome , 1995, Cell.

[22]  N. Iyer,et al.  Interactions involving the human RNA polymerase II transcription/nucleotide excision repair complex TFIIH, the nucleotide excision repair protein XPG, and Cockayne syndrome group B (CSB) protein. , 1996, Biochemistry.

[23]  R. Wood,et al.  Xeroderma Pigmentosum Group F Caused by a Defect in a Structure-Specific DNA Repair Endonuclease , 1996, Cell.

[24]  A. Sancar,et al.  Overproduction, Purification, and Characterization of the XPC Subunit of the Human DNA Repair Excision Nuclease* , 1996, The Journal of Biological Chemistry.

[25]  D. S. Hsu,et al.  Reaction Mechanism of Human DNA Repair Excision Nuclease (*) , 1996, The Journal of Biological Chemistry.

[26]  P. Sung,et al.  Nucleotide Excision Repair in Yeast Is Mediated by Sequential Assembly of Repair Factors and Not by a Pre-assembled Repairosome (*) , 1996, The Journal of Biological Chemistry.

[27]  Chikahide Masutani,et al.  XPC and human homologs of RAD23: intracellular localization and relationship to other nucleotide excision repair complexes , 1996, Nucleic Acids Res..

[28]  S. A. Leadon,et al.  A common mutational pattern in Cockayne syndrome patients from xeroderma pigmentosum group G: implications for a second XPG function. , 1997, Proceedings of the National Academy of Sciences of the United States of America.

[29]  J. Egly,et al.  DNA Damage Recognition by XPA Protein Promotes Efficient Recruitment of Transcription Factor II H* , 1997, The Journal of Biological Chemistry.

[30]  K. Kinzler,et al.  The Genetic Basis of Human Cancer , 1997 .

[31]  R. Wood,et al.  Mechanism of open complex and dual incision formation by human nucleotide excision repair factors , 1997, The EMBO journal.

[32]  L. Thompson,et al.  Reconstitution of Human Excision Nuclease with Recombinant XPF-ERCC1 Complex* , 1997, The Journal of Biological Chemistry.

[33]  R. van Driel,et al.  Nuclear distribution of transcription factors in relation to sites of transcription and RNA polymerase II. , 1997, Journal of cell science.

[34]  L. Mullenders,et al.  Enzymatic activities involved in the DNA resynthesis step of nucleotide excision repair are firmly attached to chromatin. , 1997, Nucleic acids research.

[35]  P. J. van der Spek,et al.  Xeroderma pigmentosum group C protein complex is the initiator of global genome nucleotide excision repair. , 1998, Molecular cell.

[36]  W. de Laat,et al.  DNA-binding polarity of human replication protein A positions nucleases in nucleotide excision repair. , 1998, Genes & development.

[37]  Y. Matsumura,et al.  Characterization of molecular defects in xeroderma pigmentosum group F in relation to its clinically mild symptoms. , 1998, Human molecular genetics.

[38]  W. de Laat,et al.  Molecular mechanism of nucleotide excision repair. , 1999, Genes & development.

[39]  A. Houtsmuller,et al.  Action of DNA repair endonuclease ERCC1/XPF in living cells. , 1999, Science.

[40]  A. Sancar,et al.  Order of Assembly of Human DNA Repair Excision Nuclease* , 1999, The Journal of Biological Chemistry.

[41]  R. Wood,et al.  Stable binding of human XPC complex to irradiated DNA confers strong discrimination for damaged sites. , 2000, Journal of molecular biology.

[42]  P. Hanawalt,et al.  Xeroderma pigmentosum p48 gene enhances global genomic repair and suppresses UV-induced mutagenesis. , 2000, Molecular cell.

[43]  R. Wood,et al.  Damage recognition in nucleotide excision repair of DNA. , 2000, Gene.

[44]  K. Schwarz,et al.  Reaction mechanism of , 2000 .

[45]  K. Sugasawa,et al.  The Xeroderma Pigmentosum Group C Protein Complex XPC-HR23B Plays an Important Role in the Recruitment of Transcription Factor IIH to Damaged DNA* , 2000, The Journal of Biological Chemistry.

[46]  K. Sugasawa,et al.  A multistep damage recognition mechanism for global genomic nucleotide excision repair. , 2001, Genes & development.