Small intestinal production of nitric oxide is decreased following resuscitated hemorrhage.
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M. Wilson | D. Spain | P. Matheson | R. N. Garrison | P. Harris | T. Fruchterman | A. W. Martin | R. Garrison
[1] N. Christou,et al. Increased plasma von Willebrand factor in the systemic inflammatory response syndrome is derived from generalized endothelial cell activation. , 1998, Critical care medicine.
[2] Simon C Watkins,et al. PHYSIOLOGIC AND MOLECULAR CHARACTERIZATION OF THE ROLE OF NITRIC OXIDE IN HEMORRHAGIC SHOCK: EVIDENCE THAT TYPE II NITRIC OXIDE SYNTHASE DOES NOT REGULATE VASCULAR DECOMPENSATION , 1997, Shock.
[3] M. Wilson,et al. Heparan preserves intestinal perfusion after hemorrhage and resuscitation. , 1996, The Journal of surgical research.
[4] A. M. Lefer,et al. The role of nitric oxide and cell adhesion molecules on the microcirculation in ischaemia-reperfusion. , 1996, Cardiovascular research.
[5] R. Ivatury,et al. A prospective randomized study of end points of resuscitation after major trauma: global oxygen transport indices versus organ-specific gastric mucosal pH. , 1996, Journal of the American College of Surgeons.
[6] H. Redl,et al. Significance of NO in hemorrhage-induced hemodynamic alterations, organ injury, and mortality in rats. , 1996, The American journal of physiology.
[7] J. Waymack,et al. Trauma, shock, and gut translocation. , 1996, New horizons.
[8] K. Waxman,et al. Increasing nitric oxide production improves survival in experimental hemorrhagic shock. , 1996, Resuscitation.
[9] Simon C Watkins,et al. Inhibition of nitric oxide synthase during hemorrhagic shock increases hepatic injury. , 1995, Shock.
[10] M. Wilson,et al. Alpha-adrenergic receptor antagonism prevents intestinal vasoconstriction but not hypoperfusion following resuscitated hemorrhage. , 1995, The Journal of surgical research.
[11] R. Ivatury,et al. Gastric mucosal pH and oxygen delivery and oxygen consumption indices in the assessment of adequacy of resuscitation after trauma: a prospective, randomized study. , 1995, The Journal of trauma.
[12] T. Miller,et al. Hemorrhagic shock increases gut macromolecular permeability in the rat. , 1995, Shock.
[13] C. Legrand,et al. Cross-reactivity of human molecular markers for detection of prethrombotic states in various animal species. , 1995, Blood coagulation & fibrinolysis : an international journal in haemostasis and thrombosis.
[14] W. Flynn,et al. Xanthine oxidase inhibition after resuscitated hemorrhagic shock restores mesenteric blood flow without vasodilation. , 1995 .
[15] C. Wollheim,et al. Reactive oxygen intermediates induce regulated secretion of von Willebrand factor from cultured human vascular endothelial cells. , 1995, Blood.
[16] C. Gray,et al. NITRIC OXIDE SYNTHESIS INHIBITION DOES NOT IMPROVE THE HEMODYNAMIC RESPONSE TO HEMORRHAGIC SHOCK IN DEHYDRATED CONSCIOUS SWINE , 1995, Shock.
[17] J. Beckman,et al. Evidence for in vivo peroxynitrite production in human acute lung injury. , 1995, American journal of respiratory and critical care medicine.
[18] S. Myers,et al. Oxygen free radicals regulate splanchnic nitric oxide synthesis and blood flow. , 1995, Cardiovascular surgery.
[19] J. Beckman,et al. Nitric oxide-related oxidants in acute lung injury. , 1995, New horizons.
[20] P. Kubes,et al. Time course of nitric oxide production and epithelial dysfunction during ischemia/reperfusion of the feline small intestine. , 1994, Circulatory shock.
[21] P G Anderson,et al. Extensive nitration of protein tyrosines in human atherosclerosis detected by immunohistochemistry. , 1994, Biological chemistry Hoppe-Seyler.
[22] G. Tominaga,et al. A method to determine the adequacy of resuscitation using tissue oxygen monitoring. , 1993, The Journal of trauma.
[23] C. Thiemermann,et al. Vascular hyporeactivity to vasoconstrictor agents and hemodynamic decompensation in hemorrhagic shock is mediated by nitric oxide. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[24] W. Shoemaker,et al. Prospective trial of supranormal values as goals of resuscitation in severe trauma. , 1992, Archives of surgery.
[25] S. Myers,et al. Oxygen free radical regulation of rat splanchnic blood flow. , 1992, Surgery.
[26] S. Myers,et al. Role of oxygen-derived free radicals on rat splanchnic eicosanoid production during acute hemorrhage. , 1992, Prostaglandins.
[27] H. Battifora,et al. Assessment of antigen damage in immunohistochemistry. The vimentin internal control. , 1991, American journal of clinical pathology.
[28] R. N. Garrison,et al. Prostaglandins mediate the compensatory responses to hemorrhage in the small intestine of the rat. , 1991, The Journal of surgical research.
[29] P. Kubes,et al. Nitric oxide: an endogenous modulator of leukocyte adhesion. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[30] D. Ginsburg. The von Willebrand factor gene and genetics of von Willebrand's disease. , 1991, Mayo Clinic proceedings.
[31] Fiddian-Green Rg,et al. The role of the gut in shock and multiple system organ failure. , 1991 .
[32] A. M. Lefer,et al. Time course and mechanism of endothelial dysfunction in isolated ischemic- and hypoxic-perfused rat hearts. , 1990, The American journal of physiology.
[33] S. Myers,et al. Reperfusion Inhibits Elevated Splanchnic Prostanoid Production After Hemorrhagic Shock , 1990, Annals of surgery.
[34] I. Chaudry,et al. Hemorrhage produces depression in microvascular blood flow which persists despite fluid resuscitation. , 1990, Circulatory shock.
[35] P. Sims,et al. Complement proteins C5b-9 induce secretion of high molecular weight multimers of endothelial von Willebrand factor and translocation of granule membrane protein GMP-140 to the cell surface. , 1989, The Journal of biological chemistry.
[36] S. Schwartz,et al. Injury induces increase of von Willebrand factor in rat endothelial cells. , 1989, The American journal of pathology.
[37] J. Pearson,et al. von Willebrand factor is an acute phase reactant in man. , 1989, Thrombosis research.
[38] W. Shoemaker,et al. Tissue oxygen debt as a determinant of lethal and nonlethal postoperative organ failure. , 1988, Critical care medicine.
[39] S. Moncada,et al. The anti‐aggregating properties of vascular endothelium: interactions between prostacyclin and nitric oxide , 1987, British journal of pharmacology.
[40] J. Hassett,et al. The Gut Origin Septic States in Blunt Multiple Trauma (ISS = 40) in the ICU , 1987, Annals of surgery.
[41] J. McCord,et al. Oxygen-derived free radicals in postischemic tissue injury. , 1985, The New England journal of medicine.