S100A4 and Bone Morphogenetic Protein-2 Codependently Induce Vascular Smooth Muscle Cell Migration via Phospho–Extracellular Signal-Regulated Kinase and Chloride Intracellular Channel 4
暂无分享,去创建一个
A. Schmidt | N. Ambartsumian | M. Berryman | M. Rabinovitch | C. Guignabert | E. Spiekerkoetter | V. A. de Jesus Perez | Lingli Wang | A. Lawrie | R. Ashley | T. Alastalo | Janine Powers | Tero‐Pekka Alastalo
[1] D. Hall,et al. A C. elegans CLIC-like Protein Required for Intracellular Tube Formation and Maintenance , 2003, Science.
[2] N. Schor,et al. Gremlin promotes vascular smooth muscle cell proliferation and migration. , 2008, Journal of molecular and cellular cardiology.
[3] R. Müller,et al. Proteomic profile of mouse fibroblasts with a targeted disruption of the peroxisome proliferator activated receptor‐β/δ gene , 2007 .
[4] M. Andrassy,et al. Central role of RAGE-dependent neointimal expansion in arterial restenosis. , 2003, The Journal of clinical investigation.
[5] A. Bretscher,et al. Identification of a novel member of the chloride intracellular channel gene family (CLIC5) that associates with the actin cytoskeleton of placental microvilli. , 2000, Molecular biology of the cell.
[6] J. Axelrod,et al. Bone morphogenetic protein 2 induces pulmonary angiogenesis via Wnt –-catenin and Wnt – RhoA – Rac 1 pathways , 2022 .
[7] H. Beppu,et al. Bone Morphogenetic Protein (BMP) Type II Receptor Deletion Reveals BMP Ligand-specific Gain of Signaling in Pulmonary Artery Smooth Muscle Cells* , 2005, Journal of Biological Chemistry.
[8] R. Trembath,et al. Heterozygous germline mutations in BMPR2, encoding a TGF-β receptor, cause familial primary pulmonary hypertension , 2000, Nature Genetics.
[9] L. Truong,et al. Advanced glycation end products activate Smad signaling via TGF‐β‐dependent and ‐independent mechanisms: implications for diabetic renal and vascular disease , 2004 .
[10] R. Muraro,et al. The Receptor RAGE as a Progression Factor Amplifying Arachidonate-Dependent Inflammatory and Proteolytic Response in Human Atherosclerotic Plaques: Role of Glycemic Control , 2003, Circulation.
[11] M. Nakajima,et al. The receptor for advanced glycation end‐products (RAGE) directly binds to ERK by a D‐domain‐like docking site , 2003, FEBS letters.
[12] J. Mellon,et al. Metastasis-associated protein S100A4: spotlight on its role in cell migration. , 2007, Current cancer drug targets.
[13] P. Picci,et al. Activation of metalloproteinases-2 and -9 by interleukin-1alpha in S100A4-positive liposarcoma cell line: correlation with cell invasiveness. , 2004, Anticancer research.
[14] A. Bresnick,et al. The S100A4 metastasis factor regulates cellular motility via a direct interaction with myosin-IIA. , 2006, Cancer research.
[15] R. Müller,et al. Proteomic profile of mouse fibroblasts with a targeted disruption of the peroxisome proliferator activated receptor-beta/delta gene. , 2007, Proteomics.
[16] V. Berezin,et al. The metastasis-associated Mts1(S100A4) protein could act as an angiogenic factor , 2001, Oncogene.
[17] L. Truong,et al. Advanced glycation end products activate Smad signaling via TGF-beta-dependent and independent mechanisms: implications for diabetic renal and vascular disease. , 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[18] W. Stetler-Stevenson,et al. Primate smooth muscle cell migration from aortic explants is mediated by endogenous platelet-derived growth factor and basic fibroblast growth factor acting through matrix metalloproteinases 2 and 9. , 1997, Circulation.
[19] R. Willette,et al. BMP-2 Gene Expression and Effects on Human Vascular Smooth Muscle Cells , 1999, Journal of Vascular Research.
[20] K. Desai,et al. Increased Fibulin-5 and Elastin in S100A4/Mts1 Mice With Pulmonary Hypertension , 2005, Circulation research.
[21] K. Fukunaga,et al. CLIC4 interacts with histamine H3 receptor and enhances the receptor cell surface expression. , 2008, Biochemical and Biophysical Research Communications - BBRC.
[22] D. Wilson,et al. The BMP type II receptor is located in lipid rafts, including caveolae, of pulmonary endothelium in vivo and in vitro. , 2006, Vascular pharmacology.
[23] A. Bresnick,et al. Mts1 regulates the assembly of nonmuscle myosin-IIA. , 2003, Biochemistry.
[24] N. Ambartsumian,et al. S100A4/Mts1 produces murine pulmonary artery changes resembling plexogenic arteriopathy and is increased in human plexogenic arteriopathy. , 2004, The American journal of pathology.
[25] J. Axelrod,et al. Bone morphogenetic protein 2 induces pulmonary angiogenesis via Wnt–β-catenin and Wnt–RhoA–Rac1 pathways , 2009, Journal of Cell Biology.
[26] S. Hodge,et al. Familial primary pulmonary hypertension (gene PPH1) is caused by mutations in the bone morphogenetic protein receptor-II gene. , 2000, American journal of human genetics.
[27] J. Wharton,et al. Phosphodiesterase type 4 expression and anti-proliferative effects in human pulmonary artery smooth muscle cells , 2006, Respiratory research.
[28] M. Grigorian,et al. Genetically modified mouse models to study the role of metastasis-promoting S100A4(mts1) protein in metastatic mammary cancer , 2005, Journal of Dairy Research.
[29] N. Rudarakanchana,et al. Serotonin Increases Susceptibility to Pulmonary Hypertension in BMPR2-Deficient Mice , 2006, Circulation research.
[30] Marta Costa,et al. Bmp2 is required for migration but not for induction of neural crest cells in the mouse , 2007, Developmental dynamics : an official publication of the American Association of Anatomists.
[31] G. Hansmann,et al. An antiproliferative BMP-2/PPARgamma/apoE axis in human and murine SMCs and its role in pulmonary hypertension. , 2008, The Journal of clinical investigation.
[32] M. Bacchetta,et al. Intimal Smooth Muscle Cells of Porcine and Human Coronary Artery Express S100A4, a Marker of the Rhomboid Phenotype In Vitro , 2007, Circulation research.
[33] W. Nelson,et al. Localized zones of Rho and Rac activities drive initiation and expansion of epithelial cell–cell adhesion , 2007, The Journal of cell biology.
[34] A. Steven,et al. mtCLIC/CLIC4, an Organellular Chloride Channel Protein, Is Increased by DNA Damage and Participates in the Apoptotic Response to p53 , 2002, Molecular and Cellular Biology.
[35] W. Nacken,et al. The myeloid expressed EF‐hand proteins display a diverse pattern of lipid raft association , 2004, FEBS letters.
[36] M. Berryman,et al. CLIC4 is enriched at cell-cell junctions and colocalizes with AKAP350 at the centrosome and midbody of cultured mammalian cells. , 2003, Cell motility and the cytoskeleton.
[37] R. Trembath,et al. Investigation of Second Genetic Hits at the BMPR2 Locus as a Modulator of Disease Progression in Familial Pulmonary Arterial Hypertension , 2005, Circulation.
[38] A. Aitken,et al. Chloride intracellular channel protein CLIC4 (p64H1) binds directly to brain dynamin I in a complex containing actin, tubulin and 14-3-3 isoforms. , 2001, The Biochemical journal.
[39] S. Vijayaraghavan,et al. Identification of chloride intracellular channel proteins in spermatozoa , 2004, FEBS letters.
[40] R. Markwald,et al. BMP-2 induces cell migration and periostin expression during atrioventricular valvulogenesis. , 2008, Developmental biology.
[41] A. Harmar,et al. Interdependent Serotonin Transporter and Receptor Pathways Regulate S100A4/Mts1, a Gene Associated With Pulmonary Vascular Disease , 2005, Circulation research.
[42] O. Petersen,et al. Differential expression of a chloride intracellular channel gene, CLIC4, in transforming growth factor-beta1-mediated conversion of fibroblasts to myofibroblasts. , 2002, The American journal of pathology.
[43] P. Jones,et al. Tenascin-C, proliferation and subendothelial fibronectin in progressive pulmonary vascular disease. , 1997, The American journal of pathology.