Allosteric regulation of the transcription factor NFAT1 by multiple phosphorylation sites: a mathematical analysis.
暂无分享,去创建一个
[1] Richard S Lewis,et al. Calcium signaling mechanisms in T lymphocytes. , 2001, Annual review of immunology.
[2] E. O’Shea,et al. Regulation of nuclear localization: a key to a door. , 1999, Annual review of cell and developmental biology.
[3] P. Roach,et al. Multisite and hierarchal protein phosphorylation. , 1991, The Journal of biological chemistry.
[4] J. Changeux,et al. ON THE NATURE OF ALLOSTERIC TRANSITIONS: A PLAUSIBLE MODEL. , 1965, Journal of molecular biology.
[5] Xi He,et al. Control of β-Catenin Phosphorylation/Degradation by a Dual-Kinase Mechanism , 2002, Cell.
[6] R. Nussinov,et al. Folding and binding cascades: Dynamic landscapes and population shifts , 2008, Protein science : a publication of the Protein Society.
[7] G. Crabtree. Calcium, Calcineurin, and the Control of Transcription* , 2001, The Journal of Biological Chemistry.
[8] Andreas Radbruch,et al. GATA-3 transcriptional imprinting in Th2 lymphocytes: A mathematical model , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[9] Katsuhiko Mikoshiba,et al. The Wnt/calcium pathway activates NF-AT and promotes ventral cell fate in Xenopus embryos , 2002, Nature.
[10] P. Hogan,et al. Transcription factors of the NFAT family: regulation and function. , 1997, Annual review of immunology.
[11] Keli Xu,et al. Calcium oscillations increase the efficiency and specificity of gene expression , 1998, Nature.
[12] E. Heist,et al. Protein Kinase A Negatively Modulates the Nuclear Accumulation of NF-ATc1 by Priming for Subsequent Phosphorylation by Glycogen Synthase Kinase-3* , 2002, The Journal of Biological Chemistry.
[13] Chi-Ying F. Huang,et al. Ultrasensitivity in the mitogen-activated protein kinase cascade. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[14] J. Qin,et al. Concerted dephosphorylation of the transcription factor NFAT1 induces a conformational switch that regulates transcriptional activity. , 2000, Molecular cell.
[15] Andrea Mozzarelli,et al. Is cooperative oxygen binding by hemoglobin really understood? , 1999, Nature Structural Biology.
[16] S. Schuster,et al. Modelling of simple and complex calcium oscillations , 2002 .
[17] W. R. Burack,et al. The activating dual phosphorylation of MAPK by MEK is nonprocessive. , 1997, Biochemistry.
[18] J. Harbour,et al. Cdk Phosphorylation Triggers Sequential Intramolecular Interactions that Progressively Block Rb Functions as Cells Move through G1 , 1999, Cell.
[19] P. Cohen,et al. The regulation of protein function by multisite phosphorylation--a 25 year update. , 2000, Trends in biochemical sciences.
[20] L. Nicholson,et al. Phosphorylation-induced structural changes in the amyloid precursor protein cytoplasmic tail detected by NMR. , 2001, Journal of molecular biology.
[21] D. Koshland,et al. An amplified sensitivity arising from covalent modification in biological systems. , 1981, Proceedings of the National Academy of Sciences of the United States of America.
[22] J. C. Nuño,et al. Generalization of the theory of transition times in metabolic pathways: a geometrical approach. , 1999, Biophysical journal.
[23] James E. Ferrell,et al. Mechanistic Studies of the Dual Phosphorylation of Mitogen-activated Protein Kinase* , 1997, The Journal of Biological Chemistry.
[24] K. Stankunas,et al. Monitoring the duration of antigen-receptor occupancy by calcineurin/glycogen-synthase-kinase-3 control of NF-AT nuclear shuttling. , 2001, Current opinion in immunology.
[25] W. Lipscomb,et al. Escherichia coli aspartate transcarbamoylase: the molecular basis for a concerted allosteric transition. , 1990, Trends in biochemical sciences.
[26] A. Rao,et al. Manipulating immune responses with immunosuppressive agents that target NFAT. , 2000, Immunity.
[27] F. McKeon,et al. NF-AT activation requires suppression of Crm1-dependent export by calcineurin , 1999, Nature.
[28] Uri Alon,et al. Response regulator output in bacterial chemotaxis , 1998, The EMBO journal.
[29] Christopher C. Goodnow,et al. Differential activation of transcription factors induced by Ca2+ response amplitude and duration , 1997, Nature.
[30] D E Wemmer,et al. Two-state allosteric behavior in a single-domain signaling protein. , 2001, Science.
[31] R. Heinrich,et al. The Regulation of Cellular Systems , 1996, Springer US.
[32] I. Graef,et al. Signaling through calcium, calcineurin, and NF-AT in lymphocyte activation and development. , 1999, Cold Spring Harbor symposia on quantitative biology.
[33] E. Price,et al. Role of kinases and the phosphatase calcineurin in the nuclear shuttling of transcription factor NF-AT4 , 1996, Nature.
[34] D. Koshland,et al. Comparison of experimental binding data and theoretical models in proteins containing subunits. , 1966, Biochemistry.
[35] E. O’Shea,et al. Multi-site phosphorylation of Pho4 by the cyclin-CDK Pho80-Pho85 is semi-processive with site preference. , 2001, Journal of molecular biology.
[36] N Le Novère,et al. Conformational spread in a ring of proteins: a stochastic approach to allostery. , 2001, Journal of molecular biology.
[37] F. Berberich-Siebelt,et al. The role of NF-AT transcription factors in T cell activation and differentiation. , 2000, Biochimica et biophysica acta.
[38] P. Hogan,et al. Immunosuppressive drugs prevent a rapid dephosphorylation of transcription factor NFAT1 in stimulated immune cells. , 1995, Proceedings of the National Academy of Sciences of the United States of America.