Neuronal functions, feeding behavior, and energy balance in Slc2a3+/- mice.
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H. Himmelbauer | A. Schürmann | H. Joost | P. Ruth | A. Hennige | T. Sartorius | S. Lutz | D. Montag | R. Kluge | S. Scherneck | Miriam Richter | R. Augustin | S. Schmidt | V. Gawlik | M. Richter | Verena Gawlik
[1] S. Devaskar,et al. Glucose transporter isoform-3-null heterozygous mutation causes sexually dimorphic adiposity with insulin resistance. , 2008, American journal of physiology. Endocrinology and metabolism.
[2] A. Schousboe,et al. Energy substrates to support glutamatergic and GABAergic synaptic function: Role of glycogen, glucose and lactate , 2007, Neurotoxicity Research.
[3] J. Klepper,et al. GLUT1 deficiency syndrome – 2007 update , 2007, Developmental medicine and child neurology.
[4] J. Rottman,et al. Glucose kinetics and exercise tolerance in mice lacking the GLUT4 glucose transporter , 2007, The Journal of physiology.
[5] S. Vannucci,et al. Supply and Demand in Cerebral Energy Metabolism: The Role of Nutrient Transporters , 2007, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[6] B. Shin,et al. Glucose transporter isoform-3 mutations cause early pregnancy loss and fetal growth restriction. , 2007, American journal of physiology. Endocrinology and metabolism.
[7] R. C. Honey,et al. Revisiting places passed: Sensitization of exploratory activity in rats with hippocampal lesions , 2007, Quarterly journal of experimental psychology.
[8] C. Meyer,et al. Ablation of the cholesterol transporter adenosine triphosphate-binding cassette transporter G1 reduces adipose cell size and protects against diet-induced obesity. , 2007, Endocrinology.
[9] E. Schleicher,et al. Leptin Down-regulates Insulin Action through Phosphorylation of Serine-318 in Insulin Receptor Substrate 1 1. Leptin Stimulates Ser-318 Phosphorylation in Insulin Receptor Substrate 1 , 2022 .
[10] Sylvia Ortmann,et al. Hyperphagia, lower body temperature, and reduced running wheel activity precede development of morbid obesity in New Zealand obese mice. , 2006, Physiological genomics.
[11] J. Noebels,et al. A mouse model for Glut-1 haploinsufficiency. , 2006, Human molecular genetics.
[12] H. Preissl,et al. Tissue selectivity of insulin detemir action in vivo , 2006, Diabetologia.
[13] Hong Yang,et al. Glut‐1 deficiency syndrome: Clinical, genetic, and therapeutic aspects , 2005, Annals of neurology.
[14] D. Wasserman,et al. Control of Exercise-stimulated Muscle Glucose Uptake by GLUT4 Is Dependent on Glucose Phosphorylation Capacity in the Conscious Mouse* , 2004, Journal of Biological Chemistry.
[15] A. Schürmann,et al. The glucose transporter families SGLT and GLUT: molecular basis of normal and aberrant function. , 2004, JPEN. Journal of parenteral and enteral nutrition.
[16] Angus M. Brown. Brain glycogen re‐awakened , 2004, Journal of neurochemistry.
[17] V. Routh,et al. Physiological and molecular characteristics of rat hypothalamic ventromedial nucleus glucosensing neurons. , 2004, Diabetes.
[18] P. Garnier,et al. Hypoglycemic Neuronal Death and Cognitive Impairment Are Prevented by Poly(ADP-Ribose) Polymerase Inhibitors Administered after Hypoglycemia , 2003, The Journal of Neuroscience.
[19] D. Montag,et al. Severe cognitive and motor coordination deficits in Tenascin‐R‐deficient mice , 2003, Genes, brain, and behavior.
[20] M. Schachner,et al. Misguided Axonal Projections, Neural Cell Adhesion Molecule 180 mRNA Upregulation, and Altered Behavior in Mice Deficient for the Close Homolog of L1 , 2002, Molecular and Cellular Biology.
[21] J. Rawlins,et al. Anxiolytic effects of cytotoxic hippocampal lesions in rats. , 2002, Behavioral neuroscience.
[22] M. Quirk,et al. Requirement for Hippocampal CA3 NMDA Receptors in Associative Memory Recall , 2002, Science.
[23] J. McArdle,et al. Convergence of pre- and postsynaptic influences on glucosensing neurons in the ventromedial hypothalamic nucleus. , 2001, Diabetes.
[24] Thomas D. Schmittgen,et al. Analysis of relative gene expression data using real-time quantitative PCR and the 2(-Delta Delta C(T)) Method. , 2001, Methods.
[25] F. Hanefeld,et al. Autosomal dominant Glut‐1 deficiency syndrome and familial epilepsy , 2001, Annals of neurology.
[26] J. Rawlins,et al. Cytotoxic lesions of the hippocampus increase social investigation but do not impair social-recognition memory , 2001, Experimental Brain Research.
[27] H. Joost,et al. The extended GLUT-family of sugar/polyol transport facilitators: nomenclature, sequence characteristics, and potential function of its novel members , 2001, Molecular membrane biology.
[28] S. Cushman,et al. Overexpression of the glucose transporter GLUT4 in adipose cells interferes with insulin‐stimulated translocation , 1999, FEBS letters.
[29] P. Magistretti,et al. Astrocytes Couple Synaptic Activity to Glucose Utilization in the Brain. , 1999, News in physiological sciences : an international journal of physiology produced jointly by the International Union of Physiological Sciences and the American Physiological Society.
[30] R. Schliebs,et al. Insulin‐sensitive GLUT4 glucose transporters are colocalized with GLUT3‐expressing cells and demonstrate a chemically distinct neuron‐specific localization in rat brain , 1999, Journal of neuroscience research.
[31] P. Magistretti,et al. Cellular mechanisms of brain energy metabolism and their relevance to functional brain imaging. , 1999, Philosophical transactions of the Royal Society of London. Series B, Biological sciences.
[32] V. Routh,et al. Brain glucose sensing and body energy homeostasis: role in obesity and diabetes. , 1999, American journal of physiology. Regulatory, integrative and comparative physiology.
[33] R G Shulman,et al. Energy on Demand , 1999, Science.
[34] E. Fisher,et al. Behavioral and functional analysis of mouse phenotype: SHIRPA, a proposed protocol for comprehensive phenotype assessment , 1997, Mammalian Genome.
[35] A. McCall,et al. Ultrastructural localization of GLUT 1 and GLUT 3 glucose transporters in rat brain , 1997, Journal of neuroscience research.
[36] P. Magistretti,et al. Glutamate uptake into astrocytes stimulates aerobic glycolysis: a mechanism coupling neuronal activity to glucose utilization. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[37] A. McCall,et al. Immunohistochemical localization of the neuron-specific glucose transporter (GLUT3) to neuropil in adult rat brain , 1994, Brain Research.
[38] P. Trayhurn,et al. Rapid chemiluminescent detection of facilitative glucose transporter mRNAs (GLUTs 1-4) with digoxigenin end-labelled oligonucleotides. , 1994, Biochemical Society transactions.
[39] P. Ferré,et al. Glucose transporter 2 (GLUT 2): expression in specific brain nuclei , 1994, Brain Research.
[40] G. Milligan,et al. Immunological analysis of glucose transporters expressed in different regions of the rat brain and central nervous system. , 1993, Biochemical and biophysical research communications.
[41] T. Bliss,et al. A synaptic model of memory: long-term potentiation in the hippocampus , 1993, Nature.
[42] Hans-Peter Lipp,et al. A new computer program for detailed off-line analysis of swimming navigation in the Morris water maze , 1992, Journal of Neuroscience Methods.
[43] S. Vannucci,et al. Expression of mouse-GLUT3 and human-GLUT3 glucose transporter proteins in brain. , 1992, Biochemical and biophysical research communications.
[44] L. Drewes,et al. Neurons and microvessels express the brain glucose transporter protein GLUT3. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[45] J. Kornhauser,et al. Glucose transporter expression in brain. cDNA sequence of mouse GLUT3, the brain facilitative glucose transporter isoform, and identification of sites of expression by in situ hybridization. , 1992, The Journal of biological chemistry.
[46] S. Harik,et al. Defective glucose transport across the blood-brain barrier as a cause of persistent hypoglycorrhachia, seizures, and developmental delay. , 1991, The New England journal of medicine.
[47] T. Wieloch. Hypoglycemia-induced neuronal damage prevented by an N-methyl-D-aspartate antagonist. , 1985, Science.
[48] R. Morris. Developments of a water-maze procedure for studying spatial learning in the rat , 1984, Journal of Neuroscience Methods.
[49] F. Goodwin,et al. Preliminary report of a simple animal behavior model for the anxiolytic effects of benzodiazepines , 1980, Pharmacology Biochemistry and Behavior.
[50] J. Noebels,et al. A mouse model for Glut1 haploinsufficiency , 2006 .
[51] E. McDermott,et al. Impaired neuronal glucose uptake in pathogenesis of schizophrenia - can GLUT 1 and GLUT 3 deficits explain imaging, post-mortem and pharmacological findings? , 2005, Medical hypotheses.
[52] R. Lister. The use of a plus-maze to measure anxiety in the mouse , 2004, Psychopharmacology.
[53] Conrad C. Huang,et al. BayGenomics: a resource of insertional mutations in mouse embryonic stem cells , 2003, Nucleic Acids Res..
[54] Bryan Kolb,et al. Analysis of Behavior in Laboratory Rodents , 1999 .