The role of calcium‐binding proteins in selective motoneuron vulnerability in amyotrophic lateral sclerosis
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V. La Bella | S. Appel | R. G. Smith | M. Alexianu | A. H. Mohamed | B. Ho
[1] E. Stefani,et al. Cytotoxicity of immunoglobulins from amyotrophic lateral sclerosis patients on a hybrid motoneuron cell line. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[2] E. Stefani,et al. Amyotrophic lateral sclerosis patient antibodies label Ca2+ channel α1 subunit , 1994 .
[3] D. Greenberg,et al. Calcium channels and neuromuscular disease , 1994, Annals of neurology.
[4] R. Llinás,et al. IgG from amyotrophic lateral sclerosis patients increases current through P-type calcium channels in mammalian cerebellar Purkinje cells and in isolated channel protein in lipid bilayer. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[5] M. Pericak-Vance,et al. Amyotrophic lateral sclerosis and structural defects in Cu,Zn superoxide dismutase. , 1993, Science.
[6] C. Heizmann,et al. Parvalbumin and calbindin D‐28k in the human motor system and in motor neuron disease , 1993, Neuropathology and applied neurobiology.
[7] Charles Tator,et al. Source specificity of early calcium neurotoxicity in cultured embryonic spinal neurons , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[8] M. Harding,et al. Calbindin D28K expression in transfected mouse NIH3T3 cells. , 1993, Cell calcium.
[9] J. Haines,et al. Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis , 1993, Nature.
[10] E. Stefani,et al. The action of amyotrophic lateral sclerosis immunoglobulins on mammalian single skeletal muscle Ca2+ channels. , 1993, The Journal of physiology.
[11] J. Dubinsky. Intracellular calcium levels during the period of delayed excitotoxicity , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[12] E. Stefani,et al. Serum antibodies to L-type calcium channels in patients with amyotrophic lateral sclerosis. , 1992, The New England journal of medicine.
[13] P. Emson,et al. Stable transfection of calbindin-D28k into the GH3 cell line alters calcium currents and intracellular calcium homeostasis , 1992, Neuron.
[14] E. Stefani,et al. A novel N18TG2 x mesencephalon cell hybrid expresses properties that suggest a dopaminergic cell line of substantia nigra origin , 1992, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[15] K. Baimbridge,et al. Calcium-binding proteins in the nervous system , 1992, Trends in Neurosciences.
[16] C. Heizmann,et al. Changes in Ca2+-binding proteins in human neurodegenerative disorders , 1992, Trends in Neurosciences.
[17] J. Rothstein,et al. Decreased glutamate transport by the brain and spinal cord in amyotrophic lateral sclerosis. , 1992, The New England journal of medicine.
[18] D. German,et al. Calbindin-D28K-containing neurons in animal models of neurodegeneration: possible protection from excitotoxicity. , 1992, Brain research. Molecular brain research.
[19] T. Vanaman,et al. In vitro enzyme activation with calbindin‐D28k, the vitamin D‐dependent 28 kDa calcium binding protein , 1992, FEBS letters.
[20] E. Stefani,et al. Calcium current and charge movement of mammalian muscle: action of amyotrophic lateral sclerosis immunoglobulins. , 1991, The Journal of physiology.
[21] R. Llinás,et al. Localization of P-type calcium channels in the central nervous system. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[22] M. Mattson,et al. Evidence for calcium-reducing and excitoprotective roles for the calcium-binding protein calbindin-1328k in cultured hippocampal neurons , 1991, Neuron.
[23] E. Stefani,et al. Immunoglobulins from animal models of motor neuron disease and from human amyotrophic lateral sclerosis patients passively transfer physiological abnormalities to the neuromuscular junction. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[24] A. Means,et al. Regulatory functions of calmodulin. , 1991, Pharmacology & therapeutics.
[25] M. Celio,et al. Calbindin D-28k and parvalbumin in the rat nervous system , 1990, Neuroscience.
[26] P. Emson,et al. Ontological study of calbindin‐D28k‐like and parvalbumin‐like immunoreactivities in rat spinal cord and dorsal root ganglia , 1990, The Journal of comparative neurology.
[27] P. Emson,et al. Expression of calbindin D-28K-like immunoreactivity in human SK-N-SH and SH-SY-5Y neuroblastoma cells , 1990, Brain Research.
[28] Ryoichi Shiozawa,et al. Development of ophthalmoplegia in amyotrophic lateral sclerosis during long-term use of respirators , 1990, Journal of the Neurological Sciences.
[29] A. Norman,et al. Monoclonal antibodies directed against the calcium binding protein Calbindin D-28k. , 1990, Cell calcium.
[30] S. Christakos,et al. Specific reduction of calcium-binding protein (28-kilodalton calbindin-D) gene expression in aging and neurodegenerative diseases. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[31] P. Emson,et al. Calbindin-immunoreactive cholinergic neurones in the nucleus basalis of Meynert in Alzheimer-type dementia , 1989, Brain Research.
[32] P. Cerutti,et al. Calcium chelator Quin 2 prevents hydrogen-peroxide-induced DNA breakage and cytotoxicity. , 1989, European journal of biochemistry.
[33] D. Choi. Calcium-mediated neurotoxicity: relationship to specific channel types and role in ischemic damage , 1988, Trends in Neurosciences.
[34] A. Plaitakis,et al. The neuroexcitotoxic amino acids glutamate and aspartate are altered in the spinal cord and brain in amyotrophic lateral sclerosis , 1988, Annals of neurology.
[35] W. Hunziker,et al. Rat brain calbindin D28: six domain structure and extensive amino acid homology with chicken calbindin D28. , 1988, Molecular endocrinology.
[36] C. Gerday,et al. Monoclonal antibodies directed against the calcium binding protein parvalbumin. , 1988, Cell calcium.
[37] Yasuo Kawaguchi,et al. Fast spiking cells in rat hippocampus (CA1 region) contain the calcium-binding protein parvalbumin , 1987, Brain Research.
[38] A. Means,et al. Isolation of a rat parvalbumin gene and full length cDNA. , 1986, The Journal of biological chemistry.
[39] O. Steward,et al. Distribution and subcellular localization of calmodulin in adult and developing brain tissue , 1983, Neuroscience.
[40] C. Heizmann,et al. Calcium-binding protein parvalbumin as a neuronal marker , 1981, Nature.
[41] S. Hsu,et al. Use of avidin-biotin-peroxidase complex (ABC) in immunoperoxidase techniques: a comparison between ABC and unlabeled antibody (PAP) procedures. , 1981, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[42] K. Nagashima,et al. Preservation of a certain motoneurone group of the sacral cord in amyotrophic lateral sclerosis: its clinical significance. , 1977, Journal of neurology, neurosurgery, and psychiatry.
[43] M. Showe,et al. Bacteriophage T4 prehead proteinase. II. Its cleavage from the product of gene 21 and regulation in phage-infected cells. , 1976, Journal of molecular biology.
[44] M. Nirenberg,et al. Genetic dissection of neural properties using somatic cell hybrids. , 1972, Nature: New biology.
[45] U. K. Laemmli,et al. Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4 , 1970, Nature.