Suppression of Bim by microRNA-19a may protect cardiomyocytes against hypoxia-induced cell death via autophagy activation.

[1]  D. Plas,et al.  TRPM3 and miR-204 establish a regulatory circuit that controls oncogenic autophagy in clear cell renal cell carcinoma. , 2014, Cancer cell.

[2]  H. Duan,et al.  Regulator of Calcineurin 1-1L Protects Cardiomyocytes Against Hypoxia-induced Apoptosis via Mitophagy , 2014, Journal of cardiovascular pharmacology.

[3]  Kimberley M. Smith,et al.  MicroRNAs control the apoptotic threshold in primed pluripotent stem cells through regulation of BIM , 2014, Genes & development.

[4]  K. Basso,et al.  microRNA 28 controls cell proliferation and is down-regulated in B-cell lymphomas , 2014, Proceedings of the National Academy of Sciences.

[5]  Richard W. Kriwacki,et al.  Many players in BCL-2 family affairs. , 2014, Trends in biochemical sciences.

[6]  P. Panda,et al.  Autophagy and apoptosis: where do they meet? , 2014, Apoptosis.

[7]  Eric H. Baehrecke,et al.  Self-consumption: the interplay of autophagy and apoptosis , 2014, Nature Reviews Molecular Cell Biology.

[8]  I. Rigoutsos,et al.  The miR-17/92 cluster: a comprehensive update on its genomics, genetics, functions and increasingly important and numerous roles in health and disease , 2013, Cell Death and Differentiation.

[9]  Yuejin Yang,et al.  The cardioprotection of simvastatin in reperfused swine hearts relates to the inhibition of myocardial edema by modulating aquaporins via the PKA pathway. , 2013, International journal of cardiology.

[10]  J. Ge,et al.  Glucocorticoid ameliorates early cardiac dysfunction after coronary microembolization and suppresses TGF-β1/Smad3 and CTGF expression. , 2013, International journal of cardiology.

[11]  Yaoliang Tang,et al.  MiR-17-92 cluster is a novel regulatory gene of cardiac ischemic/reperfusion injury. , 2013, Medical hypotheses.

[12]  M. Chopp,et al.  MicroRNA-17-92 Cluster Mediates the Proliferation and Survival of Neural Progenitor Cells after Stroke* , 2013, The Journal of Biological Chemistry.

[13]  Ø. Bruserud,et al.  PTEN-regulated AKT/FoxO3a/Bim signaling contributes to reactive oxygen species-mediated apoptosis in selenite-treated colorectal cancer cells , 2013, Cell Death and Disease.

[14]  D. Rubinsztein,et al.  BCL2L11/BIM , 2013, Autophagy.

[15]  D. Rubinsztein,et al.  Bim Inhibits Autophagy by Recruiting Beclin 1 to Microtubules , 2012, Molecular cell.

[16]  S. Grant,et al.  Inhibition of Bcl-2 antiapoptotic members by obatoclax potently enhances sorafenib-induced apoptosis in human myeloid leukemia cells through a Bim-dependent process. , 2012, Blood.

[17]  J. Huot,et al.  miR-20a represses endothelial cell migration by targeting MKK3 and inhibiting p38 MAP kinase activation in response to VEGF , 2012, Angiogenesis.

[18]  Mark Ziemann,et al.  Microparticles: major transport vehicles for distinct microRNAs in circulation , 2012, Cardiovascular research.

[19]  T. Crook,et al.  Paclitaxel resistance is associated with switch from apoptotic to autophagic cell death in MCF-7 breast cancer cells , 2012, Cell Death and Disease.

[20]  Yuejin Yang,et al.  No-reflow disrupts the expression and distribution of connexin 43 in a swine model. , 2011, Microvascular research.

[21]  B. Schroen,et al.  MicroRNA-18 and microRNA-19 regulate CTGF and TSP-1 expression in age-related heart failure , 2011, Aging cell.

[22]  A. Thorburn,et al.  Autophagy and apoptosis: what is the connection? , 2011, Trends in cell biology.

[23]  G. Stone,et al.  The relationship between attenuated plaque identified by intravascular ultrasound and no-reflow after stenting in acute myocardial infarction: the HORIZONS-AMI (Harmonizing Outcomes With Revascularization and Stents in Acute Myocardial Infarction) trial. , 2011, JACC. Cardiovascular interventions.

[24]  G. Cohen,et al.  The in vitro cleavage of the hAtg proteins by cell death proteases , 2010, Autophagy.

[25]  Guido Kroemer,et al.  Autophagy and the integrated stress response. , 2010, Molecular cell.

[26]  Stefanie Dimmeler,et al.  Circulating MicroRNAs in Patients With Coronary Artery Disease , 2010, Circulation research.

[27]  R. Virmani,et al.  Microvascular obstruction: underlying pathophysiology and clinical diagnosis. , 2010, Journal of the American College of Cardiology.

[28]  J. Pouysségur,et al.  Hypoxia-induced autophagy: cell death or cell survival? , 2010, Current opinion in cell biology.

[29]  K. Kersse,et al.  Caspase-mediated cleavage of Beclin-1 inactivates Beclin-1-induced autophagy and enhances apoptosis by promoting the release of proapoptotic factors from mitochondria , 2010, Cell Death and Disease.

[30]  F. Burzotta,et al.  Myocardial no-reflow in humans. , 2009, Journal of the American College of Cardiology.

[31]  W. Rottbauer,et al.  MicroRNA-21 contributes to myocardial disease by stimulating MAP kinase signalling in fibroblasts , 2008, Nature.

[32]  G. Kroemer,et al.  Autophagic cell death: the story of a misnomer , 2008, Nature Reviews Molecular Cell Biology.

[33]  R. Jaffe,et al.  Microvascular Obstruction and the No-Reflow Phenomenon After Percutaneous Coronary Intervention , 2008, Circulation.

[34]  J. Mendell.  miRiad Roles for the miR-17-92 Cluster in Development and Disease , 2008, Cell.

[35]  F. Fedele,et al.  The extent of microvascular damage during myocardial contrast echocardiography is superior to other known indexes of post-infarct reperfusion in predicting left ventricular remodeling: results of the multicenter AMICI study. , 2008, Journal of the American College of Cardiology.

[36]  Z. Jing,et al.  Post‐infarction treatment with simvastatin reduces myocardial no‐reflow by opening of the KATP channel , 2007, European journal of heart failure.

[37]  Shao-Cong Sun,et al.  Negative Regulation of JNK Signaling by the Tumor Suppressor CYLD* , 2004, Journal of Biological Chemistry.

[38]  Hiroyuki Tagawa,et al.  Identification and characterization of a novel gene, C13orf25, as a target for 13q31-q32 amplification in malignant lymphoma. , 2004, Cancer research.

[39]  G. Parodi,et al.  Impact of Microvascular Dysfunction on Left Ventricular Remodeling and Long-Term Clinical Outcome After Primary Coronary Angioplasty for Acute Myocardial Infarction , 2004, Circulation.

[40]  D. Bartel.  MicroRNAs Genomics, Biogenesis, Mechanism, and Function , 2004, Cell.

[41]  K. Lei,et al.  JNK phosphorylation of Bim-related members of the Bcl2 family induces Bax-dependent apoptosis , 2003, Proceedings of the National Academy of Sciences of the United States of America.

[42]  A. Tolkovsky,et al.  Mitochondrial disappearance from cells: a clue to the role of autophagy in programmed cell death and disease? , 2002, Biochimie.

[43]  E R McVeigh,et al.  Magnitude and time course of microvascular obstruction and tissue injury after acute myocardial infarction. , 1998, Circulation.

[44]  R. Kloner,et al.  The "no-reflow" phenomenon after temporary coronary occlusion in the dog. , 1974, The Journal of clinical investigation.

[45]  Jin Ock Kim,et al.  The miR-19a/b family positively regulates cardiomyocyte hypertrophy by targeting atrogin-1 and MuRF-1. , 2014, The Biochemical journal.

[46]  P. Codogno,et al.  Does autophagy have a license to kill mammalian cells? , 2009, Cell Death and Differentiation.