Electrical method for detection of endothelial cell shape change in real time: assessment of endothelial barrier function.
暂无分享,去创建一个
A. Malik | C. Tiruppathì | I. Giaever | C. Keese | C R Keese | I Giaever | P. del Vecchio | A B Malik | C Tiruppathi | P J Del Vecchio | P. Vecchio | Charles R. KEESEt | Ivar GIAEVERt | C. Tiruppathi
[1] J. Cooper,et al. Selectivity of the endothelial monolayer: effects of increased permeability. , 1988, Microvascular research.
[2] T. Tamaoki,et al. Calphostin C (UCN-1028C), a novel microbial compound, is a highly potent and specific inhibitor of protein kinase C. , 1989, Biochemical and biophysical research communications.
[3] G. Hunninghake,et al. Reversible oxidant-induced increases in albumin transfer across cultured endothelium: alterations in cell shape and calcium homeostasis. , 1985, Blood.
[4] J. A. Oliver. Adenylate cyclase and protein kinase C mediate opposite actions on endothelial junctions , 1990, Journal of cellular physiology.
[5] R. Bizios,et al. Thrombin‐induced increase in albumin permeability across the endothelium , 1986, Journal of cellular physiology.
[6] M. Aschner,et al. Enzymatic activity is necessary for thrombin-mediated increase in endothelial permeability. , 1990, The American journal of physiology.
[7] P. de Lanerolle,et al. Regulation of embryonic smooth muscle myosin by protein kinase C. , 1988, The Journal of biological chemistry.
[8] J. M. Mullin,et al. Effects of tumor promoters on LLC-PK1 renal epithelial tight junctions and transepithelial fluxes. , 1986, The American journal of physiology.
[9] C. Huang,et al. Vimentin, a cytoskeletal substrate of protein kinase C. , 1988, Biochemical and biophysical research communications.
[10] A. Malik,et al. Increased endothelial albumin permeability mediated by protein kinase C activation. , 1990, The Journal of clinical investigation.
[11] J. Howbert,et al. Inhibition of protein kinase C by calphostin C is light-dependent. , 1991, Biochemical and biophysical research communications.
[12] V. V. van Hinsbergh,et al. Norepinephrine and iloprost improve barrier function of human endothelial cell monolayers: role of cAMP. , 1991, The American journal of physiology.
[13] L. McIntire,et al. Permeability of human endothelial monolayers: effect of vasoactive agonists and cAMP. , 1989, Journal of applied physiology.
[14] S. Evensen,et al. Thrombin-induced shape changes of cultured endothelial cells: metabolic and functional observations. , 1983, Thrombosis research.
[15] P. Braquet,et al. Human endothelial cells are target for platelet-activating factor. I. Platelet-activating factor induces changes in cytoskeleton structures. , 1987, Journal of immunology.
[16] I. Giaever,et al. Monitoring fibroblast behavior in tissue culture with an applied electric field. , 1984, Proceedings of the National Academy of Sciences of the United States of America.
[17] M. Laposata,et al. Thrombin-induced gap formation in confluent endothelial cell monolayers in vitro. , 1983, Blood.
[18] J. Pouysségur,et al. cDNA cloning and expression of a hamster α‐thrombin receptor coupled to Ca2+ mobilization , 1991 .
[19] I. Pastan,et al. Vinculin, a cytoskeletal substrate of protein kinase C. , 1983, The Journal of biological chemistry.
[20] J. Weil,et al. Role of cyclic adenosine monophosphate in the induction of endothelial barrier properties , 1989, Journal of cellular physiology.
[21] D. Rotrosen,et al. Histamine type I receptor occupancy increases endothelial cytosolic calcium, reduces F-actin, and promotes albumin diffusion across cultured endothelial monolayers , 1986, The Journal of cell biology.
[22] C. Turner,et al. Focal adhesions: transmembrane junctions between the extracellular matrix and the cytoskeleton. , 1988, Annual review of cell biology.
[23] R. Bizios,et al. Endothelial monolayer permeability to macromolecules. , 1987, Federation proceedings.
[24] B. Hemmings,et al. Thrombin causes neurite retraction in neuronal cells through activation of cell surface receptors , 1992, Neuron.
[25] V. Wheaton,et al. Molecular cloning of a functional thrombin receptor reveals a novel proteolytic mechanism of receptor activation , 1991, Cell.
[26] I. Giaever,et al. Electric measurements can be used to monitor the attachment and spreading of cells in tissue culture. , 1991, BioTechniques.
[27] G. Ojakian. Tumor promoter-induced changes in the permeability of epithelial cell tight junctions , 1981, Cell.
[28] J. Cooper,et al. Endothelial barrier function. , 1989, The Journal of investigative dermatology.
[29] D. Lawrence,et al. Measurement of macrophage adherence and spreading with weak electric fields. , 1990, Journal of immunological methods.
[30] I. Giaever,et al. Micromotion of mammalian cells measured electrically. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[31] Ivar Giaever,et al. Use of Electric Fields to Monitor the Dynamical Aspect of Cell Behavior in Tissue Culture , 1986, IEEE Transactions on Biomedical Engineering.
[32] N. Martel,et al. Tumour promoter-mediated reversible inhibition of cell-cell communication (electrical coupling). Relationship with phorbol ester binding and de novo macromolecule synthesis. , 1983, Experimental cell research.
[33] C. Crone. Modulation of solute permeability in microvascular endothelium. , 1986, Federation proceedings.