Nonconvulsive seizures in subarachnoid hemorrhage link inflammation and outcome

Nonconvulsive seizures (NCSz) are frequent following acute brain injury and have been implicated as a cause of secondary brain injury, but mechanisms that cause NCSz are controversial. Proinflammatory states are common after many brain injuries, and inflammation‐mediated changes in blood–brain barrier permeability have been experimentally linked to seizures.

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