β1-Adrenoceptor autoimmunity in cardiomyopathy
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[1] G. Wallukat,et al. High prevalence of antibodies against beta 1- and beta 2-adrenoceptors in patients with primary electrical cardiac abnormalities. , 1995, Journal of the American College of Cardiology.
[2] G. Wallukat,et al. Molecular mimicry between the immunodominant ribosomal protein P0 of Trypanosoma cruzi and a functional epitope on the human beta 1- adrenergic receptor , 1995, The Journal of experimental medicine.
[3] G. Wallukat,et al. Anti-beta 1-adrenoceptor autoantibodies with chronotropic activity from the serum of patients with dilated cardiomyopathy: mapping of epitopes in the first and second extracellular loops. , 1995, Journal of molecular and cellular cardiology.
[4] T. Hedner,et al. Autoantibodies against cardiac G-protein-coupled receptors define different populations with cardiomyopathies but not with hypertension. , 1994, Clinical immunology and immunopathology.
[5] G. Wallukat,et al. Autoimmunity in idiopathic dilated cardiomyopathy. Characterization of antibodies against the beta 1-adrenoceptor with positive chronotropic effect. , 1994, Circulation.
[6] M. B. Rosenbaum,et al. Antibodies to Beta‐Adrenergic Receptors Disclosing Agonist‐Like Properties in Idiopathic Dilated Cardiomyopathy and Chagas’ Heart Disease , 1994, Journal of cardiovascular electrophysiology.
[7] R. Graham,et al. The high affinity state of the beta 2-adrenergic receptor requires unique interaction between conserved and non-conserved extracellular loop cysteines. , 1994, The Journal of biological chemistry.
[8] J. Hoebeke,et al. Epitope analysis of T- and B-cell response against the human β1-adrenoceptor , 1994 .
[9] K. Swedberg,et al. Beneficial effects of metoprolol in idiopathic dilated cardiomyopathy , 1993, The Lancet.
[10] F. Waagstein,et al. Localization of a functional autoimmune epitope on the muscarinic acetylcholine receptor-2 in patients with idiopathic dilated cardiomyopathy. , 1993, The Journal of clinical investigation.
[11] I. Goldenberg,et al. Assessment of immune modulation of beta-adrenergic pathways in human dilated cardiomyopathy: influence of methodologic factors. , 1992, American heart journal.
[12] G. Wallukat,et al. Functional analysis of rabbit anti-peptide antibodies which mimic autoantibodies against the beta 1-adrenergic receptor in patients with idiopathic dilated cardiomyopathy. , 1991, Journal of Autoimmunity.
[13] G. Wallukat,et al. Autoantibodies against the β-adrenergic receptor in human myocarditis and dilated cardiomyopathy: β-adrenergic agonism without desensitization , 1991 .
[14] A. Strosberg,et al. Mapping of a functional autoimmune epitope on the beta 1-adrenergic receptor in patients with idiopathic dilated cardiomyopathy. , 1990, The Journal of clinical investigation.
[15] I. Goldenberg,et al. Influence of anti-beta-receptor antibodies on cardiac adenylate cyclase in patients with idiopathic dilated cardiomyopathy. , 1990, American heart journal.
[16] M. Olivari,et al. Anti-beta-receptor antibodies in human dilated cardiomyopathy and correlation with HLA-DR antigens. , 1990, The American journal of cardiology.
[17] A. Strosberg,et al. Antigenic analysis of the second extra-cellular loop of the human beta-adrenergic receptors. , 1989, Clinical and experimental immunology.
[18] K Caidahl,et al. Long-term beta-blockade in dilated cardiomyopathy. Effects of short- and long-term metoprolol treatment followed by withdrawal and readministration of metoprolol. , 1989, Circulation.
[19] I. Goldenberg,et al. Autoantibodies against beta-adrenoceptors in human idiopathic dilated cardiomyopathy. , 1989, Circulation research.
[20] G. Wallukat,et al. Effects of the serum gamma globulin fraction of patients with allergic asthma and dilated cardiomyopathy on chronotropic beta adrenoceptor function in cultured neonatal rat heart myocytes. , 1987, Biomedica biochimica acta.
[21] E. Borda,et al. A circulating IgG in Chagas' disease which binds to beta-adrenoceptors of myocardium and modulates their activity. , 1984, Clinical and experimental immunology.