Sulforaphane induces inhibition of human umbilical vein endothelial cells proliferation by apoptosis
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H. Nagawa | J. Kitayama | N. Tsuno | Koki Takahashi | J. Yamada | S. Kaisaki | T. Osada | Y. Okaji | M. Asakage | T. Tsuchiya | S. Yoneyama
[1] H. Bartsch,et al. Inhibition of angiogenesis and endothelial cell functions are novel sulforaphane-mediated mechanisms in chemoprevention , 2006, Molecular Cancer Therapeutics.
[2] H. Nagawa,et al. Selective inhibition of cyclooxygenase (COX)‐2 inhibits endothelial cell proliferation by induction of cell cycle arrest , 2005, International journal of cancer.
[3] H. Nagawa,et al. 3-Hydroxy-3-methylglutaryl-coenzyme A reductase inhibitor (pravastatin) inhibits endothelial cell proliferation dependent on G1 cell cycle arrest , 2004, Anti-cancer drugs.
[4] S. Srivastava,et al. Sulforaphane-induced G2/M Phase Cell Cycle Arrest Involves Checkpoint Kinase 2-mediated Phosphorylation of Cell Division Cycle 25C* , 2004, Journal of Biological Chemistry.
[5] P. Hrelia,et al. Cyclin D3 and p53 mediate sulforaphane-induced cell cycle delay and apoptosis in non-transformed human T lymphocytes , 2002, Cellular and Molecular Life Sciences CMLS.
[6] Kozo Nakamura,et al. p53AIP1 regulates the mitochondrial apoptotic pathway. , 2002, Cancer research.
[7] P. Hrelia,et al. Growth inhibition, cell-cycle arrest and apoptosis in human T-cell leukemia by the isothiocyanate sulforaphane. , 2002, Carcinogenesis.
[8] T. Ahmed,et al. Sulforaphane and its metabolite mediate growth arrest and apoptosis in human prostate cancer cells. , 2002, International journal of oncology.
[9] T. Shapiro,et al. Quantitative determination of dithiocarbamates in human plasma, serum, erythrocytes and urine: pharmacokinetics of broccoli sprout isothiocyanates in humans. , 2002, Clinica chimica acta; international journal of clinical chemistry.
[10] A. Dinkova-Kostova,et al. Powerful and prolonged protection of human retinal pigment epithelial cells, keratinocytes, and mouse leukemia cells against oxidative damage: The indirect antioxidant effects of sulforaphane , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[11] Lingxiang Ye,et al. Total intracellular accumulation levels of dietary isothiocyanates determine their activity in elevation of cellular glutathione and induction of Phase 2 detoxification enzymes. , 2001, Carcinogenesis.
[12] J. Hayes,et al. Dietary indoles and isothiocyanates that are generated from cruciferous vegetables can both stimulate apoptosis and confer protection against DNA damage in human colon cell lines. , 2001, Cancer research.
[13] A. Whittemore,et al. Vegetables, fruits, legumes and prostate cancer: a multiethnic case-control study. , 2000, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.
[14] W. Willett,et al. Intakes of fruits, vegetables, and related nutrients and the risk of non-Hodgkin's lymphoma among women. , 2000, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.
[15] Y Li,et al. [Mitochondria and apoptosis]. , 2000, Zhonghua yu fang yi xue za zhi [Chinese journal of preventive medicine].
[16] Pengfei Li,et al. Sulforaphane, a naturally occurring isothiocyanate, induces cell cycle arrest and apoptosis in HT29 human colon cancer cells. , 2000, Cancer research.
[17] J. Stanford,et al. Fruit and vegetable intakes and prostate cancer risk. , 2000, Journal of the National Cancer Institute.
[18] S. Hecht,et al. INHIBITION OF CARCINOGENESIS BY ISOTHIOCYANATES* , 2000, Drug metabolism reviews.
[19] T. Tsuruo,et al. Inhibition of haematogenous metastasis of colon cancer in mice by a selective COX-2 inhibitor, JTE-522 , 1999, British Journal of Cancer.
[20] Masashi Narita,et al. Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC , 1999, Nature.
[21] S. Hecht,et al. Chemoprevention of cancer by isothiocyanates, modifiers of carcinogen metabolism. , 1999, The Journal of nutrition.
[22] P. Talalay,et al. Mechanism of differential potencies of isothiocyanates as inducers of anticarcinogenic Phase 2 enzymes. , 1998, Cancer research.
[23] J. Hankin,et al. Total Isothiocyanate Contents in Cooked Vegetables Frequently Consumed in Singapore , 1998 .
[24] J. Fahey,et al. Broccoli sprouts: an exceptionally rich source of inducers of enzymes that protect against chemical carcinogens. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[25] Douglas Hanahan,et al. Signaling Vascular Morphogenesis and Maintenance , 1997, Science.
[26] L. Kohlmeier,et al. Controversies surrounding diet and breast cancer , 1997, Proceedings of the Nutrition Society.
[27] D. Green,et al. The Release of Cytochrome c from Mitochondria: A Primary Site for Bcl-2 Regulation of Apoptosis , 1997, Science.
[28] P. Talalay,et al. Chemoprotection against cancer by isothiocyanates and glucosinolates. , 1996, Biochemical Society transactions.
[29] P. Talalay,et al. Electrophile and antioxidant regulation of enzymes that detoxify carcinogens. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[30] T. Kensler,et al. Anticarcinogenic activities of sulforaphane and structurally related synthetic norbornyl isothiocyanates. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[31] C. Cho,et al. A major inducer of anticarcinogenic protective enzymes from broccoli: isolation and elucidation of structure. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[32] L. Wattenberg. Inhibitory effects of benzyl isothiocyanate administered shortly before diethylnitrosamine or benzo[a]pyrene on pulmonary and forestomach neoplasia in A/J mice. , 1987, Carcinogenesis.
[33] S. Amin,et al. Inhibition of benzo(a)pyrene-induced lung tumorigenesis in A/J mice by dietary N-acetylcysteine conjugates of benzyl and phenethyl isothiocyanates during the postinitiation phase is associated with activation of mitogen-activated protein kinases and p53 activity and induction of apoptosis. , 2002, Cancer research.
[34] J. Fahey,et al. The chemical diversity and distribution of glucosinolates and isothiocyanates among plants. , 2001, Phytochemistry.
[35] J. Folkman. Angiogenesis in cancer, vascular, rheumatoid and other disease , 1995, Nature Medicine.