Cooperative activation of IP3 receptors by sequential binding of IP3 and Ca2+ safeguards against spontaneous activity
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[1] S. Györke,et al. Activation of single cardiac and skeletal ryanodine receptor channels by flash photolysis of caged Ca2+. , 1994, Biophysical journal.
[2] Rapid filtration studies of the effect of cytosolic Ca2+ on inositol 1,4,5-trisphosphate-induced 45Ca2+ release from cerebellar microsomes. , 1994, The Journal of biological chemistry.
[4] James Watras,et al. Bell-shaped calcium-response curves of lns(l,4,5)P3- and calcium-gated channels from endoplasmic reticulum of cerebellum , 1991, Nature.
[5] L. Combettes,et al. Rapid kinetics of myo-inositol trisphosphate binding and dissociation in cerebellar microsomes. , 1994, The Journal of biological chemistry.
[6] M B Jackson,et al. Perfection of a synaptic receptor: kinetics and energetics of the acetylcholine receptor. , 1989, Proceedings of the National Academy of Sciences of the United States of America.
[7] R. Wojcikiewicz,et al. Type I, II, and III inositol 1,4,5-trisphosphate receptors are unequally susceptible to down-regulation and are expressed in markedly different proportions in different cell types , 1995, The Journal of Biological Chemistry.
[8] C. Taylor,et al. Effects of Ca2+ chelators on purified inositol 1,4,5-trisphosphate (InsP3) receptors and InsP3-stimulated Ca2+ mobilization. , 1993, The Journal of biological chemistry.
[9] I. Parker,et al. Elementary events of InsP3-induced Ca2+ liberation in Xenopus oocytes: hot spots, puffs and blips. , 1996, Cell calcium.
[10] John M. Zempel,et al. How quickly can GABAA receptors open? , 1994, Neuron.
[11] M. Berridge. Relationship between latency and period for 5-hydroxytryptamine-induced membrane responses in the Calliphora salivary gland. , 1994, The Biochemical journal.
[12] Henry R. Bourne,et al. Molecular machines integrate coincident synaptic signals , 1993, Cell.
[13] C. Jahr. NMDA receptor kinetics and synaptic function , 1994 .
[14] I. Bezprozvanny,et al. Inositol 1,4,5-trisphosphate-gated channels in cerebellum: presence of multiple conductance states , 1991, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[15] I. Bezprozvanny,et al. Inositol (1,4,5)-trisphosphate (InsP3)-gated Ca channels from cerebellum: conduction properties for divalent cations and regulation by intraluminal calcium , 1994, The Journal of general physiology.
[16] I. Parker,et al. Inhibition by Ca2+ of inositol trisphosphate-mediated Ca2+ liberation: a possible mechanism for oscillatory release of Ca2+. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[17] I. Parker,et al. Fast kinetics of calcium liberation induced in Xenopus oocytes by photoreleased inositol trisphosphate. , 1996, Biophysical journal.
[18] L. Pearce,et al. A superfusion system designed to measure release of radiolabeled neurotransmitters on a subsecond time scale. , 1989, Analytical biochemistry.
[19] S. M. Goldin,et al. Calcium as a coagonist of inositol 1,4,5-trisphosphate-induced calcium release. , 1991, Science.
[20] J. D. Clements,et al. Activation kinetics reveal the number of glutamate and glycine binding sites on the N-methyl-d-aspartate receptor , 1991, Neuron.
[21] K. Khodakhah,et al. Fast activation and inactivation of inositol trisphosphate‐evoked Ca2+ release in rat cerebellar Purkinje neurones. , 1995, The Journal of physiology.
[22] C. Taylor,et al. Luminal Ca2+ increases the sensitivity of Ca2+ stores to inositol 1,4,5-trisphosphate. , 1992, Molecular pharmacology.
[23] G. Hajnóczky,et al. The inositol trisphosphate calcium channel is inactivated by inositol trisphosphate , 1994, Nature.
[24] J. Dufour,et al. Inositol 1,4,5-Trisphosphate and Calcium Regulate the Calcium Channel Function of the Hepatic Inositol 1,4,5-Trisphosphate Receptor* , 1997, The Journal of Biological Chemistry.
[25] M. Iino,et al. Biphasic Ca2+ dependence of inositol 1,4,5-trisphosphate-induced Ca release in smooth muscle cells of the guinea pig taenia caeci , 1990, The Journal of general physiology.
[26] M. Berridge,et al. Elementary and global aspects of calcium signalling. , 1997, The Journal of experimental biology.
[27] D. Baylor,et al. Gating kinetics of the cyclic-GMP-activated channel of retinal rods: flash photolysis and voltage-jump studies. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[28] L. Stryer,et al. Kinetics of calcium channel opening by inositol 1,4,5-trisphosphate. , 1990, Biochemistry.
[29] I. Bezprozvanny,et al. ATP modulates the function of inositol 1,4,5-trisphosphate-gated channels at two sites , 1993, Neuron.
[30] S. M. Goldin,et al. Exocytosis, Calcium Oscillations, and Novel Glutamate Release Blockers as Resolved by Rapid Superfusion a , 1994, Annals of the New York Academy of Sciences.
[31] L. Stryer,et al. Range of messenger action of calcium ion and inositol 1,4,5-trisphosphate. , 1992, Science.
[32] S Swillens,et al. Transient inositol 1,4,5-trisphosphate-induced Ca2+ release: a model based on regulatory Ca(2+)-binding sites along the permeation pathway. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[33] C W Taylor. Why do hormones stimulate Ca2+ mobilization? , 1995, Biochemical Society transactions.
[34] D. Ogden,et al. Kinetics of intracellular calcium release by inositol 1, 4, 5-trisphosphate and extracellular ATP in porcine cultured aortic endothelial cells , 1992, Proceedings of the Royal Society of London. Series B: Biological Sciences.
[35] K. Miller,et al. High acetylcholine concentrations cause rapid inactivation before fast desensitization in nicotinic acetylcholine receptors from Torpedo. , 1988, Biophysical journal.
[36] T. Capiod,et al. Kinetics of the conductance evoked by noradrenaline, inositol trisphosphate or Ca2+ in guinea‐pig isolated hepatocytes. , 1990, The Journal of physiology.
[37] C. Taylor,et al. Regulation of inositol trisphosphate receptors by luminal Ca2+ contributes to quantal Ca2+ mobilization. , 1996, The EMBO journal.
[38] M. Foti,et al. Highly cooperative Ca2+ elevations in response to Ins(1,4,5)P3 microperfusion through a patch-clamp pipette. , 1995, Biophysical journal.
[39] C. Taylor,et al. Rapid kinetic measurements of 45Ca2+ mobilization reveal that Ins(2,4,5)P3 is a partial agonist at hepatic InsP3 receptors. , 1997, The Biochemical journal.
[40] Quantal responses to inositol 1,4,5-trisphosphate are not a consequence of Ca2+ regulation of inositol 1,4,5-trisphosphate receptors. , 1995, The Biochemical journal.
[41] A. Tripathy,et al. Sarcoplasmic reticulum lumenal Ca2+ has access to cytosolic activation and inactivation sites of skeletal muscle Ca2+ release channel. , 1996, Biophysical journal.
[42] É. Doucet,et al. Fast kinetics of calcium release induced by myo-inositol trisphosphate in permeabilized rat hepatocytes. , 1989, The Journal of biological chemistry.
[43] K. Mikoshiba,et al. Kinetics of Calcium Release by Immunoaffinity-purified Inositol 1,4,5-Trisphosphate Receptor in Reconstituted Lipid Vesicles (*) , 1995, The Journal of Biological Chemistry.
[44] Y. Liu,et al. Opening rate of acetylcholine receptor channels. , 1991, Biophysical journal.