Multiplexed Phosphoproteomic Study of Brain in Patients with Alzheimer's Disease and Age-Matched Cognitively Healthy Controls.
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M. Albert | A. Pandey | G. Sathe | J. Troncoso | A. Moghekar | Ankit P Jain | K. K. Mangalaparthi | Jacqueline A. Darrow | K. Mangalaparthi | A. Pandey | A. Pandey
[1] Simon Youssef,et al. Quantitative modeling of synthetic gene transfer , 2011 .
[2] M. Albert,et al. Quantitative Proteomic Profiling of Cerebrospinal Fluid to Identify Candidate Biomarkers for Alzheimer's Disease , 2019, Proteomics. Clinical applications.
[3] M. Albert,et al. Phosphotyrosine profiling of human cerebrospinal fluid , 2018, Clinical Proteomics.
[4] E. Diamandis,et al. Brain-related proteins as potential CSF biomarkers of Alzheimer's disease: A targeted mass spectrometry approach. , 2018, Journal of proteomics.
[5] A. Levey,et al. Global quantitative analysis of the human brain proteome in Alzheimer’s and Parkinson’s Disease , 2018, Scientific Data.
[6] Henrik Zetterberg,et al. Earliest accumulation of β-amyloid occurs within the default-mode network and concurrently affects brain connectivity , 2017, Nature Communications.
[7] K. Blennow,et al. Proteomic studies of cerebrospinal fluid biomarkers of Alzheimer’s disease: an update , 2017, Expert review of proteomics.
[8] A. Bartoš,et al. Neurofilaments and tau proteins in cerebrospinal fluid and serum in dementias and neuroinflammation. , 2017, Biomedical papers of the Medical Faculty of the University Palacky, Olomouc, Czechoslovakia.
[9] C. Smith,et al. Dual RNA Processing Roles of Pat1b via Cytoplasmic Lsm1-7 and Nuclear Lsm2-8 Complexes , 2017, Cell reports.
[10] L. Petrucelli,et al. An acetylation–phosphorylation switch that regulates tau aggregation propensity and function , 2017, The Journal of Biological Chemistry.
[11] Timothy A. Miller,et al. Phosphorylated neurofilament heavy chain: A biomarker of survival for C9ORF72‐associated amyotrophic lateral sclerosis , 2017, Annals of neurology.
[12] Giuseppe Troiano,et al. The crucial role of protein phosphorylation in cell signaling and its use as targeted therapy (Review) , 2017, International journal of molecular medicine.
[13] Hui Zhang,et al. MARCKS is Necessary for Oligodendrocyte Precursor Cell Maturation , 2017, Neurochemical Research.
[14] Jian-zhi Wang 王建枝,et al. Role of microtubule-associated protein tau phosphorylation in Alzheimer’s disease , 2017, Journal of Huazhong University of Science and Technology [Medical Sciences].
[15] H. Kiyonari,et al. CRMP1 and CRMP2 have synergistic but distinct roles in dendritic development , 2016, Genes to cells : devoted to molecular & cellular mechanisms.
[16] Marco Y. Hein,et al. The Perseus computational platform for comprehensive analysis of (prote)omics data , 2016, Nature Methods.
[17] S. Pinto,et al. Phosphotyrosine profiling of curcumin-induced signaling , 2016, Clinical Proteomics.
[18] Jian Cai,et al. Quantitative phosphoproteomic analyses of the inferior parietal lobule from three different pathological stages of Alzheimer's disease. , 2015, Journal of Alzheimer's disease : JAD.
[19] G. Halliday,et al. Aβ-dependent reduction of NCAM2-mediated synaptic adhesion contributes to synapse loss in Alzheimer's disease , 2015, Nature Communications.
[20] Fabiana A. Caetano,et al. Ca2+/Calmodulin-dependent protein Kinase II interacts with group I Metabotropic Glutamate and facilitates Receptor Endocytosis and ERK1/2 signaling: role of β-Amyloid , 2015, Molecular Brain.
[21] S. Ackerman,et al. Mutations in the Microtubule-Associated Protein 1A (Map1a) Gene Cause Purkinje Cell Degeneration , 2015, The Journal of Neuroscience.
[22] A. Levey,et al. Quantitative phosphoproteomics of Alzheimer's disease reveals cross‐talk between kinases and small heat shock proteins , 2015, Proteomics.
[23] Davide Heller,et al. STRING v10: protein–protein interaction networks, integrated over the tree of life , 2014, Nucleic Acids Res..
[24] M. Mann,et al. Minimal, encapsulated proteomic-sample processing applied to copy-number estimation in eukaryotic cells , 2014, Nature Methods.
[25] Joana M. Xavier,et al. MicroRNA-34a Modulates Neural Stem Cell Differentiation by Regulating Expression of Synaptic and Autophagic Proteins , 2014, Molecular Neurobiology.
[26] D. Mash,et al. Identification of the Sites of Tau Hyperphosphorylation and Activation of Tau Kinases in Synucleinopathies and Alzheimer’s Diseases , 2013, PloS one.
[27] Chadwick M. Hales,et al. U1 small nuclear ribonucleoprotein complex and RNA splicing alterations in Alzheimer’s disease , 2013, Proceedings of the National Academy of Sciences.
[28] Kim T. Blackwell,et al. Molecular mechanisms underlying neuronal synaptic plasticity: systems biology meets computational neuroscience in the wilds of synaptic plasticity , 2013, Wiley interdisciplinary reviews. Systems biology and medicine.
[29] Jenny Wong,et al. Altered Expression of RNA Splicing Proteins in Alzheimer's Disease Patients: Evidence from Two Microarray Studies , 2013, Dementia and Geriatric Cognitive Disorders Extra.
[30] M. Berridge. Dysregulation of neural calcium signaling in Alzheimer disease, bipolar disorder and schizophrenia , 2013, Prion.
[31] M. Oellerich,et al. Phosphoproteome profiling of substantia nigra and cortex regions of Alzheimer’s disease patients , 2012, Journal of neurochemistry.
[32] M. Gorospe,et al. RNA-binding protein nucleolin in disease , 2012, RNA biology.
[33] V. Berezin,et al. NCAM2/OCAM/RNCAM: cell adhesion molecule with a role in neuronal compartmentalization. , 2012, The international journal of biochemistry & cell biology.
[34] Akhilesh Pandey,et al. TSLP Signaling Network Revealed by SILAC-Based Phosphoproteomics* , 2012, Molecular & Cellular Proteomics.
[35] P. Pinton,et al. Protein Kinases and Phosphatases in the Control of Cell Fate , 2011, Enzyme research.
[36] A. Verkhratsky,et al. Neurogenesis in Alzheimer’s disease , 2011, Journal of anatomy.
[37] D. Selkoe. Alzheimer's disease. , 2011, Cold Spring Harbor perspectives in biology.
[38] Kanae Iijima-Ando,et al. Tau Ser262 phosphorylation is critical for Abeta42-induced tau toxicity in a transgenic Drosophila model of Alzheimer's disease. , 2010, Human molecular genetics.
[39] I. Grundke‐Iqbal,et al. Phosphorylation of Tau at Thr212, Thr231, and Ser262 Combined Causes Neurodegeneration* , 2010, The Journal of Biological Chemistry.
[40] W. Klein,et al. Deleterious Effects of Amyloid β Oligomers Acting as an Extracellular Scaffold for mGluR5 , 2010, Neuron.
[41] T. Pawson,et al. Post-translational modifications in signal integration , 2010, Nature Structural &Molecular Biology.
[42] T. Bayer,et al. Inflammatory changes are tightly associated with neurodegeneration in the brain and spinal cord of the APP/PS1KI mouse model of Alzheimer's disease , 2010, Neurobiology of Aging.
[43] J. Trojanowski,et al. Total and phosphorylated tau protein as biological markers of Alzheimer’s disease , 2010, Experimental Gerontology.
[44] Akhilesh Pandey,et al. Human Protein Reference Database and Human Proteinpedia as discovery tools for systems biology. , 2009, Methods in molecular biology.
[45] A. Verkhratsky,et al. Collapsin response mediator protein‐2 hyperphosphorylation is an early event in Alzheimer’s disease progression , 2007, Journal of neurochemistry.
[46] Mark A. Smith,et al. Increased expression of the remodeling- and tumorigenic-associated factor osteopontin in pyramidal neurons of the Alzheimer's disease brain. , 2007, Current Alzheimer research.
[47] S. Sisodia,et al. Presenilins and Alzheimer disease: the calcium conspiracy , 2006, Nature Neuroscience.
[48] P. Taupin. Neurogenesis in the adult central nervous system. , 2006, Comptes rendus biologies.
[49] E. Masliah,et al. Perturbed neurogenesis in the adult hippocampus associated with presenilin-1 A246E mutation. , 2005, The American journal of pathology.
[50] Lin Xie,et al. Enhanced neurogenesis in Alzheimer's disease transgenic (PDGF-APPSw,Ind) mice. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[51] Y. Yamauchi,et al. Proteomic Analysis of Human Nop56p-associated Pre-ribosomal Ribonucleoprotein Complexes , 2003, Journal of Biological Chemistry.
[52] T. Hunter,et al. The Protein Kinase Complement of the Human Genome , 2002, Science.
[53] S. Leurgans,et al. Tau Conformational Changes Correspond to Impairments of Episodic Memory in Mild Cognitive Impairment and Alzheimer's Disease , 2002, Experimental Neurology.
[54] B. Peculis,et al. Xenopus LSm Proteins Bind U8 snoRNA via an Internal Evolutionarily Conserved Octamer Sequence , 2002, Molecular and Cellular Biology.
[55] I. Grundke‐Iqbal,et al. Hyperphosphorylation induces self-assembly of τ into tangles of paired helical filaments/straight filaments , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[56] T. Südhof,et al. Synaptotagmin I functions as a calcium regulator of release probability , 2001, Nature.
[57] Hideyuki Yamamoto,et al. Phosphorylation of MARCKS in Alzheimer disease brains , 2000, Neuroreport.
[58] G. Bokoch,et al. Inhibition of myosin light chain kinase by p21-activated kinase. , 1999, Science.
[59] D. Schomburg,et al. Ca 2+ /calmodulin-dependent protein kinase , 1997 .
[60] J. Ávila,et al. Microtubule-associated protein MAP1B showing a fetal phosphorylation pattern is present in sites of neurofibrillary degeneration in brains of Alzheimer's disease patients. , 1994, Brain research. Molecular brain research.
[61] S. Yen,et al. Phosphate analysis and dephosphorylation of modified tau associated with paired helical filaments , 1992, Brain Research.
[62] P. Janmey,et al. MARCKS is an actin filament crosslinking protein regulated by protein kinase C and calcium–calmodulin , 1992, Nature.
[63] T. Iwaki,et al. Hydrated autoclave pretreatment enhances tau immunoreactivity in formalin-fixed normal and Alzheimer's disease brain tissues. , 1991, Laboratory investigation; a journal of technical methods and pathology.
[64] L. Sternberger,et al. Aberrant neurofilament phosphorylation in Alzheimer disease. , 1985, Proceedings of the National Academy of Sciences of the United States of America.