The Cardiac-specific Nuclear δB Isoform of Ca2+/Calmodulin-dependent Protein Kinase II Induces Hypertrophy and Dilated Cardiomyopathy Associated with Increased Protein Phosphatase 2A Activity*
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Tong Zhang | John Ross | H. Schulman | J. Ross | Yusu Gu | M. Morissette | V. Sah | J. Brown | T. Rogers | Howard Schulman | Eric N Johnson | Yusu Gu | Joan Heller Brown | Darrell D Belke | Wolfgang H Dillmann | D. Belke | Marisa S Gigena | Terry B Rogers | W. Dillmann | Marisa S. Gigena | Michael R Morissette | Valerie P Sah | Tong Zhang | M. S. Gigena
[1] V. Sah,et al. Tyrosine kinase and c-Jun NH2-terminal kinase mediate hypertrophic responses to prostaglandin F2alpha in cultured neonatal rat ventricular myocytes. , 1998, Circulation research.
[2] T. McMahon,et al. Depolarization-induced neurite outgrowth in PC12 cells requires permissive, low level NGF receptor stimulation and activation of calcium/calmodulin-dependent protein kinase , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[3] N. Narayanan,et al. Phosphorylation and activation of the Ca(2+)-pumping ATPase of cardiac sarcoplasmic reticulum by Ca2+/calmodulin-dependent protein kinase. , 1993, The Journal of biological chemistry.
[4] H. Singer,et al. Identification of novel isoforms of the delta subunit of Ca2+/calmodulin-dependent protein kinase II. Differential expression in rat brain and aorta. , 1993, The Journal of biological chemistry.
[5] T Yamauchi,et al. Structural features of Ca2+/calmodulin-dependent protein kinase II revealed by electron microscopy , 1991, The Journal of cell biology.
[6] H. Schulman,et al. The multifunctional calcium/calmodulin-dependent protein kinase: from form to function. , 1995, Annual review of physiology.
[7] M. Böhm,et al. Molecular aspects of adrenergic signal transduction in cardiac failure , 1998, Journal of Molecular Medicine.
[8] R. Inoue,et al. Intracellular ATP slows time-dependent decline of muscarinic cation current in guinea pig ileal smooth muscle. , 2000, American journal of physiology. Cell physiology.
[9] I. Kameshita,et al. Purification and characterization of a brain-specific multifunctional calmodulin-dependent protein kinase from rat cerebellum. , 1992, The Journal of biological chemistry.
[10] M. Boutjdir,et al. Diminished basal phosphorylation level of phospholamban in the postinfarction remodeled rat ventricle: role of beta-adrenergic pathway, G(i) protein, phosphodiesterase, and phosphatases. , 1999, Circulation research.
[11] P. Cohen,et al. Identification of the major protein phosphatases in mammalian cardiac muscle which dephosphorylate phospholamban. , 1991, European journal of biochemistry.
[12] H. Schulman,et al. The Nuclear δB Isoform of Ca2+/Calmodulin-dependent Protein Kinase II Regulates Atrial Natriuretic Factor Gene Expression in Ventricular Myocytes* , 1997, The Journal of Biological Chemistry.
[13] C. Glembotski,et al. Induction of atrial natriuretic factor and myosin light chain-2 gene expression in cultured ventricular myocytes by electrical stimulation of contraction. , 1992, The Journal of biological chemistry.
[14] R. Passier,et al. CaM kinase signaling induces cardiac hypertrophy and activates the MEF2 transcription factor in vivo. , 2000, The Journal of clinical investigation.
[15] L. Jones,et al. Pharmacological characterization of protein phosphatase activities in preparations from failing human hearts. , 1999, The Journal of pharmacology and experimental therapeutics.
[16] K. Chien,et al. The MEKK-JNK Pathway Is Stimulated by α1-Adrenergic Receptor and Ras Activation and Is Associated with in Vitroand in Vivo Cardiac Hypertrophy* , 1997, The Journal of Biological Chemistry.
[17] H. Schulman,et al. Unique phosphorylation site on the cardiac ryanodine receptor regulates calcium channel activity. , 1991, The Journal of biological chemistry.
[18] M. Möhlig,et al. Novel and uncommon isoforms of the calcium sensing enzyme calcium/calmodulin dependent protein kinase II in heart tissue , 1995, Basic Research in Cardiology.
[19] K. Harjai. Potential New Cardiovascular Risk Factors: Left Ventricular Hypertrophy, Homocysteine, Lipoprotein(a), Triglycerides, Oxidative Stress, and Fibrinogen , 1999, Annals of Internal Medicine.
[20] A. Katz. Evolving concepts of heart failure: cooling furnace, malfunctioning pump, enlarging muscle. Part II: Hypertrophy and dilatation of the failing heart. , 1998, Journal of cardiac failure.
[21] Y. Zou,et al. Ca2+/Calmodulin-dependent Kinase II and Calcineurin Play Critical Roles in Endothelin-1-induced Cardiomyocyte Hypertrophy* , 2000, The Journal of Biological Chemistry.
[22] H. Schulman,et al. Identification and characterization of δB-CaM kinase and δC-CaM kinase from rat heart, two new multifunctional Ca2+/calmodulin-dependent protein kinase isoforms , 1994 .
[23] H. Schulman,et al. Alternative splicing introduces a nuclear localization signal that targets multifunctional CaM kinase to the nucleus , 1994, The Journal of cell biology.
[24] A. M. Watanabe,et al. Phosphorylation of phospholamban in intact myocardium. Role of Ca2+-calmodulin-dependent mechanisms. , 1985, The Journal of biological chemistry.
[25] J Ross,et al. Cardiac-specific overexpression of RhoA results in sinus and atrioventricular nodal dysfunction and contractile failure. , 1999, The Journal of clinical investigation.
[26] P. Ruth,et al. BK(Ca) channel activation by membrane-associated cGMP kinase may contribute to uterine quiescence in pregnancy. , 2000, American journal of physiology. Cell physiology.
[27] Michael G. Rosenfeld,et al. Expression of a multifunctional Ca2+/calmodulin-dependent protein kinase and mutational analysis of its autoregulation , 1989, Neuron.
[28] H. Schulman,et al. Multifunctional Ca2+/calmodulin-dependent Protein Kinase Mediates Ca(2+)-induced , 2022 .
[29] P. Karczewski,et al. The cardiac sarcoplasmic reticulum phospholamban kinase is a distinct δ‐CaM kinase isozyme , 1995 .
[30] Anirvan Ghosh,et al. Regulation of CBP-Mediated Transcription by Neuronal Calcium Signaling , 1999, Neuron.
[31] T. Hewett,et al. Ablation of the murine alpha myosin heavy chain gene leads to dosage effects and functional deficits in the heart. , 1996, The Journal of clinical investigation.
[32] M. Greenberg,et al. CREB: a Ca(2+)-regulated transcription factor phosphorylated by calmodulin-dependent kinases. , 1991, Science.
[33] Jeffrey Robbins,et al. A Calcineurin-Dependent Transcriptional Pathway for Cardiac Hypertrophy , 1998, Cell.
[34] E. Olson,et al. Prevention of cardiac hypertrophy by calcineurin inhibition: hope or hype? , 1999, Circulation research.
[35] M. Siddiqui,et al. Signal transduction and activator of transcription (STAT) protein-dependent activation of angiotensinogen promoter: a cellular signal for hypertrophy in cardiac muscle. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[36] R. Maurer,et al. Regulation of Activating Transcription Factor-1 and the cAMP Response Element-binding Protein by Ca/Calmodulin-dependent Protein Kinases Type I, II, and IV (*) , 1996, The Journal of Biological Chemistry.
[37] A. Means,et al. A signaling complex of Ca2+-calmodulin-dependent protein kinase IV and protein phosphatase 2A. , 1998, Science.