Ube2w and ataxin-3 coordinately regulate the ubiquitin ligase CHIP.
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J. Gestwicki | Junmin Peng | P. Xu | K. Scaglione | Sokol V. Todi | Srikanth Patury | Eszter Zavodszky | Svetlana Fischer | E. Rodríguez-Lebrón | A. Djarmati | John R. Konen | Henry L. Paulson
[1] A. Ciechanover,et al. Ubiquitination of E3 ligases: self-regulation of the ubiquitin system via proteolytic and non-proteolytic mechanisms , 2011, Cell Death and Differentiation.
[2] P. C. Janiesch,et al. The Machado-Joseph disease deubiquitylase ATX-3 couples longevity and proteostasis , 2011, Nature Cell Biology.
[3] Suneil K. Kalia,et al. Ubiquitinylation of α-Synuclein by Carboxyl Terminus Hsp70-Interacting Protein (CHIP) Is Regulated by Bcl-2-Associated Athanogene 5 (BAG5) , 2011, PloS one.
[4] H. Paulson,et al. Activity and Cellular Functions of the Deubiquitinating Enzyme and Polyglutamine Disease Protein Ataxin-3 Are Regulated by Ubiquitination at Lysine 117* , 2010, The Journal of Biological Chemistry.
[5] Rebecca B. Smith,et al. Native Functions of the Androgen Receptor Are Essential to Pathogenesis in a Drosophila Model of Spinobulbar Muscular Atrophy , 2010, Neuron.
[6] Huda Y. Zoghbi,et al. SCA1-like Disease in Mice Expressing Wild-Type Ataxin-1 with a Serine to Aspartic Acid Replacement at Residue 776 , 2010, Neuron.
[7] Giuseppe Nicastro,et al. Understanding the Role of the Josephin Domain in the PolyUb Binding and Cleavage Properties of Ataxin-3 , 2010, PloS one.
[8] M. Hoch,et al. Chaperone-Assisted Selective Autophagy Is Essential for Muscle Maintenance , 2010, Current Biology.
[9] S. Finkbeiner,et al. Serines 13 and 16 Are Critical Determinants of Full-Length Human Mutant Huntingtin Induced Disease Pathogenesis in HD Mice , 2009, Neuron.
[10] S. Gygi,et al. S5a promotes protein degradation by blocking synthesis of nondegradable forked ubiquitin chains , 2009, The EMBO journal.
[11] Harry T Orr,et al. Pathogenic Mechanisms of a Polyglutamine-mediated Neurodegenerative Disease, Spinocerebellar Ataxia Type 1* , 2009, Journal of Biological Chemistry.
[12] H. Paulson,et al. Ubiquitination directly enhances activity of the deubiquitinating enzyme ataxin‐3 , 2009, The EMBO journal.
[13] Zhigang Yu,et al. CHIP deletion reveals functional redundancy of E3 ligases in promoting degradation of both signaling proteins and expanded glutamine proteins. , 2008, Human molecular genetics.
[14] N. Eissa,et al. A Critical Role for CHIP in the Aggresome Pathway , 2008, Molecular and Cellular Biology.
[15] H. Paulson,et al. Polyglutamine neurodegeneration: protein misfolding revisited , 2008, Trends in Neurosciences.
[16] H. Paulson,et al. The Deubiquitinating Enzyme Ataxin-3, a Polyglutamine Disease Protein, Edits Lys63 Linkages in Mixed Linkage Ubiquitin Chains* , 2008, Journal of Biological Chemistry.
[17] K. Wilkinson,et al. Protein partners of deubiquitinating enzymes. , 2008, The Biochemical journal.
[18] S. Elledge,et al. A quantitative atlas of mitotic phosphorylation , 2008, Proceedings of the National Academy of Sciences.
[19] J. Nix,et al. Interactions between the quality control ubiquitin ligase CHIP and ubiquitin conjugating enzymes , 2008, BMC Structural Biology.
[20] Janghoo Lim,et al. Opposing effects of polyglutamine expansion on native protein complexes contribute to SCA1 , 2008, Nature.
[21] Qiuyan Wang,et al. Inhibition of p97-dependent Protein Degradation by Eeyarestatin I* , 2008, Journal of Biological Chemistry.
[22] T. Klockgether,et al. Inactivation of the mouse Atxn3 (ataxin-3) gene increases protein ubiquitination. , 2007, Biochemical and biophysical research communications.
[23] P. Brzovic,et al. E2–BRCA1 RING interactions dictate synthesis of mono- or specific polyubiquitin chain linkages , 2007, Nature Structural &Molecular Biology.
[24] F. Xia,et al. The ubiquitin-interacting motif containing protein RAP80 interacts with BRCA1 and functions in DNA damage repair response. , 2007, Cancer research.
[25] Sebastian A. Wagner,et al. E3-independent monoubiquitination of ubiquitin-binding proteins. , 2007, Molecular cell.
[26] Janghoo Lim,et al. ATAXIN-1 Interacts with the Repressor Capicua in Its Native Complex to Cause SCA1 Neuropathology , 2006, Cell.
[27] S. Gygi,et al. Deubiquitinating Enzyme Ubp6 Functions Noncatalytically to Delay Proteasomal Degradation , 2006, Cell.
[28] R. Pittman,et al. Ataxin-3 binds VCP/p97 and regulates retrotranslocation of ERAD substrates. , 2006, Human molecular genetics.
[29] A. Brice,et al. A regulated interaction with the UIM protein Eps15 implicates parkin in EGF receptor trafficking and PI(3)K–Akt signalling , 2006, Nature Cell Biology.
[30] Min Gao,et al. Activation of the E3 ubiquitin ligase Itch through a phosphorylation-induced conformational change. , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[31] C. Borchers,et al. Regulation of the Cytoplasmic Quality Control Protein Degradation Pathway by BAG2* , 2005, Journal of Biological Chemistry.
[32] L. Neckers,et al. Direct identification of ubiquitination sites on ubiquitin-conjugated CHIP using MALDI mass spectrometry. , 2005, Journal of proteome research.
[33] M. Cheetham,et al. HSJ1 Is a Neuronal Shuttling Factor for the Sorting of Chaperone Clients to the Proteasome , 2005, Current Biology.
[34] H. Paulson,et al. Ataxin-3 suppresses polyglutamine neurodegeneration in Drosophila by a ubiquitin-associated mechanism. , 2005, Molecular cell.
[35] Keiji Tanaka,et al. Co-chaperone CHIP Associates with Expanded Polyglutamine Protein and Promotes Their Degradation by Proteasomes* , 2005, Journal of Biological Chemistry.
[36] Barrington G. Burnett,et al. The polyglutamine neurodegenerative protein ataxin 3 regulates aggresome formation. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[37] S. Jentsch,et al. A Series of Ubiquitin Binding Factors Connects CDC48/p97 to Substrate Multiubiquitylation and Proteasomal Targeting , 2005, Cell.
[38] M. Karin,et al. Jun Turnover Is Controlled Through JNK-Dependent Phosphorylation of the E3 Ligase Itch , 2004, Science.
[39] H. Ishimoto,et al. Molecular clearance of ataxin‐3 is regulated by a mammalian E4 , 2004, The EMBO journal.
[40] Barrington G. Burnett,et al. The polyglutamine neurodegenerative protein ataxin-3 binds polyubiquitylated proteins and has ubiquitin protease activity. , 2003, Human molecular genetics.
[41] V. Godfrey,et al. CHIP activates HSF1 and confers protection against apoptosis and cellular stress , 2003, The EMBO journal.
[42] D. Peet,et al. Defining the Role for XAP2 in Stabilization of the Dioxin Receptor* , 2003, Journal of Biological Chemistry.
[43] T. Hashikawa,et al. CHIP is associated with Parkin, a gene responsible for familial Parkinson's disease, and enhances its ubiquitin ligase activity. , 2002, Molecular cell.
[44] D. Cyr,et al. CHIP Is a U-box-dependent E3 Ubiquitin Ligase , 2001, The Journal of Biological Chemistry.
[45] Y. Xiong,et al. The CUL1 C-Terminal Sequence and ROC1 Are Required for Efficient Nuclear Accumulation, NEDD8 Modification, and Ubiquitin Ligase Activity of CUL1 , 2000, Molecular and Cellular Biology.
[46] Angus Chen,et al. Conjugation of Nedd8 to CUL1 Enhances the Ability of the ROC1-CUL1 Complex to Promote Ubiquitin Polymerization* , 2000, The Journal of Biological Chemistry.
[47] T. Toda,et al. Covalent modifier NEDD8 is essential for SCF ubiquitin‐ligase in fission yeast , 2000, The EMBO journal.
[48] E. Lightcap,et al. A Nedd8 conjugation pathway is essential for proteolytic targeting of p27Kip1 by ubiquitination. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[49] R. Honda,et al. Modification of cullin-1 by ubiquitin-like protein Nedd8 enhances the activity of SCF(skp2) toward p27(kip1). , 2000, Biochemical and biophysical research communications.
[50] V. Chau,et al. Nedd8 Modification of Cul-1 Activates SCFβTrCP-Dependent Ubiquitination of IκBα , 2000, Molecular and Cellular Biology.
[51] J. Höhfeld,et al. The Ubiquitin-related BAG-1 Provides a Link between the Molecular Chaperones Hsc70/Hsp70 and the Proteasome* , 2000, The Journal of Biological Chemistry.
[52] Martin Rechsteiner,et al. Recognition of the polyubiquitin proteolytic signal , 2000, The EMBO journal.
[53] C. Patterson,et al. CHIP-dependent p53 regulation occurs specifically during cellular senescence. , 2011, Free radical biology & medicine.
[54] Thomas M. Durcan,et al. The Machado-Joseph disease-associated mutant form of ataxin-3 regulates parkin ubiquitination and stability. , 2011, Human molecular genetics.
[55] L. Petrucelli,et al. Brain CHIP: removing the culprits in neurodegenerative disease. , 2007, Trends in molecular medicine.
[56] M. Minami,et al. Purification and assay of the chaperone-dependent ubiquitin ligase of the carboxyl terminus of Hsc70-interacting protein. , 2005, Methods in enzymology.
[57] Raymond J. Deshaies,et al. Function and regulation of cullin–RING ubiquitin ligases , 2005, Nature Reviews Molecular Cell Biology.
[58] Holly McDonough,et al. CHIP: a link between the chaperone and proteasome systems , 2003, Cell stress & chaperones.
[59] P. Connell,et al. The co-chaperone CHIP regulates protein triage decisions mediated by heat-shock proteins , 2000, Nature Cell Biology.
[60] D. Cyr,et al. The Hsc70 co-chaperone CHIP targets immature CFTR for proteasomal degradation , 2000, Nature Cell Biology.