Impact of myocardial perfusion abnormality on prognosis in patients with non-ischemic dilated cardiomyopathy.

[1]  C. Çevik,et al.  Diffuse late gadolinium enhancement by cardiovascular magnetic resonance predicts significant intraventricular systolic dyssynchrony in patients with non-ischemic dilated cardiomyopathy. , 2010, Journal of the American Society of Echocardiography : official publication of the American Society of Echocardiography.

[2]  S. Abbara Late Gadolinium Enhancement by Cardiovascular Magnetic Resonance Heralds an Adverse Prognosis in Nonischemic Cardiomyopathy , 2009 .

[3]  T. Kubo,et al.  Improvement in prognosis of dilated cardiomyopathy in the elderly over the past 20 years. , 2008, Journal of cardiology.

[4]  Katherine C. Wu,et al.  Late gadolinium enhancement by cardiovascular magnetic resonance heralds an adverse prognosis in nonischemic cardiomyopathy. , 2008, Journal of the American College of Cardiology.

[5]  Dudley J Pennell,et al.  Cardiovascular magnetic resonance, fibrosis, and prognosis in dilated cardiomyopathy. , 2006, Journal of the American College of Cardiology.

[6]  J. Higaki,et al.  Effect of Beta-Blocker Therapy on Myocardial Perfusion Defects in Thallium-201 Scintigraphy in Patients with Dilated Cardiomyopathy , 2005, Cardiology.

[7]  P. Vardas,et al.  Regional coronary flow and contractile reserve in patients with idiopathic dilated cardiomyopathy. , 2004, Journal of the American College of Cardiology.

[8]  V. Eder,et al.  Segmental wall motion abnormalities in idiopathic dilated cardiomyopathy and their effect on prognosis. , 2004, The American journal of cardiology.

[9]  P. McCarthy,et al.  PET abnormalities in patients with nonischemic cardiomyopathy. , 2004, Journal of cardiac failure.

[10]  W. Stevenson,et al.  Endocardial and epicardial radiofrequency ablation of ventricular tachycardia associated with dilated cardiomyopathy: the importance of low-voltage scars. , 2004, Journal of the American College of Cardiology.

[11]  Jeroen J. Bax,et al.  The effects of cardiac resynchronization therapy on left ventricular function, myocardial energetics, and metabolic reserve in patients with dilated cardiomyopathy and heart failure. , 2004, Journal of the American College of Cardiology.

[12]  B. Nowak,et al.  Comparison of regional myocardial blood flow and perfusion in dilated cardiomyopathy and left bundle branch block: role of wall thickening. , 2004, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[13]  C H Lorenz,et al.  Differentiation of Heart Failure Related to Dilated Cardiomyopathy and Coronary Artery Disease Using Gadolinium‐Enhanced Cardiovascular Magnetic Resonance , 2003, Circulation.

[14]  B. Nowak,et al.  Cardiac resynchronization therapyhomogenizes myocardial glucosemetabolism and perfusion in dilatedcardiomyopathy and left bundle branch block , 2003 .

[15]  B. Nowak,et al.  Cardiac resynchronization therapy homogenizes myocardial glucose metabolism and perfusion in dilated cardiomyopathy and left bundle branch block. , 2003, Journal of the American College of Cardiology.

[16]  P. Olivier,et al.  201Tl SPECT abnormalities, documented at rest in dilated cardiomyopathy, are related to a lower than normal myocardial thickness but not to an excess in myocardial wall stress. , 2002, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[17]  R. Parker,et al.  Validation of a model of left ventricular segmentation for interpretation of SPET myocardial perfusion images , 2001, European Journal of Nuclear Medicine.

[18]  A. McCulloch,et al.  Regional heterogeneity of function in nonischemic dilated cardiomyopathy. , 2001, Cardiovascular research.

[19]  渡邉 元博 Relationship between thallium-201 myocardial SPECT and findings of endomyocardial biopsy specimens in dilated cardiomyopathy , 2000 .

[20]  H. Crijns,et al.  Regional myocardial blood flow reserve impairment and metabolic changes suggesting myocardial ischemia in patients with idiopathic dilated cardiomyopathy. , 2000, Journal of the American College of Cardiology.

[21]  G. Newton,et al.  Electrical and hemodynamic correlates of the maximal rate of pressure increase in the human left ventricle. , 1999, Journal of cardiac failure.

[22]  N. Tamaki,et al.  Comparative study of201Tl-scintigraphic image and myocardial pathologic findings in patients with dilated cardiomyopathy , 1996 .

[23]  D. Gibson,et al.  Natural history of abnormal conduction and its relation to prognosis in patients with dilated cardiomyopathy. , 1996, International journal of cardiology.

[24]  M. Schwaiger,et al.  Heterogeneity of ventricular function and myocardial oxidative metabolism in nonischemic dilated cardiomyopathy. , 1995, Journal of the American College of Cardiology.

[25]  N. Danchin,et al.  Radionuclide assessment of regional differences in left ventricular wall motion and myocardial perfusion in idiopathic dilated cardiomyopathy. , 1993, European heart journal.

[26]  C. Higgins,et al.  Variation in left ventricular regional wall stress with cine magnetic resonance imaging: normal subjects versus dilated cardiomyopathy. , 1993, American heart journal.

[27]  D G Gibson,et al.  Effects of abnormal activation on the time course of the left ventricular pressure pulse in dilated cardiomyopathy , 1992, British heart journal.

[28]  J. Poloniecki,et al.  Prognostic value of thallium-201 perfusion defects in idiopathic dilated cardiomyopathy. , 1991, The American journal of cardiology.

[29]  C Kawai,et al.  Left ventricular regional wall stress in dilated cardiomyopathy. , 1990, Circulation.

[30]  S. Larson,et al.  Myocardial perfusion abnormalities in patients with hypertrophic cardiomyopathy: assessment with thallium-201 emission computed tomography. , 1987, Circulation.

[31]  K. Tsuiki,et al.  Segmental wall motion abnormalities in dilated cardiomyopathy: hemodynamic characteristics and comparison with thallium-201 myocardial scintigraphy. , 1987, American heart journal.

[32]  A. Hakki,et al.  Resting thallium-201 myocardial perfusion patterns in patients with severe left ventricular dysfunction: differences between patients with primary cardiomyopathy, chronic coronary artery disease, or acute myocardial infarction. , 1986, American heart journal.

[33]  R. Bonow,et al.  Myocardial ischemia in patients with hypertrophic cardiomyopathy: contribution of inadequate vasodilator reserve and elevated left ventricular filling pressures. , 1985, Circulation.

[34]  R. Henkin,et al.  Segmental wall motion abnormalities in dilated cardiomyopathy: a common finding and good prognostic sign. , 1984, Journal of the American College of Cardiology.

[35]  M. Moeschberger,et al.  Factors influencing the one-year mortality of dilated cardiomyopathy. , 1984, The American journal of cardiology.

[36]  M. Hiroe,et al.  Comparison of Thallium‐201 Scanning in Idiopathic Dilated Cardiomyopathy and Severe Coronary Artery Disease , 1982, Circulation.