Abnormal changes in voltage-gated sodium channels subtypes NaV1.1, NaV1.2, NaV1.3, NaV1.6 and CaM/CaMKII pathway in low-grade astrocytoma

[1]  A. Leffler,et al.  Investigation into the role of an extracellular loop in mediating proton-evoked inhibition of voltage-gated sodium channels , 2017, Neuroscience Letters.

[2]  S. Zuberi,et al.  Dravet syndrome and its mimics: Beyond SCN1A , 2017, Epilepsia.

[3]  A. L. Goldin,et al.  Early-life febrile seizures worsen adult phenotypes in Scn1a mutants , 2017, Experimental Neurology.

[4]  M. A. Shea,et al.  Calcium triggers reversal of calmodulin on nested anti-parallel sites in the IQ motif of the neuronal voltage-dependent sodium channel NaV1.2. , 2017, Biophysical chemistry.

[5]  M. Zeng,et al.  Ion channels or aquaporins as novel molecular targets in gastric cancer , 2017, Molecular Cancer.

[6]  Christopher H. Thompson,et al.  CaMKII modulates sodium current in neurons from epileptic Scn2a mutant mice , 2017, Proceedings of the National Academy of Sciences.

[7]  D. O'Dowd,et al.  Model systems for studying cellular mechanisms of SCN1A-related epilepsy. , 2016, Journal of neurophysiology.

[8]  S. Waxman,et al.  Voltage‐gated sodium channel Nav1.5 contributes to astrogliosis in an in vitro model of glial injury via reverse Na+/Ca2+ exchange , 2014, Glia.

[9]  Shao-Wu Ou,et al.  Expression of neonatal Nav1.5 in human brain astrocytoma and its effect on proliferation, invasion and apoptosis of astrocytoma cells. , 2014, Oncology reports.

[10]  C. Vecht,et al.  Seizure characteristics and prognostic factors of gliomas , 2013, Epilepsia.

[11]  Ying Sun,et al.  Regulation of ASIC1 by Ca2+/calmodulin-dependent protein kinase II in human glioblastoma multiforme. , 2013, Oncology reports.

[12]  J. Gorter,et al.  Expression of sodium channel α subunits 1.1, 1.2 and 1.6 in rat hippocampus after kainic acid-induced epilepsy , 2013, Epilepsy Research.

[13]  W. Catterall,et al.  Correlations in timing of sodium channel expression, epilepsy, and sudden death in Dravet syndrome , 2013, Channels.

[14]  Feng Guo,et al.  Abnormal changes in voltage-gated sodium channels NaV1.1, NaV1.2, NaV1.3, NaV1.6 and in calmodulin/calmodulin-dependent protein kinase II, within the brains of spontaneously epileptic rats and tremor rats , 2013, Brain Research Bulletin.

[15]  Feng Guo,et al.  The up-regulation of voltage-gated sodium channels subtypes coincides with an increased sodium current in hippocampal neuronal culture model , 2013, Neurochemistry International.

[16]  K. Murray,et al.  Neuronal excitability and calcium/calmodulin‐dependent protein kinase type II: Location, location, location , 2012, Epilepsia.

[17]  F. van Petegem,et al.  Crystallographic basis for calcium regulation of sodium channels , 2012, Proceedings of the National Academy of Sciences.

[18]  E. Engleman,et al.  Calcium/Calmodulin-dependent Protein Kinase II (CaMKII) Inhibition Induces Neurotoxicity via Dysregulation of Glutamate/Calcium Signaling and Hyperexcitability* , 2012, The Journal of Biological Chemistry.

[19]  Xue-hong Cao,et al.  The modulation of the excitability of primary sensory neurons by Ca2+–CaM–CaMKII pathway , 2012, Neurological Sciences.

[20]  V. Velculescu,et al.  Sodium ion channel mutations in glioblastoma patients correlate with shorter survival , 2011, Molecular Cancer.

[21]  L. Isom,et al.  Electrophysiology and beyond: Multiple roles of Na+ channel β subunits in development and disease , 2010, Neuroscience Letters.

[22]  S. Waxman,et al.  Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5. , 2010, Brain : a journal of neurology.

[23]  Vadim Zinchuk,et al.  Recent advances in quantitative colocalization analysis: focus on neuroscience. , 2009, Progress in histochemistry and cytochemistry.

[24]  Z. Zong,et al.  Voltage-gated sodium channel Nav1.1, Nav1.3 and β1 subunit were up-regulated in the hippocampus of spontaneously epileptic rat , 2008, Brain Research Bulletin.

[25]  W. Catterall,et al.  International Union of Pharmacology. XLVIII. Nomenclature and Structure-Function Relationships of Voltage-Gated Calcium Channels , 2005, Pharmacological Reviews.

[26]  J. Kurz,et al.  Age dependence of pilocarpine-induced status epilepticus and inhibition of CaM kinase II activity in the rat. , 2005, Brain research. Developmental brain research.

[27]  H. Rosenberg,et al.  Prolonged changes in Ca2+/calmodulin-dependent protein kinase II after a brief pentylenetetrazol seizure; potential role in kindling , 2004, Epilepsy Research.

[28]  A. Twijnstra,et al.  For Personal Use. Only Reproduce with Permission from the Lancet Publishing Group. Presenting Symptoms and Imaging of Grade Ii Astrocytoma Supratentorial Grade Ii Astrocytoma: Biological Features and Clinical Course , 2022 .

[29]  K. Skullerud,et al.  Prevalence and prognostic significance of epilepsy in patients with gliomas. , 1998, European journal of cancer.

[30]  S. Waxman,et al.  Astrocyte Na+ channels are required for maintenance of Na+/K(+)-ATPase activity , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[31]  S. Waxman,et al.  Expression of voltage-activated ion channels by astrocytes and oligodendrocytes in the hippocampal slice. , 1993, Journal of neurophysiology.

[32]  S. Waxman,et al.  Ion channels in spinal cord astrocytes in vitro. I. Transient expression of high levels of Na+ and K+ channels. , 1992, Journal of neurophysiology.

[33]  S. Waxman,et al.  The perinodal astrocyte , 1988, Glia.