Peripheral corticotropin releasing hormone mediates post-inflammatory visceral hypersensitivity in rats.
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J. La | T. Kang | T. Sung | I. Yang | Hyun-Ju Kim | Taewan Kim
[1] B. Greenwood-Van Meerveld,et al. Corticotropin‐releasing factor receptor 1‐deficient mice show decreased anxiety and colonic sensitivity , 2007, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.
[2] M. Hongo,et al. Effect of alpha‐helical CRH on quantitative electroencephalogram in patients with irritable bowel syndrome , 2007, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.
[3] G. Holtmann,et al. Severity of mucosal inflammation as a predictor for alterations of visceral sensory function in a rat model , 2006, Pain.
[4] J. Schulkin,et al. Long-term expression of corticotropin-releasing factor (CRF) in the paraventricular nucleus of the hypothalamus in response to an acute colonic inflammation , 2006, Brain Research.
[5] Greg D. Gale,et al. Corticotropin-releasing factor receptor 1 mediates acute and delayed stress-induced visceral hyperalgesia in maternally separated Long-Evans rats. , 2005, American journal of physiology. Gastrointestinal and liver physiology.
[6] Y. Taché,et al. Role of corticotropin-releasing factor pathways in stress-related alterations of colonic motor function and viscerosensibility in female rodents. , 2005, Gender medicine.
[7] S. L. la Fleur,et al. Role of peripheral corticotropin-releasing factor and urocortin II in intestinal inflammation and motility in terminal ileum. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[8] A. Weston,et al. Terminal ileal mucosal mast cells in irritable bowel syndrome , 1993, Digestive Diseases and Sciences.
[9] M. Hongo,et al. Effect of a corticotropin releasing hormone receptor antagonist on colonic sensory and motor function in patients with irritable bowel syndrome , 2004, Gut.
[10] W. Khan,et al. Visceral hyperalgesia and intestinal dysmotility in a mouse model of postinfective gut dysfunction. , 2004, Gastroenterology.
[11] C. Pothoulakis,et al. Corticotropin-releasing hormone (CRH) requirement in Clostridium difficile toxin A-mediated intestinal inflammation. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[12] Jeom-Yong Kim,et al. Role of mucosal mast cells in visceral hypersensitivity in a rat model of irritable bowel syndrome. , 2004, Journal of veterinary science.
[13] G. Gerken,et al. Long-Term Effects of Transient Chemically Induced Colitis on the Visceromotor Response to Mechanical Colorectal Distension , 2002, Digestive Diseases and Sciences.
[14] J. La,et al. Visceral hypersensitivity and altered colonic motility after subsidence of inflammation in a rat model of colitis. , 2003, World Journal of Gastroenterology.
[15] R. Spiller,et al. Distinctive clinical, psychological, and histological features of postinfective irritable bowel syndrome , 2003, American Journal of Gastroenterology.
[16] H. Mertz. Visceral hypersensitivity , 2003, Alimentary pharmacology & therapeutics.
[17] C. Pothoulakis,et al. Corticotropin-releasing hormone antagonists possess anti-inflammatory effects in the mouse ileum. , 2002, Gastroenterology.
[18] G. Barbara,et al. A role for inflammation in irritable bowel syndrome? , 2002, Gut.
[19] M. Delvaux. Role of visceral sensitivity in the pathophysiology of irritable bowel syndrome , 2002, Gut.
[20] Y. Taché,et al. Peripheral CRF activates myenteric neurons in the proximal colon through CRF(1) receptor in conscious rats. , 2002, American journal of physiology. Gastrointestinal and liver physiology.
[21] S. Lightman,et al. CRH in chronic inflammatory stress , 2001, Peptides.
[22] Y. Taché,et al. Peripheral corticotropin-releasing factor and stress-stimulated colonic motor activity involve type 1 receptor in rats. , 2000, Gastroenterology.
[23] G. Gebhart. Visceral pain—peripheral sensitisation , 2000, Gut.
[24] P. Pasricha,et al. A new model of chronic visceral hypersensitivity in adult rats induced by colon irritation during postnatal development. , 2000, Gastroenterology.
[25] C. O'Morain,et al. Increased mast cells in the irritable bowel syndrome , 2000, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.
[26] E. Razin,et al. Mast Cell-Nerve Interactions. , 2000, News in physiological sciences : an international journal of physiology produced jointly by the International Union of Physiological Sciences and the American Physiological Society.
[27] L. Buéno,et al. Pathobiology of visceral pain: molecular mechanisms and therapeutic implications. III. Visceral afferent pathways: a source of new therapeutic targets for abdominal pain. , 2000, American journal of physiology. Gastrointestinal and liver physiology.
[28] Picard,et al. Role of kinin B1 and B2 receptors and mast cells in post intestinal infection‐induced hypersensitivity to distension , 1998, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.
[29] V. Théodorou,et al. Stress‐induced visceral hypersensitivity to rectal distension in rats: role of CRF and mast cells , 1997, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.
[30] M. Yamauchi,et al. Local production of corticotropin releasing hormone is increased in experimental intestinal inflammation in rats. , 1996, Gut.
[31] G. Chrousos,et al. Corticotropin releasing hormone in colonic mucosa in patients with ulcerative colitis. , 1995, Gut.
[32] C. Wolfe,et al. Corticotrophin-releasing hormone (CRH)-binding protein interference with CRH antibody binding: implications for direct CRH immunoassay. , 1995, The Journal of endocrinology.
[33] T. Takao,et al. Plasma CRH response to water immersion-restraint stress in rats bearing a hypothalamic knife cut. , 1994, Endocrine journal.
[34] P. Whorwell,et al. Symptoms of irritable bowel syndrome in ulcerative colitis in remission , 1983, Gut.
[35] L. Katz,et al. Mast cells in inflammatory conditions of the gastrointestinal tract. , 1962, The American journal of gastroenterology.