Consequences of Nigrostriatal Denervation on the Functioning of the Basal Ganglia in Human and Nonhuman Primates: An In Situ Hybridization Study of Cytochrome Oxidase Subunit I mRNA
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J A Obeso | Y Agid | E. Hirsch | Y. Agid | J. Obeso | M. Herrero | M. Vila | M. Ruberg | Richard Lévy | B. Faucheux | M Ruberg | B Faucheux | E C Hirsch | M Vila | R Levy | M T Herrero | R. Lévy
[1] O. Hassani,et al. Re-evaluation of the functional anatomy of the basal ganglia in normal and Parkinsonian states , 1997, Neuroscience.
[2] M. Chesselet,et al. Basal ganglia and movement disorders: an update , 1996, Trends in Neurosciences.
[3] Y. Agid,et al. Consequence of nigrostriatal denervation and L-dopa therapy on the expression of glutamic acid decarboxylase messenger RNA in the pallidum , 1996, Neurology.
[4] J. Walters,et al. Apomorphine and dopamine D1 receptor agonists increase the firing rates of subthalamic nucleus neurons , 1996, Neuroscience.
[5] O. Hassani,et al. Increased subthalamic neuronal activity after nigral dopaminergic lesion independent of disinhibition via the globus pallidus , 1996, Neuroscience.
[6] J. A. Obeso,et al. Metabolic activity of the basal ganglia in parkinsonian syndromes in human and non-human primates: A cytochrome oxidase histochemistry study , 1996, Neuroscience.
[7] Y. Agid,et al. Consequences of nigrostriatal denervation on the gamma-aminobutyric acidic neurons of substantia nigra pars reticulata and superior colliculus in parkinsonian syndromes , 1996, Neurology.
[8] J. Marshall,et al. D1 dopamine receptors influence Fos immunoreactivity in the globus pallidus and subthalamic nucleus of intact and nigrostriatal-lesioned rats , 1995, Brain Research.
[9] M. Chesselet,et al. Subthalamic nucleus lesions: widespread effects on changes in gene expression induced by nigrostriatal dopamine depletion in rats , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[10] O. Hassani,et al. Electrophysiological study of the excitatory parafascicular projection to the subthalamic nucleus and evidence for ipsi- and contralateral controls , 1995, Neuroscience.
[11] Y. Agid,et al. Effects of Nigrostriatal Denervation and L‐Dopa Therapy on the GABAergic Neurons of the Striatum in MPTP‐treated Monkeys and Parkinson's Disease: An In Situ Hybridization Study of GAD67 mRNA , 1995, The European journal of neuroscience.
[12] R. Hevner,et al. A metabolic map of cytochrome oxidase in the rat brain: Histochemical, densitometric and biochemical studies , 1995, Neuroscience.
[13] J. Greenamyre,et al. Polysynaptic regulation of glutamate receptors and mitochondrial enzyme activities in the basal ganglia of rats with unilateral dopamine depletion , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[14] A. Parent,et al. Increased glutamate decarboxylase mRNA levels in the striatum and pallidum of MPTP-treated primates , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[15] F. Wiesel,et al. Characterization of dopamine receptor binding sites in the subthalamic nucleus. , 1994, Neuroreport.
[16] H. Bergman,et al. The primate subthalamic nucleus. I. Functional properties in intact animals. , 1994, Journal of neurophysiology.
[17] H. Bergman,et al. The primate subthalamic nucleus. II. Neuronal activity in the MPTP model of parkinsonism. , 1994, Journal of neurophysiology.
[18] M. Mouroux,et al. Evidence that the parafascicular projection to the subthalamic nucleus is glutamatergic. , 1993, Neuroreport.
[19] M. Wong-Riley,et al. Mitochondrial and nuclear gene expression for cytochrome oxidase subunits are disproportionately regulated by functional activity in neurons , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[20] B Bioulac,et al. Reversal of Rigidity and Improvement in Motor Performance by Subthalamic High‐frequency Stimulation in MPTP‐treated Monkeys , 1993, The European journal of neuroscience.
[21] M. Delong,et al. Pathophysiology of parkinsonian motor abnormalities. , 1993, Advances in neurology.
[22] P. Salin,et al. Regulation of glutamic acid decarboxylase gene expression in efferent neurons of the basal ganglia. , 1993, Progress in brain research.
[23] J. Penney,et al. 6-Hydroxydopamine lesions of the nigrostriatal pathway alter the expression of glutamate decarboxylase messenger RNA in rat globus pallidus projection neurons , 1992, Neuroscience.
[24] I. Mitchell,et al. A 2-deoxyglucose study of the effects of dopamine agonists on the parkinsonian primate brain. Implications for the neural mechanisms that mediate dopamine agonist-induced dyskinesia. , 1992, Brain : a journal of neurology.
[25] M. Chesselet,et al. Effects of nigrostriatal lesions on the levels of messenger RNAs encoding two isoforms of glutamate decarboxylase in the globus pallidus and entopeduncular nucleus of the rat , 1992, Synapse.
[26] R. Hevner,et al. Coordination of ATP production and consumption in brain: Parallel regulation of cytochrome oxidase and Na+, K+-ATPase , 1992, Neuroscience Letters.
[27] C. Marescaux,et al. Contralateral disappearance of parkinsonian signs after subthalamic hematoma , 1992, Neurology.
[28] M. Wong-Riley,et al. Neuronal expression of nuclear and mitochondrial genes for cytochrome oxidase (CO) subunits analyzed by in situ hybridization: comparison with CO activity and protein , 1991, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[29] L. Tremblay,et al. Abnormal spontaneous activity of globus pallidus neurons in monkeys with MPTP-induced parkinsonism , 1991, Brain Research.
[30] T. Aziz,et al. Lesion of the subthalamic nucleus for the alleviation of 1‐methyl‐4‐phenyl‐1,2,3,6‐tetrahydropyridine (MPTP)‐induced parkinsonism in the primate , 1991, Movement disorders : official journal of the Movement Disorder Society.
[31] E. Hirsch,et al. Decreased tyrosine hydroxylase messenger RNA in the surviving dopamine neurons of the substantia nigra in parkinson's disease: An in situ hybridization study , 1990, Neuroscience.
[32] H. Bergman,et al. Reversal of experimental parkinsonism by lesions of the subthalamic nucleus. , 1990, Science.
[33] M. Delong,et al. Primate models of movement disorders of basal ganglia origin , 1990, Trends in Neurosciences.
[34] G. E. Alexander,et al. Functional architecture of basal ganglia circuits: neural substrates of parallel processing , 1990, Trends in Neurosciences.
[35] M. Wong-Riley,et al. Regulation of cytochrome oxidase protein levels by functional activity in the macaque monkey visual system , 1990, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[36] R. G. Robertson,et al. Neural mechanisms underlying parkinsonian symptoms based upon regional uptake of 2-deoxyglucose in monkeys exposed to 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine , 1989, Neuroscience.
[37] J. Penney,et al. The functional anatomy of basal ganglia disorders , 1989, Trends in Neurosciences.
[38] T. Klockgether,et al. Excitatory amino acids and the basal ganglia: implications for the therapy of Parkinson's disease , 1989, Trends in Neurosciences.
[39] M. Wong-Riley. Cytochrome oxidase: an endogenous metabolic marker for neuronal activity , 1989, Trends in Neurosciences.
[40] A. Crossman. Neural mechanisms in disorders of movement. , 1989, Comparative biochemistry and physiology. A, Comparative physiology.
[41] C. Feuerstein,et al. Similar Time Course Changes in Striatal Levels of Glutamic Acid Decarboxylase and Proenkephalin mRNA Following Dopaminergic Deafferentation in the Rat , 1988, Journal of neurochemistry.
[42] J P Changeux,et al. Detection of the nicotinic acetylcholine receptor alpha‐subunit mRNA by in situ hybridization at neuromuscular junctions of 15‐day‐old chick striated muscles. , 1988, The EMBO journal.
[43] J. Walters,et al. Unilateral lesion of the nigrostriatal pathway decreases the firing rate and alters the firing pattern of globus pallidus neurons in the rat , 1988, Synapse.
[44] M. Chesselet,et al. Comparative distribution of mRNAs for glutamic acid decatrboxylase, tyrosine hydroxylase, and tachykinins in the basal ganglia: An in situ hybridization study in the rodent brain , 1987, The Journal of comparative neurology.
[45] A. Crane,et al. Local cerebral metabolic effects of L-dopa therapy in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced parkinsonism in monkeys. , 1987, Proceedings of the National Academy of Sciences of the United States of America.
[46] M. Delong,et al. Altered Tonic Activity of Neurons in the Globus Pallidus and Subthalamic Nucleus in the Primate MPTP Model of Parkinsonism , 1987 .
[47] B. Kadenbach,et al. Evolution of a regulatory enzyme: cytochrome-c oxidase (complex IV) , 1987 .
[48] C. Hammond,et al. Excitatory effect of iontophoretically applied dopamine on identified neurons of the rat subthalamic nucleus , 1986, Brain Research.
[49] F. F. Weight,et al. Dopaminergic mechanisms in subthalamic nucleus of rat: analysis using horseradish peroxidase and microiontophoresis , 1985, Brain Research.
[50] M. Wong-Riley,et al. Effect of impulse blockage on cytochrome oxidase activity in monkey visual system , 1984, Nature.
[51] B. Kadenbach,et al. Separation of mammalian cytochrome c oxidase into 13 polypeptides by a sodium dodecyl sulfate-gel electrophoretic procedure. , 1983, Analytical biochemistry.
[52] M. Wong-Riley,et al. Histochemical changes in cytochrome oxidase of cortical barrels after vibrissal removal in neonatal and adult mice. , 1980, Proceedings of the National Academy of Sciences of the United States of America.
[53] M. Filion. Effects of interruption of the nigrostriatal pathway and of dopaminergic agents on the spontaneous activity of globus pallidus neurons in the awake monkey , 1979, Brain Research.
[54] M. Wong-Riley. Changes in the visual system of monocularly sutured or enucleated cats demonstrable with cytochrome oxidase histochemistry , 1979, Brain Research.
[55] C. W. Ragsdale,et al. Histochemically distinct compartments in the striatum of human, monkeys, and cat demonstrated by acetylthiocholinesterase staining. , 1978, Proceedings of the National Academy of Sciences of the United States of America.
[56] Michael M. Merzenich,et al. Changes in endogenous enzymatic reactivity to DAB induced by neuronal inactivity , 1978, Brain Research.