D-Allulose cooperates with glucagon-like peptide-1 and activates proopiomelanocortin neurons in the arcuate nucleus and central injection inhibits feeding in mice.

[1]  S. M. Fortin,et al.  The role of glia in the physiology and pharmacology of glucagon‐like peptide‐1: implications for obesity, diabetes, neurodegeneration and glaucoma , 2021, British journal of pharmacology.

[2]  Y. Seino,et al.  Onion component, isoalliin, stimulates feeding and activates the arcuate nucleus neuropeptide Y, ghrelin- and Ninjin'yoeito-responsive neurons , 2021, Neuropeptides.

[3]  Yoichi Chiba,et al.  Glucose, Fructose, and Urate Transporters in the Choroid Plexus Epithelium , 2020, International journal of molecular sciences.

[4]  Myung‐Sook Choi,et al.  Alteration of Microbiome Profile by D-Allulose in Amelioration of High-Fat-Diet-Induced Obesity in Mice , 2020, Nutrients.

[5]  B. Puri Calcium Signaling and Gene Expression. , 2020, Advances in experimental medicine and biology.

[6]  A. Inui,et al.  Ninjin-yoeito activates ghrelin-responsive and unresponsive NPY neurons in the arcuate nucleus and counteracts cisplatin-induced anorexia , 2019, Neuropeptides.

[7]  Myung‐Sook Choi,et al.  A Preliminary Study for Evaluating the Dose-Dependent Effect of d-Allulose for Fat Mass Reduction in Adult Humans: A Randomized, Double-Blind, Placebo-Controlled Trial , 2018, Nutrients.

[8]  D. Drucker,et al.  GLP-1 release and vagal afferent activation mediate the beneficial metabolic and chronotherapeutic effects of D-allulose , 2018, Nature Communications.

[9]  Takako Yamada,et al.  Rare Sugar Syrup Containing d-Allulose but Not High-Fructose Corn Syrup Maintains Glucose Tolerance and Insulin Sensitivity Partly via Hepatic Glucokinase Translocation in Wistar Rats. , 2017, Journal of agricultural and food chemistry.

[10]  Myung‐Sook Choi,et al.  d-Allulose supplementation normalized the body weight and fat-pad mass in diet-induced obese mice via the regulation of lipid metabolism under isocaloric fed condition. , 2016, Molecular nutrition & food research.

[11]  M. Tokuda,et al.  Rare sugar D-allulose: Potential role and therapeutic monitoring in maintaining obesity and type 2 diabetes mellitus. , 2015, Pharmacology & therapeutics.

[12]  Akane Kanasaki,et al.  D-psicose, an epimer of D-fructose, favorably alters lipid metabolism in Sprague-Dawley rats. , 2015, Journal of agricultural and food chemistry.

[13]  L. Sui,et al.  Intestinal absorption, organ distribution, and urinary excretion of the rare sugar D-psicose , 2014, Drug design, development and therapy.

[14]  J. Hecksher-Sørensen,et al.  The arcuate nucleus mediates GLP-1 receptor agonist liraglutide-dependent weight loss. , 2014, The Journal of clinical investigation.

[15]  Takako Yamada,et al.  d-Psicose increases energy expenditure and decreases body fat accumulation in rats fed a high-sucrose diet , 2014, International journal of food sciences and nutrition.

[16]  Ernesto Carafoli,et al.  Neuronal calcium signaling: function and dysfunction , 2014, Cellular and Molecular Life Sciences.

[17]  Takako Yamada,et al.  Inhibition by Dietary D-Psicose of Body Fat Accumulation in Adult Rats Fed a High-Sucrose Diet , 2013, Bioscience, biotechnology, and biochemistry.

[18]  P. Carmeliet,et al.  Tanycytic VEGF-A boosts blood-hypothalamus barrier plasticity and access of metabolic signals to the arcuate nucleus in response to fasting. , 2013, Cell metabolism.

[19]  David P. Olson,et al.  Central nervous system control of metabolism , 2012, Nature.

[20]  A. Nishiyama,et al.  Rare sugar D-psicose improves insulin sensitivity and glucose tolerance in type 2 diabetes Otsuka Long-Evans Tokushima Fatty (OLETF) rats. , 2011, Biochemical and biophysical research communications.

[21]  B. Lowell,et al.  Direct insulin and leptin action on pro-opiomelanocortin neurons is required for normal glucose homeostasis and fertility. , 2010, Cell metabolism.

[22]  Takako Yamada,et al.  Study on the Postprandial Blood Glucose Suppression Effect of D-Psicose in Borderline Diabetes and the Safety of Long-Term Ingestion by Normal Human Subjects , 2010, Bioscience, biotechnology, and biochemistry.

[23]  W. Mu,et al.  Characterization of d-tagatose-3-epimerase from Rhodobacter sphaeroides that converts d-fructose into d-psicose , 2009, Biotechnology Letters.

[24]  S. Lumyong,et al.  Efficient conversion of allitol to D-psicose by Bacillus pallidus Y25. , 2007, Journal of bioscience and bioengineering.

[25]  Robert A. McGovern,et al.  Divergence of Melanocortin Pathways in the Control of Food Intake and Energy Expenditure , 2005, Cell.

[26]  T. Yada,et al.  Ghrelin directly interacts with neuropeptide-Y-containing neurons in the rat arcuate nucleus: Ca2+ signaling via protein kinase A and N-type channel-dependent mechanisms and cross-talk with leptin and orexin. , 2003, Diabetes.

[27]  T. Matsuo,et al.  Dietary D-psicose, a C-3 epimer of D-fructose, suppresses the activity of hepatic lipogenic enzymes in rats. , 2001, Asia Pacific journal of clinical nutrition.

[28]  M. Nakata,et al.  Pituitary adenylate cyclase activating polypeptide is an extraordinarily potent intra-pancreatic regulator of insulin secretion from islet beta-cells. , 1994, The Journal of biological chemistry.