Interferon lambda alleles predict innate antiviral immune responses and hepatitis C virus permissiveness.

[1]  Charles M. Rice,et al.  Corrigendum: A diverse range of gene products are effectors of the type I interferon antiviral response , 2015, Nature.

[2]  Raymond T Chung,et al.  Kinetic differences in the induction of interferon stimulated genes by interferon‐α and interleukin 28B are altered by infection with hepatitis C virus , 2014, Hepatology.

[3]  T. Liang,et al.  Association of IL28B genotype with fibrosis progression and clinical outcomes in patients with chronic hepatitis C: A longitudinal analysis , 2013, Hepatology.

[4]  R. Gilroy,et al.  IL28B genotype and the expression of ISGs in normal liver , 2013, Liver international : official journal of the International Association for the Study of the Liver.

[5]  J. Kaiser.  Biomedical research. NIH to phase out most chimp research. , 2013, Science.

[6]  M. Gale,et al.  Regulation of hepatic innate immunity by hepatitis C virus , 2013, Nature Medicine.

[7]  M. Heim.  25 years of interferon-based treatment of chronic hepatitis C: an epoch coming to an end , 2013, Nature Reviews Immunology.

[8]  M. Bochud,et al.  IL28B expression depends on a novel TT/-G polymorphism which improves HCV clearance prediction , 2013, The Journal of experimental medicine.

[9]  Barbara Rehermann,et al.  A variant upstream of IFNL3 (IL28B) creating a new interferon gene IFNL4 is associated with impaired clearance of hepatitis C virus , 2013, Nature Genetics.

[10]  L. Kaderali,et al.  Identification of type I and type II interferon‐induced effectors controlling hepatitis C virus replication , 2012, Hepatology.

[11]  A. Folgori,et al.  IL‐29 is the dominant type III interferon produced by hepatocytes during acute hepatitis C virus infection , 2012, Hepatology.

[12]  L. Terracciano,et al.  Interferon-γ-stimulated genes, but not USP18, are expressed in livers of patients with acute hepatitis C. , 2012, Gastroenterology.

[13]  R. Lempicki,et al.  Dysregulation of innate immunity in hepatitis C virus genotype 1 IL28B‐unfavorable genotype patients: Impaired viral kinetics and therapeutic response , 2012, Hepatology.

[14]  Qisheng Li,et al.  HCV infection induces a unique hepatic innate immune response associated with robust production of type III interferons. , 2012, Gastroenterology.

[15]  T. Liang,et al.  Hepatitis C: The End of the Beginning and Possibly the Beginning of the End , 2012, Annals of Internal Medicine.

[16]  Christopher T. Jones,et al.  Expression of Paramyxovirus V Proteins Promotes Replication and Spread of Hepatitis C Virus in Cultures of Primary Human Fetal Liver Cells , 2011, Hepatology.

[17]  Christopher T. Jones,et al.  Hepatitis C virus induces interferon‐λ and interferon‐stimulated genes in primary liver cultures , 2011, Hepatology.

[18]  K. Shianna,et al.  IL28B genotype is associated with differential expression of intrahepatic interferon‐stimulated genes in patients with chronic hepatitis C , 2010, Hepatology.

[19]  R. Hamatake,et al.  Gene Expression Profiling Indicates the Roles of Host Oxidative Stress, Apoptosis, Lipid Metabolism, and Intracellular Transport Genes in the Replication of Hepatitis C Virus , 2010, Journal of Virology.

[20]  Sangeeta N. Bhatia,et al.  Persistent hepatitis C virus infection in microscale primary human hepatocyte cultures , 2010, Proceedings of the National Academy of Sciences.

[21]  Sangeeta N. Bhatia,et al.  Real-time imaging of hepatitis C virus infection using a fluorescent cell-based reporter system , 2010, Nature Biotechnology.

[22]  F. Chisari,et al.  Hepatitis C virus blocks interferon effector function by inducing protein kinase R phosphorylation. , 2009, Cell host & microbe.

[23]  David B. Goldstein,et al.  Genetic variation in IL28B and spontaneous clearance of hepatitis C virus , 2009, Nature.

[24]  Tuya Shilagard,et al.  Visualizing hepatitis C virus infections in human liver by two-photon microscopy. , 2009, Gastroenterology.

[25]  Jacques Fellay,et al.  Genetic variation in IL28B predicts hepatitis C treatment-induced viral clearance , 2009, Nature.

[26]  B. Sáinz,et al.  Three-dimensional Huh7 cell culture system for the study of Hepatitis C virus infection , 2009, Virology Journal.

[27]  Michael G. Katze,et al.  Genomic Analysis Reveals a Potential Role for Cell Cycle Perturbation in HCV-Mediated Apoptosis of Cultured Hepatocytes , 2009, PLoS pathogens.

[28]  Christopher T. Jones,et al.  Cell culture–produced hepatitis C virus does not infect peripheral blood mononuclear cells , 2008, Hepatology.

[29]  Thomas D. Schmittgen,et al.  Analyzing real-time PCR data by the comparative CT method , 2008, Nature Protocols.

[30]  Edward J Oakeley,et al.  Interferon signaling and treatment outcome in chronic hepatitis C , 2008, Proceedings of the National Academy of Sciences.

[31]  Pardis C Sabeti,et al.  Genome-wide detection and characterization of positive selection in human populations , 2007, Nature.

[32]  Toshiaki Maruyama,et al.  Complete Replication of Hepatitis C Virus in Cell Culture , 2005, Science.

[33]  S. Lemon,et al.  Distinct Poly(I-C) and Virus-activated Signaling Pathways Leading to Interferon-β Production in Hepatocytes* , 2005, Journal of Biological Chemistry.

[34]  S. Lemon,et al.  Intrahepatic Gene Expression during Chronic Hepatitis C Virus Infection in Chimpanzees , 2004, Journal of Virology.

[35]  L. Wodicka,et al.  Genomic analysis of the host response to hepatitis C virus infection , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[36]  R. Lanford,et al.  DNA Microarray Analysis of Chimpanzee Liver during Acute Resolving Hepatitis C Virus Infection , 2001, Journal of Virology.

[37]  E. Petricoin,et al.  Laser Capture Microdissection , 1996, Science.

[38]  C. Rui.  Genetic variation in IL-28B and spontaneous clearance of hepatitis C virus , 2012 .