High-Shear Stress Sensitizes Platelets to Subsequent Low-Shear Conditions

[1]  Deepak L. Bhatt,et al.  Importance of platelets and platelet response in acute coronary syndromes , 2009, Cleveland Clinic Journal of Medicine.

[2]  Alberto Redaelli,et al.  Platelet Activation Due to Hemodynamic Shear Stresses: Damage Accumulation Model and Comparison to In Vitro Measurements , 2008, ASAIO journal.

[3]  P. Bray Platelet hyperreactivity: predictive and intrinsic properties. , 2007, Hematology/oncology clinics of North America.

[4]  A. Piccin,et al.  Circulating microparticles: pathophysiology and clinical implications. , 2007, Blood reviews.

[5]  P. Bray,et al.  Platelet hyperreactivity generalizes to multiple forms of stimulation , 2006, Journal of thrombosis and haemostasis : JTH.

[6]  J. Hathcock Flow effects on coagulation and thrombosis. , 2006, Arteriosclerosis, thrombosis, and vascular biology.

[7]  Danny Bluestein,et al.  Towards optimization of the thrombogenic potential of blood recirculating cardiovascular devices using modeling approaches , 2006, Expert review of medical devices.

[8]  P. Bray,et al.  Aggregometry detects platelet hyperreactivity in healthy individuals. , 2005, Blood.

[9]  Danny Bluestein,et al.  Flow-induced platelet activation in a St. Jude mechanical heart valve, a trileaflet polymeric heart valve, and a St. Jude tissue valve. , 2005, Artificial organs.

[10]  J. Freedman,et al.  Pathologic high shear stress induces apoptosis events in human platelets. , 2004, Biochemical and biophysical research communications.

[11]  Ajit P Yoganathan,et al.  Fluid mechanics of heart valves. , 2004, Annual review of biomedical engineering.

[12]  J. Jesty,et al.  A Shear-restricted Pathway of Platelet Procoagulant Activity Is Regulated by IQGAP1* , 2004, Journal of Biological Chemistry.

[13]  P. Hufnagl,et al.  Antiinflammatory activity of astragaloside IV is mediated by inhibition of NF-κB activation and adhesion molecule expression , 2003, Thrombosis and Haemostasis.

[14]  P. Bray,et al.  Duration of exposure to high fluid shear stress is critical in shear-induced platelet activation-aggregation , 2003, Thrombosis and Haemostasis.

[15]  A. Fraser,et al.  A New Scoring System to Determine Thromboembolic Risk After Heart Valve Replacement , 2003, Circulation.

[16]  H. Shankaran,et al.  Aspects of hydrodynamic shear regulating shear-induced platelet activation and self-association of von Willebrand factor in suspension. , 2003, Blood.

[17]  P. Perrotta,et al.  Platelet activation in a circulating flow loop: combined effects of shear stress and exposure time , 2003, Platelets.

[18]  Larry V McIntire,et al.  Platelet Aggregation and Activation under Complex Patterns of Shear Stress , 2002, Thrombosis and Haemostasis.

[19]  Wei Yin,et al.  Flow induced platelet activation in mechanical heart valves - in vitro studies , 2002, Proceedings of the Second Joint 24th Annual Conference and the Annual Fall Meeting of the Biomedical Engineering Society] [Engineering in Medicine and Biology.

[20]  S. Nomura Function and Clinical Significance of Platelet-Derived Microparticles , 2001, International journal of hematology.

[21]  Hwa Liang Leo,et al.  Bileaflet Aortic Valve Prosthesis Pivot Geometry Influences Platelet Secretion and Anionic Phospholipid Exposure , 2001, Annals of Biomedical Engineering.

[22]  M Grigioni,et al.  A discussion on the threshold limit for hemolysis related to Reynolds shear stress. , 1999, Journal of biomechanics.

[23]  J Jesty,et al.  Acetylated prothrombin as a substrate in the measurement of the procoagulant activity of platelets: elimination of the feedback activation of platelets by thrombin. , 1999, Analytical biochemistry.

[24]  Y Komiyama,et al.  High shear stress can initiate both platelet aggregation and shedding of procoagulant containing microparticles. , 1996, Blood.

[25]  J. Moake,et al.  Platelets and shear stress. , 1996, Blood.

[26]  C. Akins,et al.  Results with mechanical cardiac valvular prostheses. , 1995, The Annals of thoracic surgery.

[27]  E. Simons,et al.  Cytoplasmic Ca2+ is necessary for thrombin-induced platelet activation. , 1989, The Journal of biological chemistry.

[28]  G. Grunkemeier,et al.  Low risk of thrombosis and serious embolic events despite low-intensity anticoagulation. Experience with 1,004 Medtronic Hall valves. , 1988, Circulation.

[29]  L. Edmunds,et al.  Thrombotic and bleeding complications of prosthetic heart valves. , 1987, The Annals of thoracic surgery.

[30]  H. Goldsmith,et al.  Rheological Aspects of Thrombosis and Haemostasis: Basic Principles and Applications , 1986, Thrombosis and Haemostasis.

[31]  H Schmid-Schönbein,et al.  Platelet and Coagulation Parameters Following Millisecond Exposure to Laminar Shear Stress , 1985, Thrombosis and Haemostasis.

[32]  J. Moake,et al.  Platelet response to shear stress: changes in serotonin uptake, serotonin release, and ADP induced aggregation. , 1978, Thrombosis research.

[33]  Katherine Schulz-Heik,et al.  Pathophysiology of Haemostasis and Thrombosis Preprint Copy The Extent of Platelet Activation under Shear Depends on Platelet Count : Differential Expression of Anionic Phospholipid and Factor Va , 2006 .

[34]  D. Ku,et al.  Fluid mechanics of vascular systems, diseases, and thrombosis. , 1999, Annual review of biomedical engineering.

[35]  T. Nome,et al.  Shear stress activation of platelets with subsequent refractoriness. , 1987, Thrombosis research.

[36]  C. Guimont,et al.  Experimental investigation of the rheological activation of blood platelets. , 1985, Biorheology.

[37]  L Zuckerman,et al.  Shear-induced activation of platelets. , 1979, Journal of biomechanics.

[38]  J. Moake,et al.  THE RESPONSE OF HUMAN PLATELETS TO SHEAR STRESS AT SHORT EXPOSURE TIMES , 1977, Transactions - American Society for Artificial Internal Organs.

[39]  S P Sutera,et al.  Deformation and fragmentation of human red blood cells in turbulent shear flow. , 1975, Biophysical journal.

[40]  J D Hellums,et al.  Response of human platelets to sheer stress. , 1975, Transactions - American Society for Artificial Internal Organs.