Incidence, molecular characteristics, and imaging features of “clinically-defined pseudoprogression” in newly diagnosed glioblastoma treated with chemoradiation
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W. Mason | T. Cloughesy | R. Henriksson | O. Chinot | R. Nishikawa | L. Abrey | B. Ellingson | F. Saran | Catalina Raymond | H. Tatekawa | Jacob Schlossman | W. Wick | Josep Garcia | S. Shabani | A. Hagiwara | Akifumi Hagiwara | H. Tatekawa
[1] Ajit S. Divakaruni,et al. “Aerobic glycolytic imaging” of human gliomas using combined pH-, oxygen-, and perfusion-weighted magnetic resonance imaging , 2021, NeuroImage: Clinical.
[2] H. Uetani,et al. Preferential tumor localization in relation to 18F-FDOPA uptake for lower‐grade gliomas , 2021, Journal of Neuro-Oncology.
[3] V. Puduvalli,et al. Characterizing benefit from temozolomide in MGMT promoter unmethylated and methylated glioblastoma: a systematic review and meta-analysis , 2020, Neuro-oncology advances.
[4] M. Nagane,et al. Survival in patients with glioblastoma at a first progression does not correlate with isocitrate dehydrogenase (IDH)1 gene mutation status , 2020, Japanese journal of clinical oncology.
[5] S. Aoki,et al. Variability and Standardization of Quantitative Imaging , 2020, Investigative radiology.
[6] Martin Bendszus,et al. Location-Dependent Patient Outcome and Recurrence Patterns in IDH1-Wildtype Glioblastoma , 2019, Cancers.
[7] S. Choi,et al. Incorporating diffusion- and perfusion-weighted MRI into a radiomics model improves diagnostic performance for pseudoprogression in glioblastoma patients , 2018, Neuro-oncology.
[8] Marion Smits,et al. Pseudoprogression of brain tumors , 2018, Journal of magnetic resonance imaging : JMRI.
[9] T. Huber,et al. Retrospective Analysis of Radiological Recurrence Patterns in Glioblastoma, Their Prognostic Value And Association to Postoperative Infarct Volume , 2018, Scientific Reports.
[10] E. Dezamis,et al. Prognostic factors for survival in adult patients with recurrent glioblastoma: a decision-tree-based model , 2018, Journal of Neuro-Oncology.
[11] P. Lambin,et al. Radiomics: the bridge between medical imaging and personalized medicine , 2017, Nature Reviews Clinical Oncology.
[12] C. Caeiro,et al. Current Standards of Care in Glioblastoma Therapy , 2017 .
[13] J. Boxerman,et al. Pseudoprogression, radionecrosis, inflammation or true tumor progression? challenges associated with glioblastoma response assessment in an evolving therapeutic landscape , 2017, Journal of Neuro-Oncology.
[14] P. V. van Laar,et al. Incidence of Tumour Progression and Pseudoprogression in High-Grade Gliomas: a Systematic Review and Meta-Analysis , 2017, Clinical Neuroradiology.
[15] P. Wen,et al. Baseline pretreatment contrast enhancing tumor volume including central necrosis is a prognostic factor in recurrent glioblastoma: evidence from single and multicenter trials , 2017, Neuro-oncology.
[16] Steffen Löck,et al. Image biomarker standardisation initiative , 2016 .
[17] M. Davis. Glioblastoma: Overview of Disease and Treatment. , 2016, Clinical journal of oncology nursing.
[18] R. Bourgon,et al. Patients With Proneural Glioblastoma May Derive Overall Survival Benefit From the Addition of Bevacizumab to First-Line Radiotherapy and Temozolomide: Retrospective Analysis of the AVAglio Trial. , 2015, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[19] Benjamin M Ellingson,et al. Pros and cons of current brain tumor imaging. , 2014, Neuro-oncology.
[20] Benjamin M Ellingson,et al. Recurrent glioblastoma treated with bevacizumab: contrast-enhanced T1-weighted subtraction maps improve tumor delineation and aid prediction of survival in a multicenter clinical trial. , 2014, Radiology.
[21] K. Hoang-Xuan,et al. Bevacizumab plus radiotherapy-temozolomide for newly diagnosed glioblastoma. , 2014, The New England journal of medicine.
[22] T. Tominaga,et al. The Association of Subventricular Zone Involvement at Recurrence with Survival after Repeat Surgery in Patients with Recurrent Glioblastoma , 2013, Neurologia medico-chirurgica.
[23] Tae Min Kim,et al. Differentiation of true progression from pseudoprogression in glioblastoma treated with radiation therapy and concomitant temozolomide: comparison study of standard and high-b-value diffusion-weighted imaging. , 2013, Radiology.
[24] Robert J. Harris,et al. Anatomic localization of O6-methylguanine DNA methyltransferase (MGMT) promoter methylated and unmethylated tumors: A radiographic study in 358 de novo human glioblastomas , 2012, NeuroImage.
[25] A G Sorensen,et al. Pseudoprogression and Pseudoresponse: Imaging Challenges in the Assessment of Posttreatment Glioma , 2011, American Journal of Neuroradiology.
[26] W. Shi,et al. Potential utility of conventional MRI signs in diagnosing pseudoprogression in glioblastoma , 2011, Neurology.
[27] P. Black,et al. Scale to predict survival after surgery for recurrent glioblastoma multiforme. , 2010, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[28] Jianhui Zhong,et al. Changes in relative cerebral blood volume 1 month after radiation-temozolomide therapy can help predict overall survival in patients with glioblastoma. , 2010, Radiology.
[29] A. Sahgal,et al. Pseudoprogression Following Chemoradiotherapy for Glioblastoma Multiforme , 2010, Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques.
[30] P. Box. Changes in relative cerebral blood volume 1 month after radiation-temozolomide therapy can help predict overall survival in patients with glioblastoma , 2010 .
[31] J. Fike,et al. CNS complications of radiotherapy and chemotherapy , 2009, The Lancet.
[32] Bart Neyns,et al. Pseudoprogression after radiotherapy with concurrent temozolomide for high-grade glioma: clinical observations and working recommendations. , 2009, Surgical neurology.
[33] P. Wen,et al. Effect of adding temozolomide to radiation therapy on the incidence of pseudo-progression , 2009, Journal of Neuro-Oncology.
[34] Dieta Brandsma,et al. Incidence of early pseudo‐progression in a cohort of malignant glioma patients treated with chemoirradiation with temozolomide , 2008, Cancer.
[35] S. Karimi,et al. Pseudoprogression (PsPr) after concurrent radiotherapy (RT) and temozolomide (TMZ) for newly diagnosed glioblastoma multiforme (GBM) , 2008 .
[36] Dieta Brandsma,et al. Clinical features, mechanisms, and management of pseudoprogression in malignant gliomas. , 2008, The Lancet. Oncology.
[37] A. Brandes,et al. MGMT promoter methylation status can predict the incidence and outcome of pseudoprogression after concomitant radiochemotherapy in newly diagnosed glioblastoma patients. , 2008, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[38] N. Burnet,et al. Interpretation of Early Imaging after Concurrent Radiotherapy and Temozolomide for Glioblastoma , 2007 .
[39] M. J. van den Bent,et al. Immediate post-radiotherapy changes in malignant glioma can mimic tumor progression , 2004, Neurology.
[40] W. J. Oakes,et al. Reversible neurotoxicity following hyperfractionated radiation therapy of brain stem glioma. , 1991, Medical and pediatric oncology.
[41] H. Hirschberg,et al. Reversible oedema and necrosis after irradiation of the brain. Diagnostic procedures and clinical manifestations. , 1990, Acta oncologica.
[42] M. Langer,et al. [Reversible computed tomographic changes following brain tumor irradiation induced by the "early-delayed reaction" after radiation]. , 1986, Der Radiologe.