Associations of interactions between NLRP3 SNPs and HLA mismatch with acute and extensive chronic graft-versus-host diseases

[1]  G. Hill,et al.  Chronic graft-versus-host disease: biological insights from preclinical and clinical studies. , 2017, Blood.

[2]  G. Núñez,et al.  Mechanism and Regulation of NLRP3 Inflammasome Activation. , 2016, Trends in biochemical sciences.

[3]  Lara E Sucheston-Campbell,et al.  Replication of associations between genetic polymorphisms and chronic graft-versus-host disease. , 2016, Blood.

[4]  A. Sher,et al.  T helper 1 immunity requires complement-driven NLRP3 inflammasome activity in CD4+ T cells , 2016, Science.

[5]  Gabor T. Marth,et al.  A global reference for human genetic variation , 2015, Nature.

[6]  D. Munn,et al.  GVHD-associated, inflammasome-mediated loss of function in adoptively transferred myeloid-derived suppressor cells. , 2015, Blood.

[7]  A. Dickinson,et al.  Non‐HLA genomics: does it have a role in predicting haematopoietic stem cell transplantation outcome? , 2015, International journal of immunogenetics.

[8]  M. Idzko,et al.  MicroRNA-155-deficient dendritic cells cause less severe GVHD through reduced migration and defective inflammasome activation. , 2015, Blood.

[9]  B. Ryffel,et al.  The receptor NLRP3 is a transcriptional regulator of TH2 differentiation , 2015, Nature Immunology.

[10]  H. Greinix,et al.  National Institutes of Health Consensus Development Project on Criteria for Clinical Trials in Chronic Graft-versus-Host Disease , 2015 .

[11]  K. Matsuo,et al.  Biological significance of HLA locus matching in unrelated donor bone marrow transplantation. , 2015, Blood.

[12]  R. Suzuki,et al.  Impact of pretransplant body mass index on the clinical outcome after allogeneic hematopoietic SCT , 2014, Bone Marrow Transplantation.

[13]  P. Ljungman.  The role of cytomegalovirus serostatus on outcome of hematopoietic stem cell transplantation , 2014, Current opinion in hematology.

[14]  J. Ritz,et al.  Current issues in chronic graft-versus-host disease. , 2014, Blood.

[15]  J. Tiercy.  HLA-C Incompatibilities in Allogeneic Unrelated Hematopoietic Stem Cell Transplantation , 2014, Front. Immunol..

[16]  S. Jenna,et al.  Genetic interaction networks: better understand to better predict , 2013, Front. Genet..

[17]  A. Ganser,et al.  CMV serostatus still has an important prognostic impact in de novo acute leukemia patients after allogeneic stem cell transplantation: a report from the Acute Leukemia Working Party of EBMT. , 2013, Blood.

[18]  K. Schwarz,et al.  High-resolution HLA matching in hematopoietic stem cell transplantation: a retrospective collaborative analysis. , 2013, Blood.

[19]  M. Saleh,et al.  Functions of NOD-Like Receptors in Human Diseases , 2013, Front. Immunol..

[20]  M. Idzko,et al.  The Nlrp3 inflammasome regulates acute graft-versus-host disease , 2013, The Journal of experimental medicine.

[21]  P. Goswami,et al.  Association of NALP2 polymorphism with arsenic induced skin lesions and other health effects. , 2013, Mutation research.

[22]  S. Iacobelli.  Suggestions on the use of statistical methodologies in studies of the European Group for Blood and Marrow Transplantation , 2013, Bone Marrow Transplantation.

[23]  R. Spritz,et al.  NLRP1 haplotypes associated with vitiligo and autoimmunity increase interleukin-1β processing via the NLRP1 inflammasome , 2013, Proceedings of the National Academy of Sciences.

[24]  J. Madrigal,et al.  NOD2 Polymorphisms and Their Impact on Haematopoietic Stem Cell Transplant Outcome , 2012, Bone marrow research.

[25]  H. Inoko,et al.  Single nucleotide polymorphisms and outcome risk in unrelated mismatched hematopoietic stem cell transplantation: an exploration study. , 2012, Blood.

[26]  J. Bertin,et al.  Autolytic Proteolysis within the Function to Find Domain (FIIND) Is Required for NLRP1 Inflammasome Activity* , 2012, The Journal of Biological Chemistry.

[27]  L. Zhao,et al.  Evaluation of published single nucleotide polymorphisms associated with acute GVHD. , 2012, Blood.

[28]  Richard A. Flavell,et al.  Inflammasomes in health and disease , 2012, Nature.

[29]  M. Horowitz,et al.  Risk factors for acute GVHD and survival after hematopoietic cell transplantation. , 2012, Blood.

[30]  P. Menu,et al.  The NLRP3 inflammasome in health and disease: the good, the bad and the ugly , 2011, Clinical and experimental immunology.

[31]  Gonçalo R. Abecasis,et al.  The variant call format and VCFtools , 2011, Bioinform..

[32]  Yusuke Nakamura,et al.  Association analysis of the NOD2 gene with susceptibility to graft-versus-host disease in a Japanese population , 2011, International journal of hematology.

[33]  R. Storb,et al.  Comparative analysis of risk factors for acute graft-versus-host disease and for chronic graft-versus-host disease according to National Institutes of Health consensus criteria. , 2011, Blood.

[34]  F. Aversa,et al.  Regression modeling of competing risk using R: an in depth guide for clinicians , 2010, Bone Marrow Transplantation.

[35]  Yusuke Nakamura,et al.  Associations of functional NLRP3 polymorphisms with susceptibility to food-induced anaphylaxis and aspirin-induced asthma. , 2009, The Journal of allergy and clinical immunology.

[36]  B. Sandmaier,et al.  Reduced-intensity conditioning regimen workshop: defining the dose spectrum. Report of a workshop convened by the center for international blood and marrow transplant research. , 2009, Biology of blood and marrow transplantation : journal of the American Society for Blood and Marrow Transplantation.

[37]  E. Montserrat,et al.  Common variants in NLRP2 and NLRP3 genes are strong prognostic factors for the outcome of HLA-identical sibling allogeneic stem cell transplantation. , 2008, Blood.

[38]  Y. Kodera.  The Japan Marrow Donor Program, the Japan Cord Blood Bank Network and the Asia Blood and Marrow Transplant Registry , 2008, Bone Marrow Transplantation.

[39]  Harriet Noreen,et al.  High-resolution donor-recipient HLA matching contributes to the success of unrelated donor marrow transplantation. , 2007, Blood.

[40]  Y. Kodera,et al.  Chronic graft‐versus‐host disease after allogeneic bone marrow transplantation from an unrelated donor: incidence, risk factors and association with relapse. A report from the Japan Marrow Donor Program , 2007, British journal of haematology.

[41]  Daniel Wolff,et al.  National Institutes of Health Consensus Development Project on Criteria for Clinical Trials in Chronic Graft-versus-Host Disease: I. The 2014 Diagnosis and Staging Working Group report. , 2005, Biology of blood and marrow transplantation : journal of the American Society for Blood and Marrow Transplantation.

[42]  Christian Stehlik,et al.  PAN1/NALP2/PYPAF2, an Inducible Inflammatory Mediator That Regulates NF-κB and Caspase-1 Activation in Macrophages* , 2004, Journal of Biological Chemistry.

[43]  F. Martinon,et al.  NALP3 forms an IL-1beta-processing inflammasome with increased activity in Muckle-Wells autoinflammatory disorder. , 2004, Immunity.

[44]  J. Bertin,et al.  CARD-8 Protein, a New CARD Family Member That Regulates Caspase-1 Activation and Apoptosis* , 2002, The Journal of Biological Chemistry.

[45]  E D Thomas,et al.  1994 Consensus Conference on Acute GVHD Grading. , 1995, Bone marrow transplantation.

[46]  K. Sullivan,et al.  Chronic graft-versus-host syndrome in man. A long-term clinicopathologic study of 20 Seattle patients. , 1980, The American journal of medicine.

[47]  P. Neiman,et al.  CLINICAL MANIFESTATIONS OF GRAFT‐VERSUS-HOST DISEASE IN HUMAN RECIPIENTS OF MARROW FROM HL‐A-MATCHED SIBLING DONOR,S , 1974, Transplantation.

[48]  Y. Atsuta.  Introduction of Transplant Registry Unified Management Program 2 (TRUMP2): scripts for TRUMP data analyses, part I (variables other than HLA-related data) , 2015, International Journal of Hematology.

[49]  I. Blau,et al.  Association between low uric acid levels and acute graft-versus-host disease , 2014, Annals of Hematology.

[50]  G. Alterovitz,et al.  Genomic studies of GVHD—lessons learned thus far , 2013, Bone Marrow Transplantation.