Flawed experimental design reveals the need for guidelines requiring appropriate positive controls in endocrine disruption research.
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Laura N. Vandenberg | Patricia A Hunt | Cheryl S Watson | Ana M Soto | Carlos Sonnenschein | John Peterson Myers | Gilbert Schoenfelder | Vasantha Padmanabhan | Cheryl S Rosenfeld | Frederick S vom Saal | Benson T Akingbemi | Scott M Belcher | David A Crain | David Crews | Linda C Guidice | Csaba Leranth | Angel Nadal | Nicholas Olea | Alan Schneyer | Richard W Stahlhut | Shanna H Swan | Laura N Vandenberg | Hong-Sheng Wang | Wade V Welshons | Robert T Zoeller | C. Léránth | P. Hunt | S. Swan | R. Zoeller | C. Sonnenschein | A. Soto | L. Vandenberg | D. A. Crain | W. Welshons | J. P. Myers | C. Rosenfeld | R. Stahlhut | B. Akingbemi | F. V. vom Saal | S. Belcher | V. Padmanabhan | D. Crews | C. Watson | A. Nadal | A. Schneyer | Hong-Sheng Wang | N. Olea | L. Guidice | G. Schoenfelder | D. Crain
[1] C. Richter,et al. In vivo effects of bisphenol A in laboratory rodent studies. , 2007, Reproductive toxicology.
[2] John Peterson Myers,et al. A Clash of Old and New Scientific Concepts in Toxicity, with Important Implications for Public Health , 2009, Environmental health perspectives.
[3] Frederick S vom Saal,et al. Estrogenic chemicals in plastic and oral contraceptives disrupt development of the fetal mouse prostate and urethra. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[4] Koji Arizono,et al. Why Public Health Agencies Cannot Depend on Good Laboratory Practices as a Criterion for Selecting Data: The Case of Bisphenol A , 2008, Environmental health perspectives.
[5] Saralees Nadarajah,et al. Letter to the editor , 2007, Int. Trans. Oper. Res..
[6] L. R. Harris,et al. Normal reproductive organ development in CF-1 mice following prenatal exposure to bisphenol A. , 1999, Toxicological sciences : an official journal of the Society of Toxicology.
[7] J. Furr,et al. Gestational and lactational exposure to ethinyl estradiol, but not bisphenol A, decreases androgen-dependent reproductive organ weights and epididymal sperm abundance in the male long evans hooded rat. , 2008, Toxicological sciences : an official journal of the Society of Toxicology.
[8] K. Crofton,et al. In utero and lactational exposure to bisphenol A, in contrast to ethinyl estradiol, does not alter sexually dimorphic behavior, puberty, fertility, and anatomy of female LE rats. , 2010, Toxicological sciences : an official journal of the Society of Toxicology.
[9] R. Tyl,et al. Two-generation reproductive toxicity study of dietary bisphenol A in CD-1 (Swiss) mice. , 2008, Toxicological sciences : an official journal of the Society of Toxicology.
[10] S Z Cagen,et al. Three-generation reproductive toxicity study of dietary bisphenol A in CD Sprague-Dawley rats. , 2002, Toxicological sciences : an official journal of the Society of Toxicology.
[11] J. Haseman,et al. Altered prostate growth and daily sperm production in male mice exposed prenatally to subclinical doses of 17alpha-ethinyl oestradiol. , 2001, Human reproduction.
[12] P. Phillips,et al. Time-course, dose-response, and age comparative sensitivity of N-methyl carbamates in rats. , 2010, Toxicological sciences : an official journal of the Society of Toxicology.
[13] W. Welshons,et al. Large effects from small exposures. II. The importance of positive controls in low-dose research on bisphenol A. , 2006, Environmental research.