in patients with congestive heart failure Hemodynamic and neurohumoral effects of continuous infusion of levosimendan
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M. Nieminen | W. Remme | G. Hasenfuss | V. Mitrović | O. Nyquist | J. Akkila | F. Kleber
[1] L. Toivonen,et al. Effects of Levosimendan on Cardiac Arrhythmia: Electrophysiologic and Ambulatory Electrocardiographic Findings in Phase II and Phase III Clinical Studies in Cardiac Failure , 1999 .
[2] G. Hasenfuss,et al. Influence of the novel inotropic agent levosimendan on isometric tension and calcium cycling in failing human myocardium. , 1998, Circulation.
[3] M. Sunagawa,et al. The novel calcium sensitizer levosimendan activates the ATP-sensitive K+ channel in rat ventricular cells. , 1997, The Journal of pharmacology and experimental therapeutics.
[4] M. Sunagawa,et al. Levosimendan, a novel Ca2+ sensitizer, activates the glibenclamide-sensitive K+ channel in rat arterial myocytes. , 1997, European journal of pharmacology.
[5] P. Kaheinen,et al. The role of cAMP- and cGMP-dependent protein kinases in the cardiac actions of the new calcium sensitizer, levosimendan. , 1997, Cardiovascular research.
[6] K. Någren,et al. Myocardial efficiency during calcium sensitization with levosimendan: A noninvasive study with positron emission tomography and echocardiography in healthy volunteers , 1997, Clinical pharmacology and therapeutics.
[7] A. Skene,et al. Randomised study of effect of ibopamine on survival in patients with advanced severe heart failure , 1997, The Lancet.
[8] C. Holubarsch. New inotropic concepts: rationale for and differences between calcium sensitizers and phosphodiesterase inhibitors. , 1997, Cardiology.
[9] N. Alpert,et al. Influence of isoproterenol and ouabain on excitation-contraction coupling, cross-bridge function, and energetics in failing human myocardium. , 1996, Circulation.
[10] J. Cohn,et al. The management of chronic heart failure. , 1996, The New England journal of medicine.
[11] L. Donck. Calcium‐Sensitizing Drugs: Positive Inotropy by Enhanced Sensitivity of the Contractile Apparatus to Calcium , 1996 .
[12] J. Levijoki,et al. Cardiac troponin C as a target protein for a novel calcium sensitizing drug, levosimendan. , 1995, Journal of molecular and cellular cardiology.
[13] J. Levijoki,et al. Troponin C–Mediated Calcium Sensitization Induced by Levosimendan Does Not Impair Relaxation , 1995, Journal of cardiovascular pharmacology.
[14] N. Alpert,et al. Effects of calcium sensitizers on intracellular calcium handling and myocardial energetics. , 1995, Journal of cardiovascular pharmacology.
[15] N. Kalkkinen,et al. Binding of a new Ca2+ sensitizer, levosimendan, to recombinant human cardiac troponin C. A molecular modelling, fluorescence probe, and proton nuclear magnetic resonance study. , 1994, The Journal of biological chemistry.
[16] M. Packer. The neurohormonal hypothesis: a theory to explain the mechanism of disease progression in heart failure. , 1992, Journal of the American College of Cardiology.
[17] D. DeMets,et al. Effect of oral milrinone on mortality in severe chronic heart failure. The PROMISE Study Research Group. , 1991, The New England journal of medicine.
[18] P. Merlet,et al. Physiologic Assessment of Milrinone Therapy in Severe Heart Failure Patients , 1991, Journal of Cardiovascular Pharmacology.
[19] K. Swedberg,et al. Hormones regulating cardiovascular function in patients with severe congestive heart failure and their relation to mortality. CONSENSUS Trial Study Group. , 1990, Circulation.
[20] M. Packer,et al. Hemodynamic changes mimicking a vasodilator drug response in the absence of drug therapy after right heart catheterization in patients with chronic heart failure. , 1985, Circulation.