Hypobaric Hypoxia Postconditioning Reduces Brain Damage and Improves Antioxidative Defense in the Model of Birth Asphyxia in 7-Day-Old Rats

[1]  N. Jones,et al.  The neuroprotective actions of hypoxic preconditioning and postconditioning in a neonatal rat model of hypoxic–ischemic brain injury , 2013, Brain Research.

[2]  A. Gunn,et al.  Window of Opportunity for Neuroprotection with an Antioxidant, Allene Oxide Synthase, after Hypoxia–Ischemia in Adult Male Rats , 2012, CNS neuroscience & therapeutics.

[3]  E. Rybnikova,et al.  Postconditioning by mild hypoxic exposures reduces rat brain injury caused by severe hypoxia , 2012, Neuroscience Letters.

[4]  Min Zhang,et al.  Intermittent Hypobaric Hypoxia Preconditioning Induced Brain Ischemic Tolerance by Up-Regulating Glial Glutamate Transporter-1 in Rats , 2012, Neurochemical Research.

[5]  K. Hendricks-Muñoz,et al.  Current management of the infant who presents with neonatal encephalopathy. , 2011, Current problems in pediatric and adolescent health care.

[6]  M. Johnston,et al.  Treatment advances in neonatal neuroprotection and neurointensive care , 2011, The Lancet Neurology.

[7]  M. Samoilov,et al.  Effects of Moderate Hypobaric Hypoxic Preconditioning on the Expression of the Transcription Factors pCREB and NF-κB in the Rat Hippocampus Before and After Severe Hypoxia , 2010, Neuroscience and Behavioral Physiology.

[8]  M. Maze,et al.  Preconditioning and Postinsult Therapies for Perinatal Hypoxic–Ischemic Injury at Term , 2010, Anesthesiology.

[9]  M. Bernaudin,et al.  Delayed Hypoxic Postconditioning Protects Against Cerebral Ischemia in the Mouse , 2009, Stroke.

[10]  M. Śmiałowska,et al.  Changes in the NPY immunoreactivity in gerbil hippocampus after hypoxic and ischemic preconditioning , 2009, Neuropeptides.

[11]  G. Pignataro,et al.  Post‐ischemic brain damage: effect of ischemic preconditioning and postconditioning and identification of potential candidates for stroke therapy , 2009, The FEBS journal.

[12]  G. Niu,et al.  Delayed Postconditioning Protects against Focal Ischemic Brain Injury in Rats , 2008, PloS one.

[13]  M. Gill,et al.  Hypoxia Ischemia-Mediated Cell Death in Neonatal Rat Brain , 2008, Neurochemical Research.

[14]  B. Luo,et al.  Ischemic Postconditioning Protects Against Global Cerebral Ischemia/Reperfusion-Induced Injury in Rats , 2008, Stroke.

[15]  S. Basu,et al.  Oxidative stress in perinatal asphyxia. , 2008, Pediatric neurology.

[16]  R. Simon,et al.  In Vivo and In Vitro Characterization of a Novel Neuroprotective Strategy for Stroke: Ischemic Postconditioning , 2008 .

[17]  F. Gao,et al.  Hypoxic postconditioning enhances the survival and inhibits apoptosis of cardiomyocytes following reoxygenation: role of peroxynitrite formation , 2006, Apoptosis.

[18]  J. Grotta,et al.  15d-Prostaglandin J2 Activates Peroxisome Proliferator-Activated Receptor-γ, Promotes Expression of Catalase, and Reduces Inflammation, Behavioral Dysfunction, and Neuronal Loss after Intracerebral Hemorrhage in Rats , 2006, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[19]  R. Sapolsky,et al.  Interrupting Reperfusion as a Stroke Therapy: Ischemic Postconditioning Reduces Infarct Size after Focal Ischemia in Rats , 2006, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[20]  W. Danysz,et al.  Neuroprotective potential of group I metabotropic glutamate receptor antagonists in two ischemic models , 2006, Neurochemistry International.

[21]  F. Kamme,et al.  Ischemic preconditioning prevents protein aggregation after transient cerebral ischemia , 2005, Neuroscience.

[22]  M. Samoilov,et al.  The effect of preconditioning on the Cu, Zn superoxide dismutase expression and enzyme activity in rat brain at the early period after severe hypobaric hypoxia , 2005, Neuroscience Research.

[23]  S. Arslan,et al.  Alterations in Antioxidant Enzyme Activities in Cerebrospinal Fluid Related with Severity of Hypoxic Ischemic Encephalopathy in Newborns , 2005, Neonatology.

[24]  R. Guyton,et al.  Hypoxic postconditioning reduces cardiomyocyte loss by inhibiting ROS generation and intracellular Ca2+ overload. , 2005, American journal of physiology. Heart and circulatory physiology.

[25]  H. Hagberg,et al.  Hypoxic Preconditioning Confers Long-Term Reduction of Brain Injury and Improvement of Neurological Ability in Immature Rats , 2005, Pediatric Research.

[26]  A. Leviton,et al.  Preconditioning and the developing brain. , 2004, Seminars in perinatology.

[27]  M. Samoilov,et al.  The augmentation of brain thioredoxin-1 expression after severe hypobaric hypoxia by the preconditioning in rats , 2004, Neuroscience Letters.

[28]  R. Vannucci,et al.  Secondary Energy Failure after Cerebral Hypoxia–Ischemia in the Immature Rat , 2004, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[29]  S. Vannucci,et al.  Hypoxia–ischemia in the immature brain , 2004, Journal of Experimental Biology.

[30]  I. Batinic-Haberle,et al.  Oxidants, antioxidants and the ischemic brain , 2004, Journal of Experimental Biology.

[31]  N. Knuckey,et al.  The protective effect of hypoxic preconditioning on cortical neuronal cultures is associated with increases in the activity of several antioxidant enzymes , 2004, Brain Research.

[32]  T. Kirino.  Ischemic Tolerance , 2002, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[33]  G. Buonocore,et al.  Free Radicals and Brain Damage in the Newborn , 2001, Neonatology.

[34]  P. Chan.  Reactive Oxygen Radicals in Signaling and Damage in the Ischemic Brain , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[35]  G. Lu,et al.  Changes of Superoxide Dismutase, Glutathione Perioxidase and Lipid Peroxides in the Brain of Mice Preconditioned by Hypoxia , 1999, Neurosignals.

[36]  R. Vannucci,et al.  Hypoxic Preconditioning and Hypoxic‐Ischemic Brain Damage in the Immature Rat: Pathologic and Metabolic Correlates , 1998, Journal of neurochemistry.

[37]  C. Epstein,et al.  Copper/zinc superoxide dismutase transgenic brain accumulates hydrogen peroxide after perinatal hypoxia ischemia , 1998, Annals of neurology.

[38]  Michael V. Johnston,et al.  Neurotransmitters and vulnerability of the developing brain , 1995, Brain and Development.

[39]  Marzena Wylezinska,et al.  Delayed (“Secondary”) Cerebral Energy Failure after Acute Hypoxia-Ischemia in the Newborn Piglet: Continuous 48-Hour Studies by Phosphorus Magnetic Resonance Spectroscopy , 1994, Pediatric Research.

[40]  R. Vannucci,et al.  Cerebral energy metabolism during hypoxia-ischemia and early recovery in immature rats. , 1992, The American journal of physiology.

[41]  R. M. Hays,et al.  ADH-induced depolymerization of F-actin in the toad bladder granular cell: a confocal microscope study. , 1992, The American journal of physiology.

[42]  J. Rice,et al.  The influence of immaturity on hypoxic‐ischemic brain damage in the rat , 1981, Annals of neurology.

[43]  Heng Zhao.  The Protective Effect of Ischemic Postconditioning Against Ischemic Injury: From the Heart to the Brain , 2008, Journal of Neuroimmune Pharmacology.