Activation of Notch-1 in oral epithelial cells by P. gingivalis triggers the expression of the antimicrobial protein PLA2-IIA
暂无分享,去创建一个
A. Stromberg | A. Al-Attar | J. Ebersole | Y. Alimova | S. Kirakodu | M. J. Novak | L. Orraca | J. Gonzalez-Martinez | Melween I. Martínez | O. A. Gonzalez | Anastasia Kozal | M. Martínez
[1] L. Journet,et al. Salmonella Typhimurium utilizes a T6SS-mediated antibacterial weapon to establish in the host gut , 2016, Proceedings of the National Academy of Sciences.
[2] Nattiya Hirankarn,et al. Hepatitis B Virus HBx Activates Notch Signaling via Delta-Like 4/Notch1 in Hepatocellular Carcinoma , 2016, PloS one.
[3] R. Lamont,et al. FOXO responses to Porphyromonas gingivalis in epithelial cells , 2015, Cellular microbiology.
[4] M. Gelb,et al. Platelet microparticles are internalized in neutrophils via the concerted activity of 12-lipoxygenase and secreted phospholipase A2-IIA , 2015, Proceedings of the National Academy of Sciences.
[5] Y. Miki,et al. A new era of secreted phospholipase A2 , 2015, Journal of Lipid Research.
[6] R. Lamont,et al. Polymicrobial synergy and dysbiosis in inflammatory disease. , 2015, Trends in molecular medicine.
[7] G. Hajishengallis,et al. Periodontitis: from microbial immune subversion to systemic inflammation , 2014, Nature Reviews Immunology.
[8] D. Leduc,et al. Pseudomonas aeruginosa eradicates Staphylococcus aureus by manipulating the host immunity , 2014, Nature Communications.
[9] J. Ebersole,et al. Cytokine gene expression profiles during initiation, progression and resolution of periodontitis. , 2014, Journal of clinical periodontology.
[10] David Artis,et al. Intestinal epithelial cells: regulators of barrier function and immune homeostasis , 2014, Nature Reviews Immunology.
[11] S. Wallet,et al. Type 1 Diabetes–associated TLR Responsiveness of Oral Epithelial Cells , 2014, Journal of dental research.
[12] A. Iuga,et al. Mouse and human Notch-1 regulate mucosal immune responses , 2014, Mucosal Immunology.
[13] R. Lamont,et al. Establishment and characterization of a telomerase immortalized human gingival epithelial cell line. , 2013, Journal of periodontal research.
[14] R. Kruse,et al. The periodontal pathogen Porphyromonas gingivalis changes the gene expression in vascular smooth muscle cells involving the TGFbeta/Notch signalling pathway and increased cell proliferation , 2013, BMC Genomics.
[15] M. Blaut,et al. Differences in Mucosal Gene Expression in the Colon of Two Inbred Mouse Strains after Colonization with Commensal Gut Bacteria , 2013, PloS one.
[16] E. Elinav,et al. IL-22 Deficiency Alters Colonic Microbiota To Be Transmissible and Colitogenic , 2013, The Journal of Immunology.
[17] D. Fairlie,et al. An Inhibitor of Phospholipase A2 Group IIA Modulates Adipocyte Signaling and Protects Against Diet-Induced Metabolic Syndrome in Rats , 2012, Diabetes.
[18] K. G. Guruharsha,et al. The Notch signalling system: recent insights into the complexity of a conserved pathway , 2012, Nature Reviews Genetics.
[19] John D Lambris,et al. Low-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complement. , 2011, Cell host & microbe.
[20] A. Capobianco,et al. Notch signalling in solid tumours: a little bit of everything but not all the time , 2011, Nature Reviews Cancer.
[21] J. Ebersole,et al. HIV-1 Reactivation Induced by the Periodontal Pathogens Fusobacterium nucleatum and Porphyromonas gingivalis Involves Toll-Like Receptor 2 and 9 Activation in Monocytes/Macrophages , 2010, Clinical and Vaccine Immunology.
[22] Qiang Xu,et al. Porphyromonas gingivalis lipopolysaccharide inhibits the osteoblastic differentiation of preosteoblasts by activating Notch1 signaling , 2010, Journal of cellular physiology.
[23] J. Ebersole,et al. HIV-1 Reactivation Induced by the Periodontal Pathogens Fusobacterium nucleatum and Porphyromonas gingivalis Involves Toll-Like Receptor 4 and 9 Activation in Monocytes/Macrophages , 2010, Clinical and Vaccine Immunology.
[24] M. Charvéron,et al. TLR2 sensing of F. nucleatum and S. sanguinis distinctly triggered gingival innate response. , 2009, Cytokine.
[25] Jiahuai Han,et al. Integrated regulation of Toll-like receptor responses by Notch and interferon-gamma pathways. , 2008, Immunity.
[26] M. Steffen,et al. Effects of Age and Oral Disease on Systemic Inflammatory and Immune Parameters in Nonhuman Primates , 2008, Clinical and Vaccine Immunology.
[27] M. Herzberg,et al. Porphyromonas gingivalis induces CCR5-dependent transfer of infectious HIV-1 from oral keratinocytes to permissive cells , 2008, Retrovirology.
[28] L. L. Reed,et al. Notch-independent regulation of Hes-1 expression by c-Jun N-terminal kinase signaling in human endothelial cells , 2006, Laboratory Investigation.
[29] E. Robertson,et al. Intracellular Activated Notch1 Is Critical for Proliferation of Kaposi's Sarcoma-Associated Herpesvirus-Associated B-Lymphoma Cell Lines In Vitro , 2006, Journal of Virology.
[30] D. Leduc,et al. Neisseria meningitidis pili induce type‐IIA phospholipase A2 expression in alveolar macrophages , 2005, FEBS letters.
[31] S. Way,et al. Porphyromonas gingivalis Lipopolysaccharide Contains Multiple Lipid A Species That Functionally Interact with Both Toll-Like Receptors 2 and 4 , 2004, Infection and Immunity.
[32] J. Potempa,et al. Gingipains, the major cysteine proteinases and virulence factors of Porphyromonas gingivalis: structure, function and assembly of multidomain protein complexes. , 2003, Current protein & peptide science.
[33] R. Maier,et al. Porphyromonas gingivalis Lipopolysaccharide Is Both Agonist and Antagonist for p38 Mitogen-Activated Protein Kinase Activation , 2002, Infection and Immunity.
[34] T. Taga,et al. Cerebrospinal fluid interleukin‐6 levels in hypertensive encephalopathy: A possible marker of disease activity , 2001, Annals of neurology.
[35] D. Roopenian,et al. Genetic Control of Susceptibility toPorphyromonas gingivalis-Induced Alveolar Bone Loss in Mice , 2000, Infection and Immunity.
[36] K. Scott,et al. Type IIA Secretory Phospholipase A2 Up-Regulates Cyclooxygenase-2 and Amplifies Cytokine-Mediated Prostaglandin Production in Human Rheumatoid Synoviocytes1 , 2000, The Journal of Immunology.
[37] M. Gessler,et al. Comparative analysis of the human and mouse Hey1 promoter: Hey genes are new Notch target genes. , 2000, Biochemical and biophysical research communications.
[38] W. Hahn,et al. Human Keratinocytes That Express hTERT and Also Bypass a p16INK4a-Enforced Mechanism That Limits Life Span Become Immortal yet Retain Normal Growth and Differentiation Characteristics , 2000, Molecular and Cellular Biology.
[39] A. Israël,et al. Delta-1 Activation of Notch-1 Signaling Results inHES-1 Transactivation , 1998, Molecular and Cellular Biology.
[40] S. Holt,et al. Immunization with Porphyromonas gingivalis cysteine protease: effects on experimental gingivitis and ligature-induced periodontitis in Macaca fascicularis. , 1998, Journal of periodontology.
[41] C. M. Belton,et al. Local Chemokine Paralysis, a Novel Pathogenic Mechanism for Porphyromonas gingivalis , 1998, Infection and Immunity.
[42] S. Socransky,et al. Microbial complexes in subgingival plaque. , 1998, Journal of clinical periodontology.
[43] J. Mudgett,et al. A Natural Disruption of the Secretory Group II Phospholipase A2 Gene in Inbred Mouse Strains (*) , 1995, The Journal of Biological Chemistry.
[44] H. Tojo,et al. Group II phospholipase A2 in human gingiva with periodontal disease , 1995, Mediators of inflammation.
[45] L. Fuentes,et al. Secreted phospholipase A2 type IIA as a mediator connecting innate and adaptive immunity: new role in atherosclerosis. , 2009, Cardiovascular research.
[46] Tetsuya Nakamura,et al. Requirement of Notch activation during regeneration of the intestinal epithelia. , 2009, American journal of physiology. Gastrointestinal and liver physiology.
[47] G. Graham,et al. Antibacterial actions of secreted phospholipases A2. Review. , 2008, Biochimica et biophysica acta.
[48] T. Imamura. The role of gingipains in the pathogenesis of periodontal disease. , 2003, Journal of periodontology.
[49] H. Asakura,et al. Correlation between serum phospholipase A(2) IIA levels and histological activity in patients with ulcerative colitis. , 2002, International journal of colorectal disease.
[50] T. Nagata,et al. Phospholipase A2 Activity in Gingival Crevicular Fluid from Patients with Periodontal Disease: A possible Marker of Disease Activity , 1994, Mediators of inflammation.