Site‐specific immunophenotyping of keloid disease demonstrates immune upregulation and the presence of lymphoid aggregates
暂无分享,去创建一个
A. Bayat | R. Paus | Werner Müller | I. Chaudhry | R. Byers | R. Bagabir
[1] R. Dobson,et al. British Association of Dermatologists , 2014 .
[2] A. Bayat,et al. Long‐term organ culture of keloid disease tissue , 2012, Experimental dermatology.
[3] A. Bayat,et al. Extracellular matrix molecules implicated in hypertrophic and keloid scarring , 2012, Journal of the European Academy of Dermatology and Venereology : JEADV.
[4] Michael Sorkin,et al. Mechanical force prolongs acute inflammation via T‐cell‐dependent pathways during scar formation , 2011, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[5] V. Jala,et al. Pivotal role of dermal IL-17-producing γδ T cells in skin inflammation. , 2011, Immunity.
[6] T. Wynn,et al. Protective and pathogenic functions of macrophage subsets , 2011, Nature Reviews Immunology.
[7] A. Bayat,et al. Upregulation of Toll-like receptors (TLRs) 6, 7, and 8 in keloid scars. , 2011, The Journal of investigative dermatology.
[8] Tomoko Mori,et al. Keloid exacerbation associated with adult T-cell leukemia/lymphoma cell infiltration. , 2011, European journal of dermatology : EJD.
[9] H. Ehrlich,et al. Modulatory effects of connexin‐43 expression on gap junction intercellular communications with mast cells and fibroblasts , 2011, Journal of cellular biochemistry.
[10] J. Colombel,et al. Lymphatics, tertiary lymphoid organs and the granulomas of Crohn’s disease: an immunohistochemical study , 2011, Alimentary pharmacology & therapeutics.
[11] C. Chizzolini,et al. Fibrosis and immune dysregulation in systemic sclerosis. , 2011, Autoimmunity reviews.
[12] N. Ayuob,et al. Cell Talk: A Phenomenon Observed in the Keloid Scar by Immunohistochemical Study , 2011, Applied immunohistochemistry & molecular morphology : AIMM.
[13] R. Byers,et al. Quantum dots brighten biological imaging. , 2011, Progress in histochemistry and cytochemistry.
[14] M. Si-Tahar,et al. Microbiota-induced tertiary lymphoid tissues aggravate inflammatory disease in the absence of RORγt and LTi cells , 2011, The Journal of experimental medicine.
[15] D. McGrouther,et al. Fibroblasts from the growing margin of keloid scars produce higher levels of collagen I and III compared with intralesional and extralesional sites: clinical implications for lesional site‐directed therapy , 2011, The British journal of dermatology.
[16] D. Evans,et al. A comparative study of quantitative immunohistochemistry and quantum dot immunohistochemistry for mutation carrier identification in Lynch syndrome , 2010, Journal of Clinical Pathology.
[17] K. Khosrotehrani,et al. Skin wound healing modulation by macrophages. , 2010, International journal of clinical and experimental pathology.
[18] Luke Barron,et al. Macrophages: Master Regulators of Inflammation and Fibrosis , 2010, Seminars in liver disease.
[19] P. De Baetselier,et al. Different tumor microenvironments contain functionally distinct subsets of macrophages derived from Ly6C(high) monocytes. , 2010, Cancer research.
[20] A. Mukhopadhyay,et al. Comparative proteomic analysis between normal skin and keloid scar , 2010, The British journal of dermatology.
[21] W. R. Lee,et al. Kimura’s disease mimicking an earlobe keloid , 2010, Clinical and experimental dermatology.
[22] R. Kimble,et al. Microscopic inflammatory foci in burn scars: data from a porcine burn model , 2010, Journal of cutaneous pathology.
[23] R. Paus,et al. Corticotropin-releasing hormone stimulates the in situ generation of mast cells from precursors in the human hair follicle mesenchyme. , 2010, The Journal of investigative dermatology.
[24] J. Roes,et al. Differential Roles of Macrophages in Diverse Phases of Skin Repair , 2010, The Journal of Immunology.
[25] A. Bayat,et al. Molecular dissection of abnormal wound healing processes resulting in keloid disease , 2010, Wound Repair and Regeneration.
[26] A. Bayat,et al. The Influence of Surgical Excision Margins on Keloid Prognosis , 2010, Annals of plastic surgery.
[27] S. Shi,et al. Tumor-Like Stem Cells Derived from Human Keloid Are Governed by the Inflammatory Niche Driven by IL-17/IL-6 Axis , 2009, PloS one.
[28] Scott N. Mueller,et al. Stromal cell contributions to the homeostasis and functionality of the immune system , 2009, Nature Reviews Immunology.
[29] G. Juckett,et al. Management of keloids and hypertrophic scars. , 2009, American Family Physician.
[30] E. Clementi,et al. Inflammatory and alternatively activated human macrophages attract vessel‐associated stem cells, relying on separate HMGB1‐ and MMP‐9‐dependent pathways , 2009, Journal of leukocyte biology.
[31] A. Cumano,et al. Inflammation Recapitulates the Ontogeny of Lymphoid Stromal Cells1 , 2009, The Journal of Immunology.
[32] J. Stark,et al. Coronary intraplaque hemorrhage evokes a novel atheroprotective macrophage phenotype. , 2009, The American journal of pathology.
[33] Y. Inoue,et al. Immunohistochemical characterization of the cellular infiltrate in localized scleroderma , 2008, International journal of dermatology.
[34] P. Scott,et al. Increased TGF‐β–producing CD4+ T lymphocytes in postburn patients and their potential interaction with dermal fibroblasts in hypertrophic scarring , 2007, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[35] T. Uchiyama,et al. Involvement of OX40 ligand+ mast cells in chronic GVHD after allogeneic hematopoietic stem cell transplantation , 2007, Bone Marrow Transplantation.
[36] M. Gaxiola,et al. Inducible bronchus-associated lymphoid tissue (iBALT) in patients with pulmonary complications of rheumatoid arthritis. , 2006, The Journal of clinical investigation.
[37] P. Arck,et al. Neuroimmunology of stress: skin takes center stage. , 2006, The Journal of investigative dermatology.
[38] M. Cesta. Normal Structure, Function, and Histology of Mucosa-Associated Lymphoid Tissue , 2006, Toxicologic pathology.
[39] N. Araki,et al. AM-3K, an Anti-macrophage Antibody, Recognizes CD163, a Molecule Associated with an Anti-inflammatory Macrophage Phenotype , 2006, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[40] V. Soumelis,et al. Cutting Edge: Nonproliferating Mature Immune Cells Form a Novel Type of Organized Lymphoid Structure in Idiopathic Pulmonary Fibrosis1 , 2006, The Journal of Immunology.
[41] T. Mcclanahan,et al. IL-23 is essential for T cell-mediated colitis and promotes inflammation via IL-17 and IL-6. , 2006, The Journal of clinical investigation.
[42] S. Liao,et al. Lymphoid organ development: from ontogeny to neogenesis , 2006, Nature Immunology.
[43] M. Delehanty,et al. Polarized Th2 cytokine production in patients with hypertrophic scar following thermal injury. , 2006, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.
[44] M. Tsai,et al. Mast Cells Enhance T Cell Activation: Importance of Mast Cell Costimulatory Molecules and Secreted TNF1 , 2006, The Journal of Immunology.
[45] D. Messadi,et al. Hypoxia-induced HIF-1 alpha accumulation is augmented in a co-culture of keloid fibroblasts and human mast cells: involvement of ERK1/2 and PI-3K/Akt. , 2006, Experimental cell research.
[46] R. Paus,et al. Immunology of the human nail apparatus: the nail matrix is a site of relative immune privilege. , 2005, The Journal of investigative dermatology.
[47] M. Zabel,et al. LYVE-1 expression on high endothelial venules (HEVs) of lymph nodes. , 2005, Lymphology.
[48] T. Cupedo,et al. Induction of secondary and tertiary lymphoid structures in the skin. , 2004, Immunity.
[49] P. Chu,et al. CD163: a specific marker of macrophages in paraffin-embedded tissue samples. , 2004, American journal of clinical pathology.
[50] Y. Okayama,et al. T Cell Proliferation by Direct Cross-Talk between OX40 Ligand on Human Mast Cells and OX40 on Human T Cells: Comparison of Gene Expression Profiles between Human Tonsillar and Lung-Cultured Mast Cells1 , 2004, The Journal of Immunology.
[51] M. Longaker,et al. Complex epithelial–mesenchymal interactions modulate transforming growth factor‐β expression in keloid‐derived cells , 2004, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[52] H. Ehrlich,et al. Mast cells promote fibroblast populated collagen lattice contraction through gap junction intercellular communication , 2004, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[53] R. Mebius. Organogenesis of lymphoid tissues , 2003, Nature Reviews Immunology.
[54] F. Wood,et al. [International clinical recommendations on scar management]. , 2002, Zentralblatt fur Chirurgie.
[55] Hung Huynh,et al. Fibroblasts cocultured with keloid keratinocytes: normal fibroblasts secrete collagen in a keloidlike manner. , 2002, American journal of physiology. Cell physiology.
[56] C. Castagnoli,et al. Role of T‐lymphocytes and cytokines in post‐burn hypertrophic scars , 2002, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[57] K. Harding,et al. Inflammatory-cell subpopulations in keloid scars. , 2001, British journal of plastic surgery.
[58] M. Santucci,et al. Keloids and hypertrophic scars of Caucasians show distinctive morphologic and immunophenotypic profiles , 2001, Virchows Archiv.
[59] M. Longaker,et al. Investigation of the influence of keloid-derived keratinocytes on fibroblast growth and proliferation in vitro. , 2001, Plastic and reconstructive surgery.
[60] B. Antus,et al. Influence of alternatively and classically activated macrophages on fibrogenic activities of human fibroblasts. , 2000, Cellular immunology.
[61] M. Kurosawa,et al. Effect of mast cell-derived mediators and mast cell-related neutral proteases on human dermal fibroblast proliferation and type I collagen production. , 2000, The Journal of allergy and clinical immunology.
[62] D. Tobin,et al. The human hair follicle immune system: cellular composition and immune privilege , 2000, The British journal of dermatology.
[63] D. Jackson,et al. LYVE-1, a New Homologue of the CD44 Glycoprotein, Is a Lymph-specific Receptor for Hyaluronan , 1999, The Journal of cell biology.
[64] C. Castagnoli,et al. Functional analysis of T lymphocytes infiltrating the dermis and epidermis of post-burn hypertrophic scar tissues. , 1999, Burns : journal of the International Society for Burn Injuries.
[65] C. Castagnoli,et al. Characterization of T-cell subsets infiltrating post-burn hypertrophic scar tissues. , 1997, Burns : journal of the International Society for Burn Injuries.
[66] J. Garlick,et al. Human mast cells activate fibroblasts: tryptase is a fibrogenic factor stimulating collagen messenger ribonucleic acid synthesis and fibroblast chemotaxis. , 1997, Journal of immunology.
[67] C. W. Martin,et al. The role of lymphocytes in wound healing. , 1990, British journal of plastic surgery.
[68] Koonin Aj. THE AETIOLOGY OF KELOIDS: A REVIEW OF THE LITERATURE AND A NEW HYPOTHESIS. , 1964 .
[69] R. Ogawa,et al. Keloid and hypertrophic scar: neurogenic inflammation hypotheses. , 2008, Medical hypotheses.
[70] A. J. Koonin. THE AETIOLOGY OF KELOIDS: A REVIEW OF THE LITERATURE AND A NEW HYPOTHESIS. , 1964, South African medical journal = Suid-Afrikaanse tydskrif vir geneeskunde.