Effects of whole body vibration on outer hair cells’ hearing response to distortion product otoacoustic emissions

[1]  N. Muluk,et al.  Effects of GSM-like radiofrequency on distortion product otoacoustic emissions of rabbits: comparison of infants versus adults. , 2009, International journal of pediatric otorhinolaryngology.

[2]  N. Muluk,et al.  Effects of extremely low frequency electromagnetic fields on distortion product otoacoustic emissions in rabbits. , 2009, Auris, nasus, larynx.

[3]  B. Xiao,et al.  Long-term administration of salicylate enhances prestin expression in rat cochlea , 2009, International journal of audiology.

[4]  Miao Zhu,et al.  Prestin up-regulation in chronic salicylate (aspirin) administration: An implication of functional dependence of prestin expression , 2008, Cellular and Molecular Life Sciences.

[5]  Jianhua Peng,et al.  Long-term sound conditioning increases distortion product otoacoustic emission amplitudes and decreases olivocochlear efferent reflex strength , 2007, Neuroreport.

[6]  Michael J. Griffin,et al.  Guide to good practice on whole-body vibration: non-binding guide to good practice for implementing Directive 2002/44/EC on the minimum health and safety requirements regarding the exposure of workers to the risks arising from physical agents (vibrations) , 2006 .

[7]  Hong-Bo Zhao,et al.  Paradoxical enhancement of active cochlear mechanics in long-term administration of salicylate. , 2005, Journal of neurophysiology.

[8]  Wojciech Dziewiszek,et al.  Experimental vibratory damage of the inner ear , 2005, European Archives of Oto-Rhino-Laryngology and Head & Neck.

[9]  B. Cone-Wesson,et al.  Asymmetric Cochlear Processing Mimics Hemispheric Specialization , 2004, Science.

[10]  Roger Frost,et al.  International Organization for Standardization (ISO) , 2004 .

[11]  Sebastiaan W. F. Meenderink,et al.  Distortion product otoacoustic emissions provide clues hearing mechanisms in the frog ear. , 2003, The Journal of the Acoustical Society of America.

[12]  A. Abou-Setta,et al.  The interaction of whole body vibration and noise on the cochlea , 2003 .

[13]  R. Hebbel,et al.  Amendment history : Erratum ( September 2000 ) Hypoxia / reoxygenation causes inflammatory response in transgenic sickle mice but not in normal mice , 2018 .

[14]  D. Homnick,et al.  Effects of chronic tobramycin treatment on distortion product otoacoustic emissions. , 1999, Ear and hearing.

[15]  Hugh W. Davies,et al.  Whole body vibrations and back disorders among motor vehicle drivers and heavy equipment operators: a review of the scientific evidence , 1999 .

[16]  R. Harrison,et al.  Increased otoacoustic-emission amplitude secondary to cochlear lesions. , 1998, The Journal of otolaryngology.

[17]  N. Harel,et al.  Basal Cochlear Lesions Result in Increased Amplitude of Otoacoustic Emissions , 1998, Audiology and Neurotology.

[18]  Masashi Suzuki,et al.  Expression of Intercellular Adhesion Molecule-1 during Inner Ear Inflammation , 1995, The Annals of otology, rhinology, and laryngology.

[19]  D. Henderson,et al.  The Relationship Among Distortion‐Product Otoacoustic Emissions, Evoked Potential Thresholds, and Outer Hair Cells Following Interrupted Noise Exposures , 1994, Ear and hearing.

[20]  B L Lonsbury-Martin,et al.  Evidence for two discrete sources of 2f1-f2 distortion-product otoacoustic emission in rabbit: I. Differential dependence on stimulus parameters. , 1992, The Journal of the Acoustical Society of America.

[21]  Glen K. Martin,et al.  Distortion Product Emissions in Humans , 1990, The Annals of otology, rhinology & laryngology. Supplement.

[22]  A. M. Brown,et al.  Acoustic distortion from rodent ears: A comparison of responses from rats, guinea pigs and gerbils , 1987, Hearing Research.

[23]  I. Pyykkö,et al.  Sensory-neural hearing loss during combined noise and vibration exposure , 1987, International archives of occupational and environmental health.

[24]  D. T. Kemp,et al.  Otoacoustic emissions, travelling waves and cochlear mechanisms , 1986, Hearing Research.

[25]  E. B. Clark,et al.  Workers' Compensation Board of British Columbia , 1981 .

[26]  D. Henderson,et al.  The interaction of whole body vibration and impulse noise. , 1980, The Journal of the Acoustical Society of America.

[27]  D. Kemp,et al.  The evoked cochlear mechanical response in laboratory primates , 1979, Archives of oto-rhino-laryngology.

[28]  J. Kimm,et al.  Auditory sensitivity in the rabbit determined by a conditional nictitating of membrane response , 1977 .

[29]  H. Miyake,et al.  Temporary hearing loss induced by noise and vibration. , 1972, The Journal of the Acoustical Society of America.

[30]  B. Lonsbury-Martin,et al.  Otoacoustic Emissions: Basic Studies in Mammalian Models , 2008 .

[31]  David T. Kemp,et al.  Otoacoustic Emissions: Concepts and Origins , 2008 .

[32]  Thomas Janssen,et al.  Otoacoustic Emissions as a Diagnostic Tool in a Clinical Context , 2008 .

[33]  Geoffrey A. Manley,et al.  Active processes and otoacoustic emissions in hearing , 2007 .

[34]  S. Torvinen Effect of whole body vibration on muscular performance, balance, and bone , 2003 .

[35]  Michael J. Griffin,et al.  Whole-body vibration , 2001 .

[36]  H. Takahashi,et al.  Sexual dimorphism and development of the human cochlea. Computer 3-D measurement. , 1991, Acta oto-laryngologica.

[37]  Barbara Harazin,et al.  Isolated and combined effects of prolonged exposures to noise and whole-body vibration on hearing, vision and strain , 1988, International archives of occupational and environmental health.

[38]  R. Salvi,et al.  Influence of vibration on asymptotic threshold shift produced by impulse noise. , 1981, Audiology : official organ of the International Society of Audiology.

[39]  S. Osako,et al.  Temporary threshold shifts produced by exposure to vibration, noise, and vibration-plus-noise. , 1974, Acta oto-laryngologica.

[40]  Leo L. Beranek,et al.  Noise and vibration control , 1971 .