Combination of a p53-activating CP-31398 and an MDM2 or a FAK inhibitor produces growth suppressive effects in mesothelioma with wild-type p53 genotype
暂无分享,去创建一个
M. Shingyoji | M. Tagawa | H. Shimada | K. Hiroshima | Y. Tada | Michiko Hanazono | T. Morinaga | Boya Zhong | T. Nguyễn
[1] Xiao-lan Li,et al. Hippo component YAP promotes focal adhesion and tumour aggressiveness via transcriptionally activating THBS1/FAK signalling in breast cancer , 2018, Journal of experimental & clinical cancer research : CR.
[2] D. Sterman,et al. Updates in the diagnosis and treatment of malignant pleural mesothelioma , 2018, Current opinion in pulmonary medicine.
[3] M. Shingyoji,et al. Heat shock protein 90 inhibitors augment endogenous wild-type p53 expression but down-regulate the adenovirally-induced expression by inhibiting a proteasome activity , 2018, Oncotarget.
[4] R. Plummer,et al. A phase I, pharmacokinetic and pharmacodynamic study of GSK2256098, a focal adhesion kinase inhibitor, in patients with advanced solid tumors. , 2016, Annals of oncology : official journal of the European Society for Medical Oncology.
[5] Thomas D. Wu,et al. Comprehensive genomic analysis of malignant pleural mesothelioma identifies recurrent mutations, gene fusions and splicing alterations , 2016, Nature Genetics.
[6] M. Serrano,et al. Stabilization of p21 by mTORC1/4E-BP1 predicts clinical outcome of head and neck cancers , 2016, Nature Communications.
[7] I. Vitale,et al. Trial Watch: Targeting ATM–CHK2 and ATR–CHK1 pathways for anticancer therapy , 2015, Molecular & Cellular Oncology.
[8] M. Meyerson,et al. Whole-exome sequencing reveals frequent genetic alterations in BAP1, NF2, CDKN2A, and CUL1 in malignant pleural mesothelioma. , 2015, Cancer research.
[9] C. Hanemann,et al. The p53/mouse double minute 2 homolog complex deregulation in merlin‐deficient tumours , 2015, Molecular oncology.
[10] Andrea I. McClatchey,et al. Merlin Deficiency Predicts FAK Inhibitor Sensitivity: A Synthetic Lethal Relationship , 2014, Science Translational Medicine.
[11] T. Leto,et al. Wild-type and mutant p53 differentially regulate NADPH oxidase 4 in TGF-β-mediated migration of human lung and breast epithelial cells , 2014, British Journal of Cancer.
[12] A. Giordano,et al. Pharmacological targeting of p53 through RITA is an effective antitumoral strategy for malignant pleural mesothelioma , 2014, Cell cycle.
[13] M. Donadelli,et al. Autophagy induced by p53-reactivating molecules protects pancreatic cancer cells from apoptosis , 2013, Apoptosis.
[14] F. Speleman,et al. Antitumor activity of the selective MDM2 antagonist nutlin-3 against chemoresistant neuroblastoma with wild-type p53. , 2009, Journal of the National Cancer Institute.
[15] W. El-Deiry,et al. Structural and Functional Basis for Therapeutic Modulation of p53 Signaling , 2008, Clinical Cancer Research.
[16] V. Golubovskaya,et al. p53 regulates FAK expression in human tumor cells , 2008, Molecular carcinogenesis.
[17] Susan J Fisher,et al. Nuclear FAK promotes cell proliferation and survival through FERM-enhanced p53 degradation. , 2008, Molecular cell.
[18] David R Bickers,et al. CP-31398 restores mutant p53 tumor suppressor function and inhibits UVB-induced skin carcinogenesis in mice. , 2007, The Journal of clinical investigation.
[19] Amie Y Lee,et al. Update on the molecular biology of malignant mesothelioma , 2007, Cancer.
[20] R. Stahel,et al. p53-induced apoptosis occurs in the absence of p14(ARF) in malignant pleural mesothelioma. , 2006, Neoplasia.
[21] K. Kito,et al. Novel orthotopic implantation model of human malignant pleural mesothelioma (EHMES‐10 cells) highly expressing vascular endothelial growth factor and its receptor , 2006, Cancer science.
[22] V. Golubovskaya,et al. Direct Interaction of the N-terminal Domain of Focal Adhesion Kinase with the N-terminal Transactivation Domain of p53* , 2005, Journal of Biological Chemistry.
[23] M. Demma,et al. CP-31398 Restores DNA-binding Activity to Mutant p53 in Vitro but Does Not Affect p53 Homologs p63 and p73* , 2004, Journal of Biological Chemistry.
[24] W. El-Deiry,et al. Restoring p53-Dependent Tumor Suppression , 2003, Cancer biology & therapy.
[25] Gang Li,et al. The p53 stabilizing compound CP-31398 induces apoptosis by activating the intrinsic Bax/mitochondrial/caspase-9 pathway. , 2002, Experimental cell research.
[26] Galina Selivanova,et al. Characterization of the p53-rescue drug CP-31398 in vitro and in living cells , 2002, Oncogene.
[27] W. El-Deiry,et al. The Mutant p53-Conformation Modifying Drug, CP-31398, Can Induce Apoptosis , 2002, Cancer biology & therapy.
[28] B. Foster,et al. Pharmacological rescue of mutant p53 conformation and function. , 1999, Science.
[29] M. Shingyoji,et al. A p53-stabilizing agent, CP-31398, induces p21 expression with increased G2/M phase through the YY1 transcription factor in esophageal carcinoma defective of the p53 pathway. , 2019, American journal of cancer research.