The chloride channel/transporter Slc26a9 regulates the systemic arterial pressure and renal chloride excretion

[1]  M. Soleimani,et al.  Slc26a11, a chloride transporter, localizes with the vacuolar H(+)-ATPase of A-intercalated cells of the kidney. , 2011, Kidney international.

[2]  I. J. Lynch,et al.  The renal H,K-ATPases , 2010, Current opinion in nephrology and hypertension.

[3]  D. Pearce,et al.  Role of Epithelial Sodium Channels and Their Regulators in Hypertension* , 2010, The Journal of Biological Chemistry.

[4]  S. Petrovic,et al.  Deletion of the anion exchanger Slc26a4 (pendrin) decreases apical Cl(-)/HCO3(-) exchanger activity and impairs bicarbonate secretion in kidney collecting duct. , 2010, American journal of physiology. Cell physiology.

[5]  I. Martins,et al.  Osmoregulation of ceroid neuronal lipofuscinosis type 3 in the renal medulla. , 2010, American journal of physiology. Cell physiology.

[6]  M. Rajagopal,et al.  Adenosine Activates A2b Receptors and Enhances Chloride Secretion in Kidney Inner Medullary Collecting Duct Cells , 2010, Hypertension.

[7]  M. Rajagopal,et al.  Adenosine Activates A 2 b Receptors and Enhances Chloride Secretion in Kidney Inner Medullary Collecting Duct Cells , 2010 .

[8]  S. Alper,et al.  Deletion of the Chloride Transporter Slc26a7 Causes Distal Renal Tubular Acidosis and Impairs Gastric Acid Secretion* , 2009, The Journal of Biological Chemistry.

[9]  Christopher S. Law,et al.  Tonicity-dependent induction of Sgk1 expression has a potential role in dehydration-induced natriuresis in rodents. , 2009, The Journal of clinical investigation.

[10]  M. Romero,et al.  Slc26a9—Anion Exchanger, Channel and Na+ Transporter , 2009, Journal of Membrane Biology.

[11]  J. Pilewski,et al.  SLC26A9 is a constitutively active, CFTR-regulated anion conductance in human bronchial epithelia , 2009, The Journal of general physiology.

[12]  Jian Zuo,et al.  Slc2a5 (Glut5) Is Essential for the Absorption of Fructose in the Intestine and Generation of Fructose-induced Hypertension* , 2009, Journal of Biological Chemistry.

[13]  S. Alper,et al.  Deletion of the chloride transporter Slc26a9 causes loss of tubulovesicles in parietal cells and impairs acid secretion in the stomach , 2008, Proceedings of the National Academy of Sciences.

[14]  U. Seidler,et al.  M1724 The Apical Chloride/Base Exchanger Slc26a6 (Pat1) and the Fructose-Absorbing Transporter Slc2a5 (Glut5) Play An Essential Role in the Pathogenesis of Fructose-Induced Hypertension , 2008 .

[15]  J. Donald,et al.  The effect of water deprivation on the tonicity responsive enhancer binding protein (TonEBP) and TonEBP-regulated genes in the kidney of the Spinifex hopping mouse, Notomys alexis , 2008, Journal of Experimental Biology.

[16]  K. Ishibashi,et al.  Aquaporin water channels in mammals , 1997, Clinical and Experimental Nephrology.

[17]  S. Muallem,et al.  SLC26A9 is a Cl− channel regulated by the WNK kinases , 2007, The Journal of physiology.

[18]  M. Soleimani Expression, regulation and the role of SLC26 Cl-/HCO3- exchangers in kidney and gastrointestinal tract. , 2006, Novartis Foundation symposium.

[19]  D. Spyropoulos,et al.  slc26a3 (dra)-deficient Mice Display Chloride-losing Diarrhea, Enhanced Colonic Proliferation, and Distinct Up-regulation of Ion Transporters in the Colon* , 2006, Journal of Biological Chemistry.

[20]  M. Soleimani,et al.  SLC26 chloride/base exchangers in the kidney in health and disease. , 2006, Seminars in nephrology.

[21]  D. Vandorpe,et al.  Anion exchangers in flux: functional differences between human and mouse SLC26A6 polypeptides. , 2006, Novartis Foundation symposium.

[22]  M. Romero,et al.  Physiology of electrogenic SLC26 paralogues. , 2006, Novartis Foundation symposium.

[23]  D. Witte,et al.  SLC26A9 is expressed in gastric surface epithelial cells, mediates Cl-/HCO3- exchange, and is inhibited by NH4+. , 2005, American journal of physiology. Cell physiology.

[24]  J. Lorenz,et al.  Renal and intestinal transport defects in Slc26a6-null mice. , 2005, American journal of physiology. Cell physiology.

[25]  S. Muallem,et al.  SLC26A7 Is a Cl– Channel Regulated by Intracellular pH* , 2005, Journal of Biological Chemistry.

[26]  D. Witte,et al.  SLC 26 A 9 is expressed in gastric surface epithelial cells , mediates Cl / HCO 3 exchange , and is inhibited by NH 4 , 2005 .

[27]  N. Simmons,et al.  Kinetics and regulation of a Ca2+-activated Cl- conductance in mouse renal inner medullary collecting duct cells. , 2004, American journal of physiology. Renal physiology.

[28]  J. Whelan,et al.  Role of WNK kinases in regulating tubular salt and potassium transport and in the development of hypertension , 2004 .

[29]  E. Green,et al.  Deoxycorticosterone Upregulates PDS (Slc26a4) in Mouse Kidney: Role of Pendrin in Mineralocorticoid-Induced Hypertension , 2003, Hypertension.

[30]  D. Wallace,et al.  Electrolyte and fluid secretion by cultured human inner medullary collecting duct cells. , 2002, American journal of physiology. Renal physiology.

[31]  J. Kere,et al.  Functional Characterization of Three Novel Tissue-specific Anion Exchangers SLC26A7, -A8, and -A9* , 2002, The Journal of Biological Chemistry.

[32]  E. Mann,et al.  Identification of an apical Cl(-)/HCO3(-) exchanger in the small intestine. , 2002, American journal of physiology. Gastrointestinal and liver physiology.

[33]  L. Rome,et al.  cAMP-dependent fluid secretion in rat inner medullary collecting ducts. , 2001, American journal of physiology. Renal physiology.

[34]  E. Green,et al.  Pendrin, encoded by the Pendred syndrome gene, resides in the apical region of renal intercalated cells and mediates bicarbonate secretion , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[35]  P. Kopp,et al.  Pendrin: an apical Cl-/OH-/HCO3- exchanger in the kidney cortex. , 2001, American journal of physiology. Renal physiology.

[36]  A. Vandewalle,et al.  Vasopressin-stimulated chloride transport in transimmortalized mouse cell lines derived from the distal convoluted tubule and cortical and inner medullary collecting ducts. , 2001, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[37]  N. Simmons,et al.  Bradykinin regulation of salt transport across mouse inner medullary collecting duct epithelium involves activation of a Ca2+‐dependent Cl− conductance , 2000, British journal of pharmacology.

[38]  J. Kere,et al.  Mapping of five new putative anion transporter genes in human and characterization of SLC26A6, a candidate gene for pancreatic anion exchanger. , 2000, Genomics.

[39]  N. Simmons,et al.  Ca2+ and cAMP‐activated Cl− conductances mediate Cl− secretion in a mouse renal inner medullary collecting duct cell line , 2000, The Journal of physiology.

[40]  R. Silver,et al.  H+-K+-ATPases: regulation and role in pathophysiological states. , 1999, American journal of physiology. Renal physiology.

[41]  D. Vandorpe,et al.  Vasopressin and cAMP stimulate electrogenic chloride secretion in an IMCD cell line. , 1995, The American journal of physiology.

[42]  V. Schuster Function and regulation of collecting duct intercalated cells. , 1993, Annual review of physiology.

[43]  G. Schwartz,et al.  Plasticity of functional epithelial polarity , 1985, Nature.