Absence of BBSome function leads to astrocyte reactivity in the brain
暂无分享,去创建一个
[1] M. Nachury. The molecular machines that traffic signaling receptors into and out of cilia. , 2018, Current opinion in cell biology.
[2] G. Jicha,et al. High complement levels in astrocyte‐derived exosomes of Alzheimer disease , 2018, Annals of neurology.
[3] V. Sheffield,et al. BBSome function is required for both the morphogenesis and maintenance of the photoreceptor outer segment , 2017, PLoS genetics.
[4] A. Nager,et al. BBSome trains remove activated GPCRs from cilia by enabling passage through the transition zone , 2017, bioRxiv.
[5] D. Valverde,et al. Bardet-Biedl Syndrome as a Chaperonopathy: Dissecting the Major Role of Chaperonin-Like BBS Proteins (BBS6-BBS10-BBS12) , 2017, Front. Mol. Biosci..
[6] Manoj Kumar,et al. INGE GRUNDKE-IQBAL AWARD FOR ALZHEIMER’S RESEARCH: NEUROTOXIC REACTIVE ASTROCYTES ARE INDUCED BY ACTIVATED MICROGLIA , 2019, Alzheimer's & Dementia.
[7] F. Shi,et al. Astrocyte-derived interleukin-15 exacerbates ischemic brain injury via propagation of cellular immunity , 2016, Proceedings of the National Academy of Sciences.
[8] J. Cerqueira,et al. S100B as a Potential Biomarker and Therapeutic Target in Multiple Sclerosis , 2015, Molecular Neurobiology.
[9] S. Kirov,et al. Adrenergic activation attenuates astrocyte swelling induced by hypotonicity and neurotrauma , 2016, Glia.
[10] V. Sheffield,et al. Mutations in C8ORF37 cause Bardet Biedl syndrome (BBS21). , 2016, Human molecular genetics.
[11] J. Badano,et al. Bardet–Biedl syndrome: Is it only cilia dysfunction? , 2015, FEBS letters.
[12] C. Escartin,et al. Elusive roles for reactive astrocytes in neurodegenerative diseases , 2015, Front. Cell. Neurosci..
[13] M. Asanuma,et al. Visualization of astrocytic primary cilia in the mouse brain by immunofluorescent analysis using the cilia marker Arl13b. , 2014, Acta medica Okayama.
[14] L. Facci,et al. Toll-Like Receptors 2, -3 and -4 Prime Microglia but not Astrocytes Across Central Nervous System Regions for ATP-Dependent Interleukin-1β Release , 2014, Scientific Reports.
[15] M. Pekny,et al. Astrocyte reactivity and reactive astrogliosis: costs and benefits. , 2014, Physiological reviews.
[16] M. Knight,et al. The primary cilium influences interleukin-1β-induced NFκB signalling by regulating IKK activity , 2014, Cellular signalling.
[17] Milos Pekny,et al. The dual role of astrocyte activation and reactive gliosis , 2014, Neuroscience Letters.
[18] I. Ouertani,et al. Update on the Genetics of Bardet-Biedl Syndrome , 2013, Molecular Syndromology.
[19] Y. Weihong,et al. Association between brain‐derived neurothropic factor variants and asthma in Chinese Han children , 2013, Acta paediatrica.
[20] W. Pan,et al. Brain interleukin-15 in neuroinflammation and behavior , 2013, Neuroscience & Biobehavioral Reviews.
[21] A. Quintana,et al. Interleukin-6, a Major Cytokine in the Central Nervous System , 2012, International journal of biological sciences.
[22] P. Walther,et al. Nuclear Factor κB Activation Impairs Ependymal Ciliogenesis and Links Neuroinflammation to Hydrocephalus Formation , 2012, The Journal of Neuroscience.
[23] P. Beales,et al. Bardet–Biedl syndrome , 2012, European Journal of Human Genetics.
[24] B. Barres,et al. Genomic Analysis of Reactive Astrogliosis , 2012, The Journal of Neuroscience.
[25] M. Knight,et al. Primary cilia elongation in response to interleukin-1 mediates the inflammatory response , 2012, Cellular and Molecular Life Sciences.
[26] R. Ransohoff,et al. Innate immunity in the central nervous system. , 2012, The Journal of clinical investigation.
[27] Ove Almkvist,et al. Evidence for Astrocytosis in Prodromal Alzheimer Disease Provided by 11C-Deuterium-L-Deprenyl: A Multitracer PET Paradigm Combining 11C-Pittsburgh Compound B and 18F-FDG , 2012, The Journal of Nuclear Medicine.
[28] P. Nopoulos,et al. Brain tissue- and region-specific abnormalities on volumetric MRI scans in 21 patients with Bardet-Biedl syndrome (BBS) , 2011, BMC Medical Genetics.
[29] A. Kriegstein,et al. Developmental genetics of vertebrate glial–cell specification , 2010, Nature.
[30] M. Guillermier,et al. In vivo expression of polyglutamine-expanded huntingtin by mouse striatal astrocytes impairs glutamate transport: a correlation with Huntington's disease subjects , 2010, Human molecular genetics.
[31] A. Álvarez-Buylla,et al. Role of primary cilia in brain development and cancer , 2010, Current Opinion in Neurobiology.
[32] M. Sofroniew,et al. Astrocytes: biology and pathology , 2009, Acta Neuropathologica.
[33] N. Katsanis,et al. Identification of 11 novel mutations in eight BBS genes by high-resolution homozygosity mapping , 2009, Journal of Medical Genetics.
[34] D. Butterfield,et al. Proteomics-determined differences in the concanavalin-A-fractionated proteome of hippocampus and inferior parietal lobule in subjects with Alzheimer's disease and mild cognitive impairment: implications for progression of AD. , 2009, Journal of proteome research.
[35] J. Arellano,et al. Primary cilia regulate hippocampal neurogenesis by mediating sonic hedgehog signaling , 2008, Proceedings of the National Academy of Sciences.
[36] J. García-Verdugo,et al. Hedgehog signaling and primary cilia are required for the formation of adult neural stem cells , 2008, Nature Neuroscience.
[37] H. Omran,et al. When cilia go bad: cilia defects and ciliopathies , 2007, Nature Reviews Molecular Cell Biology.
[38] Milos Pekny,et al. Redefining the concept of reactive astrocytes as cells that remain within their unique domains upon reaction to injury , 2006, Proceedings of the National Academy of Sciences.
[39] Lars Funke,et al. Synapse-Specific and Developmentally Regulated Targeting of AMPA Receptors by a Family of MAGUK Scaffolding Proteins , 2006, Neuron.
[40] Nicholas Katsanis,et al. The ciliopathies: an emerging class of human genetic disorders. , 2006, Annual review of genomics and human genetics.
[41] T. Kielian. Toll‐like receptors in central nervous system glial inflammation and homeostasis , 2006, Journal of neuroscience research.
[42] S. Grant,et al. Organization of brain complexity--synapse proteome form and function. , 2006, Briefings in functional genomics & proteomics.
[43] S. Grant,et al. The proteomes of neurotransmitter receptor complexes form modular networks with distributed functionality underlying plasticity and behaviour , 2006, Molecular systems biology.
[44] M. Nilsson,et al. Astrocyte activation and reactive gliosis , 2005, Glia.
[45] E. Schuman,et al. A proteasome-sensitive connection between PSD-95 and GluR1 endocytosis , 2004, Neuropharmacology.
[46] J. Grassi,et al. Secretion of interleukin‐1β by astrocytes mediates endothelin‐1 and tumour necrosis factor‐α effects on human brain microvascular endothelial cell permeability , 2003 .
[47] E. Hansson,et al. Glial neuronal signaling in the central nervous system , 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[48] Jean-Claude Béïque,et al. PSD‐95 regulates synaptic transmission and plasticity in rat cerebral cortex , 2003, The Journal of physiology.
[49] R. Nicoll,et al. PSD-95 involvement in maturation of excitatory synapses. , 2000, Science.
[50] W. Walz. Role of astrocytes in the clearance of excess extracellular potassium , 2000, Neurochemistry International.
[51] S. K. Malhotra,et al. Reactive astrocytes: cellular and molecular cues to biological function , 1997, Trends in Neurosciences.
[52] D. Nowlan,et al. Costs and benefits. , 1980, Irish medical journal.
[53] J. Vanderhaeghen,et al. An acidic protein isolated from fibrous astrocytes. , 1971, Brain research.
[54] J. Grassi,et al. Secretion of interleukin-1beta by astrocytes mediates endothelin-1 and tumour necrosis factor-alpha effects on human brain microvascular endothelial cell permeability. , 2003, Journal of neurochemistry.
[55] N. Niikawa,et al. [Bardet-Biedl syndrome(BBS)]. , 2000, Ryoikibetsu shokogun shirizu.
[56] T. Shnitka,et al. Rat glioma cell line as a model for astrogliosis. , 1995, Cytobios.