p18(INK4c) collaborates with other CDK‐inhibitory proteins in the regenerating liver
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T. Luedde | F. Tacke | C. Trautwein | D. Brenner | Y. Xiong | M. E. Rodriguez | D. Brenner | María Elena Rodríguez
[1] G. Higgins,et al. Experimental pathology of the liver , 1931 .
[2] I. Tsukamoto,et al. The sex difference in the regulation of liver regeneration after partial hepatectomy in the rat. , 1990, Biochimica et biophysica acta.
[3] Variations in regenerative growth of mouse liver following partial hepatectomy. , 1990 .
[4] J. Nevins,et al. E2F: a link between the Rb tumor suppressor protein and viral oncoproteins. , 1992, Science.
[5] R Pepperkok,et al. Regulation of the cell cycle by the cdk2 protein kinase in cultured human fibroblasts , 1993, The Journal of cell biology.
[6] C. O'keefe,et al. Growth suppression by p18, a p16INK4/MTS1- and p14INK4B/MTS2-related CDK6 inhibitor, correlates with wild-type pRb function. , 1994, Genes & development.
[7] S. Shurtleff,et al. D-type cyclin-dependent kinase activity in mammalian cells , 1994, Molecular and cellular biology.
[8] James M. Roberts,et al. Inhibitors of mammalian G1 cyclin-dependent kinases. , 1995, Genes & development.
[9] Stephen J. Elledge,et al. Mice Lacking p21 CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint control , 1995, Cell.
[10] David O. Morgan,et al. Principles of CDK regulation , 1995, Nature.
[11] James M. Roberts,et al. Human cyclin E, a nuclear protein essential for the G1-to-S phase transition , 1995, Molecular and cellular biology.
[12] K. Manova-Todorova,et al. Enhanced Growth of Mice Lacking the Cyclin-Dependent Kinase Inhibitor Function of p27 Kip1 , 1996, Cell.
[13] D. Franklin,et al. Induction of p18INK4c and its predominant association with CDK4 and CDK6 during myogenic differentiation. , 1996, Molecular biology of the cell.
[14] S. Elledge,et al. Cdk inhibitors in development and cancer. , 1996, Current opinion in genetics & development.
[15] P. Nurse,et al. Regulating S Phase: CDKs, Licensing and Proteolysis , 1996, Cell.
[16] Y. Xiong,et al. Immunoprecipitation and Immunoblotting in Cell Cycle Studies , 1996 .
[17] R. Sclafani,et al. Cyclin dependent kinase activating kinases. , 1996, Current opinion in cell biology.
[18] G. Michalopoulos,et al. Liver Regeneration , 1997, Science.
[19] F. Zindy,et al. Expression of INK4 inhibitors of cyclin D-dependent kinases during mouse brain development. , 1997, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[20] F. Zindy,et al. Expression of the p16INK4a tumor suppressor versus other INK4 family members during mouse development and aging , 1997, Oncogene.
[21] J. Baldassare,et al. Sustained activation of extracellular-signal-regulated kinase 1 (ERK1) is required for the continued expression of cyclin D1 in G1 phase. , 1997, The Biochemical journal.
[22] D. Franklin,et al. Coupled Transcriptional and Translational Control of Cyclin-Dependent Kinase Inhibitor p18INK4cExpression during Myogenesis , 1998, Molecular and Cellular Biology.
[23] J. Albrecht,et al. Involvement of p21 and p27 in the regulation of CDK activity and cell cycle progression in the regenerating liver , 1998, Oncogene.
[24] J. Albrecht,et al. Expression of cyclin‐dependent kinase inhibitor p21 in human liver , 1998, Hepatology.
[25] V. Godfrey,et al. CDK inhibitors p18(INK4c) and p27(Kip1) mediate two separate pathways to collaboratively suppress pituitary tumorigenesis. , 1998, Genes & development.
[26] M. Roussel,et al. Assembly of cyclin D-dependent kinase and titration of p27Kip1 regulated by mitogen-activated protein kinase kinase (MEK1). , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[27] R. Hoess,et al. Defining the substrate specificity of cdk4 kinase-cyclin D1 complex. , 1999, Carcinogenesis.
[28] James M. Roberts,et al. CDK inhibitors: positive and negative regulators of G1-phase progression. , 1999, Genes & development.
[29] K. Engeland,et al. Decreased expression of p27 protein is associated with advanced tumor stage in hepatocellular carcinoma , 2000, International journal of cancer.
[30] V. Godfrey,et al. Functional Collaboration between Different Cyclin-Dependent Kinase Inhibitors Suppresses Tumor Growth with Distinct Tissue Specificity , 2000, Molecular and Cellular Biology.
[31] T. Luedde,et al. Interleukin 6 and liver regeneration , 2000, Gut.
[32] M. Serra,et al. Predictors of morbidity and mortality after the first episode of upper gastrointestinal bleeding in liver cirrhosis. , 2000, Journal of hepatology.
[33] P. Gruppuso,et al. A potential role for p15Ink4b and p57Kip2 in liver development , 2000 .
[34] M. Chao,et al. p21cip1 is required for the differentiation of oligodendrocytes independently of cell cycle withdrawal , 2001, EMBO reports.
[35] M. Manns,et al. Ras adenoviruses modulate cyclin E protein expression and DNA synthesis after partial hepatectomy , 2001, Oncogene.
[36] I. Ng,et al. Expression of p27KIP1 and p21WAF1/CIP1 in primary hepatocellular carcinoma: Clinicopathologic correlation and survival analysis , 2001 .
[37] J. Albrecht,et al. Transient expression of cyclin D1 is sufficient to promote hepatocyte replication and liver growth in vivo. , 2001, Cancer research.
[38] M. Grompe,et al. Loss of p27(Kip1) enhances the transplantation efficiency of hepatocytes transferred into diseased livers. , 2001, The Journal of clinical investigation.
[39] A. Cuenca,et al. Calorie Restriction Influences Cell Cycle Protein Expression and DNA Synthesis during Liver Regeneration , 2001, Experimental biology and medicine.
[40] D. Scadden,et al. Cell Cycle Entry of Hematopoietic Stem and Progenitor Cells Controlled by Distinct Cyclin-Dependent Kinase Inhibitors , 2002, International journal of hematology.